Even though cutaneous larva migrans (CLM) is self-limited, the intense pruritus and risk for infection mandate treatment. Prevention is key and involves avoidance of direct skin contact with fecally contaminated soil.
Prior to the 1960s, topical modalities such as ethyl chloride spray, liquid nitrogen, phenol, carbon dioxide snow, piperazine citrate, electrocautery, and radiation therapy were used unsuccessfully because the larvae of cutaneous larva migrans might be missed and/or not be killed. Chemotherapy with chloroquine, antimony, and diethylcarbamazine were also unsuccessful. Thiabendazole is currently considered the agent of choice for the treatment of cutaneous larva migrans. [17, 18, 19, 20]
A secondary bacterial infection in patients with cutaneous larva migrans (CLM), usually with Streptococcus pyogenes, may lead to cellulitis.
Allergic reactions may occur.
On rare occasions, Loeffler syndrome has been reported. 
Consultation with a dermatologist may be warranted.
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