Traumatic Cataract Clinical Presentation
- Author: Robert H Graham, MD; Chief Editor: Hampton Roy Sr, MD more...
History
Note the following:
- Mechanism of injury - Sharp versus blunt
- Past ocular history - Previous eye surgery, glaucoma, retinal detachment, diabetic eye disease
- Past medical history - Diabetes, sickle cell, Marfan syndrome, homocystinuria, hyperlysinemia, sulfate oxidase deficiency
- Visual complaints - Decreased vision (cataract, lens subluxation, lens dislocation, ruptured globe, traumatic optic neuropathy, vitreous hemorrhage, retinal detachment); monocular diplopia (lens subluxation with partial phakic and aphakic vision); binocular diplopia (traumatic nerve palsy, orbital fracture); pain (glaucoma secondary to hyphema, pupillary block, or lens particles; retrobulbar hemorrhage; iritis)
Physical
Complete ophthalmic examination (defer in case of globe compromise), to include the following:
- Vision and pupils - Presence of afferent pupillary defect (APD) indicative of traumatic optic neuropathy
- Extraocular motility - Orbital fractures or traumatic nerve palsy
- Intraocular pressure - Secondary glaucoma, retrobulbar hemorrhage
- Anterior chamber - Hyphema, iritis, shallow chamber, iridodonesis, angle recession
- Lens - Subluxation, dislocation, capsular integrity (anterior and posterior), cataract (extent and type), swelling, phacodonesis
- Vitreous - Presence or absence of hemorrhage, posterior vitreous detachment
- Fundus - Retinal detachment, choroidal rupture, commotio retinae, preretinal hemorrhage, intraretinal hemorrhage, subretinal hemorrhage, optic nerve pallor, optic nerve avulsion
Causes
Traumatic cataracts occur secondary to blunt or penetrating ocular trauma.
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