Dermatologic Manifestations of Paronychia Treatment & Management

  • Author: Allison Vidimos, MD, RPh; Chief Editor: Dirk M Elston, MD   more...
 
Updated: Jun 10, 2010
 

Medical Care

The treatment of choice depends on the extent of the infection. If diagnosed early, acute paronychia without obvious abscess can be treated nonsurgically. If an abscess has developed, incision and drainage must be performed. Surgical debridement may be required if fulminant infection is present.

Herpetic whitlow and paronychia must be distinguished because the treatments are drastically different. Misdiagnosis and mistreatment may do more harm than good. Once herpetic whitlow is ruled out, one must determine whether the paronychia is acute or chronic and then treat it accordingly.[5]

Acute paronychia

Warm water soaks of the affected finger 3-4 times per day until symptoms resolve are helpful.

Oral antibiotics with gram-positive coverage against S aureus, such as amoxicillin and clavulanic acid (Augmentin) or clindamycin (Cleocin), are usually administered concomitantly with warm water soaks. Cleocin and Augmentin also have anaerobic activity; therefore, they are useful in treating patients with paronychia due to oral anaerobes contracted through nail biting or finger sucking. Cleocin should be used instead of Augmentin in patients who are allergic to penicillin.

If the paronychia does not resolve or if it progresses to an abscess, it should be drained promptly.

Chronic paronychia

The initial treatment of chronic paronychia consists of the avoidance of inciting factors such as exposure to moist environments or skin irritants. Keeping the affected lesion dry is essential for proper recovery. Choice of footgear may also be considered.

Any manipulation of the nail, such as manicuring, finger sucking, or attempting to incise and drain the lesion, should be avoided; these manipulations may lead to secondary bacterial infections.

Mild cases of chronic paronychia may be treated with warm soaks.

The initial medical treatment consists of the application of topical antifungal agents. Topical miconazole may be used as the initial agent. Oral ketoconazole or fluconazole may be added in more severe cases.

Patients with diabetes and those who are immunocompromised need more aggressive treatment because the response to therapy is slower in these patients than in others.

In cases induced by retinoids or protease inhibitors, the paronychia usually resolves if the medication is discontinued.

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Surgical Care

If paronychia does not resolve despite best medical efforts, surgical intervention may be indicated. Also, if an abscess has developed, incision and drainage must be performed. Surgical debridement may be required if fulminant infection is present.

Acute paronychia

The no-incision technique is as follows:

  • Less advanced paronychial abscesses can be drained simply by gently elevating the eponychial fold from the nail by using a small blunt instrument such as a metal probe or an elevator (see the images below). This separation is performed at the junction of the perionychium and the eponychium and extends proximally enough to permit visualization of the proximal nail edge. Then, the proximal third of the nail can be excised with scissors and the pus evacuated. Typical appearance of paronychia. Typical appearance of paronychia. Simple acute paronychia can be drained by elevatinSimple acute paronychia can be drained by elevating the eponychial fold from the nail with a small blunt instrument such as a metal probe or elevator.
  • This technique does not require an incision into the matrix. Often, no excision of any tissues is made because only blunt dissection and separation are needed to evacuate the pus from the paronychia.
  • The wound should be well irrigated with isotonic sodium chloride solution, and plain gauze packing should be inserted under the fold to keep the cavity open and allow drainage.
  • The patient should receive oral antibiotics for 5-7 days.
  • The packing is removed after 2 days, and warm sodium chloride solution soaks are begun

The single- and double-incision techniques are as follows:

  • If the paronychia is more advanced, it may need to be incised and drained.
  • A digital anesthetic block is usually necessary. If an anesthetic agent is used, it should consist of 1% lidocaine (Xylocaine) with no epinephrine for a ring block. The local injection of the anesthetic agent into the paronychia or the wound is often inadequate and more painful than the administration of drugs of a digital ring block.
  • If the paronychia involves only 1 lateral fold of the finger, a single longitudinal incision should be placed with either a number-11 or a number-15 blade directed away from the nail fold to prevent proximal injury with a subsequent nail growth abnormality. If both lateral folds of the finger are involved, incisions may be made on both sides of the nail, extending proximally to the base of the nail.
  • After the single or double incision is made, the entire eponychial fold is elevated to expose the base of the nail and drain the pus.
  • The proximal third of the nail is removed by using the technique described for the no-incision technique.
  • After the abscess is drained, the pocket should be well irrigated with isotonic sodium chloride solution, packed with plain packing, and dressed.
  • The patient should receive oral antibiotics for 5-7 days.
  • The dressing and packing are removed in approximately 2 days, and the affected finger is treated with warm soaks for 10-15 minutes 3-4 times per day.

Chronic paronychia

Treatment is as follows:

  • The most common surgical technique used to treat chronic paronychia is called eponychial marsupialization.
  • In this technique, the affected digit is first anesthetized with 1% lidocaine (Xylocaine) with no epinephrine by using the digital ring block method.
  • Tourniquet control of the proximal digit is accomplished by using a finger of a latex glove with the distal end cut off.
  • With a No. 15 blade, a crescent-shaped incision is made proximal to the distal edge of the eponychial fold.
  • The distal incision is made approximately 1 mm proximal to the distal edge of the eponychium and along its curve.
  • At its widest end, the proximal incision is approximately 5 mm from the distal incision.
  • The incision should appear symmetric and extend to the edge of the nail fold on each side.
  • All affected tissue within the boundaries of the crescent and extending down to, but not including, the germinal matrix is excised. In effect, this procedure exteriorizes the infected and obstructed nail matrix and allows its drainage.
  • If the nail plate is grossly deformed at the time of surgery, it may be removed.
  • The excised region is packed with plain gauze wick, which is changed every 2-3 days.
  • Epithelialization of the excised defect occurs over the next 2-3 weeks.
  • Nail improvement occurs over the next 6-9 months but may require as long as 12 months to become apparent.
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Diet

No change in diet is required.

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Activity

Patients with either acute or chronic paronychia should keep affected areas clean and dry, and they should avoid any further trauma or manipulation of the nail.

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Contributor Information and Disclosures
Author

Allison Vidimos, MD, RPh  Staff, Section of Micrographic Surgery (Mohs) and Oncology; Chairman, Department of Dermatology; Cleveland Clinic

Allison Vidimos, MD, RPh is a member of the following medical societies: American Academy of Dermatology, American College of Mohs Micrographic Surgery and Cutaneous Oncology, American Medical Association, American Society for Dermatologic Surgery, American Society for Laser Medicine and Surgery, and International Society for Dermatologic Surgery

Disclosure: Nothing to disclose.

Coauthor(s)

Elizabeth M Billingsley, MD  Professor of Dermatology, Penn State University College of Medicine; Director, Dermatologic Surgery and Mohs Micrographic Surgery, Penn State Hersey Medical Center

Elizabeth M Billingsley, MD is a member of the following medical societies: American Academy of Dermatology, American College of Mohs Micrographic Surgery and Cutaneous Oncology, Association of Professors of Dermatology, Council for Nail Disorders, and Pennsylvania Academy of Dermatology

Disclosure: Nothing to disclose.

Specialty Editor Board

Richard K Scher, MD  Professor of Dermatology, University of North Carolina

Richard K Scher, MD is a member of the following medical societies: Alpha Omega Alpha, American Academy of Dermatology, American College of Cryosurgery, American College of Physicians, American Dermatological Association, American Geriatrics Society, American Medical Association, Association of Military Surgeons of the US, International Society for Dermatologic Surgery, New York Academy of Sciences, Noah Worcester Dermatological Society, Rhode Island Medical Society, and Society for Investigative Dermatology

Disclosure: Nothing to disclose.

David F Butler, MD  Professor of Dermatology, Texas A&M University College of Medicine; Chair, Department of Dermatology, Director, Dermatology Residency Training Program, Scott and White Clinic, Northside Clinic

David F Butler, MD is a member of the following medical societies: Alpha Omega Alpha, American Academy of Dermatology, American Medical Association, American Society for Dermatologic Surgery, American Society for MOHS Surgery, Association of Military Dermatologists, and Phi Beta Kappa

Disclosure: Nothing to disclose.

Julia R Nunley, MD  Professor, Program Director, Dermatology Residency, Department of Dermatology, Virginia Commonwealth University Medical Center

Julia R Nunley, MD is a member of the following medical societies: American Academy of Dermatology, American College of Physicians, American Society of Nephrology, International Society of Nephrology, Medical Dermatology Society, Medical Society of Virginia, National Kidney Foundation, Phi Beta Kappa, and Women's Dermatologic Society

Disclosure: Novartis Grant/research funds Consulting; Biolex Grant/research funds sub-investigator

Glen H Crawford, MD  Assistant Clinical Professor, Department of Dermatology, University of Pennsylvania School of Medicine; Chief, Division of Dermatology, The Pennsylvania Hospital

Glen H Crawford, MD is a member of the following medical societies: Alpha Omega Alpha, American Academy of Dermatology, American Medical Association, Phi Beta Kappa, and Society of USAF Flight Surgeons

Disclosure: Nothing to disclose.

Chief Editor

Dirk M Elston, MD  Director, Department of Dermatology, Geisinger Medical Center

Dirk M Elston, MD is a member of the following medical societies: American Academy of Dermatology

Disclosure: Nothing to disclose.

References
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Depicted are the nail fold (A), dorsal roof (B), ventral floor (C), nail wall (D), perionychium (E), lunula (F), nail bed (G), germinal matrix (H), sterile matrix (I), nail plate (J), hyponychium (K), distal groove (L), fascial septa (M), fat pad (N), distal interphalangeal joint (O), and extensor tendon insertion (P).
Typical appearance of paronychia.
Simple acute paronychia can be drained by elevating the eponychial fold from the nail with a small blunt instrument such as a metal probe or elevator.
 
 
 
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