Traumatic Cataract

Author: Robert H Graham, MD; Chief Editor: Hampton Roy Sr, MD more...

Updated: Feb 4, 2009

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Background
Pathophysiology
Epidemiology
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Background

Traumatic cataracts occur secondary to blunt or penetrating ocular trauma. Infrared energy (glass-blower's cataract), electric shock, and ionizing radiation are other rare causes of traumatic cataracts.^[1]

Cataracts caused by blunt trauma classically form stellate- or rosette-shaped posterior axial opacities that may be stable or progressive, whereas penetrating trauma with disruption of the lens capsule forms cortical changes that may remain focal if small or may progress rapidly to total cortical opacification.

Note the images below.

Classic rosette-shaped cataract in a 36-year-old man, 4 weeks after blunt ocular injury.

Same cataract as seen in previous image, viewed by retroillumination.

Lens dislocation and subluxation are commonly found in conjunction with traumatic cataract. Other associated complications include phacolytic, phacomorphic, pupillary block, and angle-recession glaucoma; phacoanaphylactic uveitis; retinal detachment; choroidal rupture; hyphema; retrobulbar hemorrhage; traumatic optic neuropathy; and globe rupture.^{[2, 3, 4]}

Traumatic cataract can present many medical and surgical challenges to the ophthalmologist. Careful examination and a management plan can simplify these difficult cases and provide the best possible outcome.^{[4, 5]}

Pathophysiology

Blunt trauma is responsible for coup and contrecoup ocular injury. Coup is the mechanism of direct impact. It is responsible for Vossius ring (imprinted iris pigment) sometimes found on the anterior lens capsule following blunt injury. Contrecoup refers to distant injury caused by shockwaves traveling along the line of concussion.

When the anterior surface of the eye is struck bluntly, there is a rapid anterior-posterior shortening accompanied by equatorial expansion. This equatorial stretching can disrupt the lens capsule, zonules, or both. Combination of coup, contrecoup, and equatorial expansion is responsible for formation of traumatic cataract following blunt ocular injury.^{[6, 7]}

Penetrating trauma that directly compromises the lens capsule leads to cortical opacification at the site of injury. If the rent is sufficiently large, the entire lens rapidly opacifies, but when small, cortical cataract can seal itself off and remain localized.

Frequency

United States

Approximately 2.5 million eye injuries occur annually in the United States. It is estimated that approximately 4-5% of a comprehensive ophthalmologist's patients are seen secondary to ocular injury. Traumatic cataract may present as acute, subacute, or late sequela of ocular trauma.

Mortality/Morbidity

Trauma is the leading cause of monocular blindness in people younger than 45 years. Annually, approximately 50,000 people are left unable to read newsprint as a result of ocular trauma. Only 85% patients who experience anterior segment injury reach a final visual acuity of 20/40 or better, whereas only 40% patients with posterior segment injury reach this level.^{[6, 7]}

Sex

The male-to-female ratio in cases of ocular trauma is 4:1.

Age

Work- and sports-related eye injuries most commonly occur in children and young adults. Between 1985-1991, a National Eye Trauma System study reported a median age of 28 years in 648 assault-related cases.

Proceed to Clinical Presentation

Contributor Information and Disclosures

Author

Robert H Graham, MD Senior Associate Consultant, Department of Ophthalmology, Mayo Clinic, Scottsdale, Arizona

Robert H Graham, MD is a member of the following medical societies: American Academy of Ophthalmology, American Medical Association, and Arizona Ophthalmological Society

Disclosure: WebMD/eMedicine Salary Employment

Coauthor(s)

Brian C Mulrooney, MD Private Practice, Ophthalmology, Crestwood Hospital

Brian C Mulrooney, MD is a member of the following medical societies: American Academy of Ophthalmology

Disclosure: Nothing to disclose.

Specialty Editor Board

Richard W Allinson, MD Associate Professor, Department of Ophthalmology, Texas A&M University Health Science Center; Senior Staff Ophthalmologist, Scott and White Clinic

Richard W Allinson, MD, is a member of the following medical societies: American Academy of Ophthalmology, American Medical Association, and Texas Medical Association

Disclosure: Nothing to disclose.

Simon K Law, MD, PharmD Assistant Professor of Ophthalmology, Jules Stein Eye Institute; Chief of Section of Ophthalmology Surgical Services, Department of Veterans Affairs Healthcare Center, West Los Angeles

Simon K Law, MD, PharmD is a member of the following medical societies: American Academy of Ophthalmology, American Glaucoma Society, and Association for Research in Vision and Ophthalmology

Disclosure: Nothing to disclose.

J James Rowsey, MD Former Director of Corneal Services, St Luke's Cataract and Laser Institute, Florida

J James Rowsey, MD is a member of the following medical societies: American Academy of Ophthalmology, American Association for the Advancement of Science, American Medical Association, Association for Research in Vision and Ophthalmology, Florida Medical Association, Pan-American Association of Ophthalmology, Sigma Xi, and Southern Medical Association

Disclosure: Nothing to disclose.

Lance L Brown, OD, MD Ophthalmologist, Affiliated With Freeman Hospital and St John's Hospital, Regional Eye Center, Joplin, Missouri

Disclosure: Nothing to disclose.

Chief Editor

Hampton Roy Sr, MD Associate Clinical Professor, Department of Ophthalmology, University of Arkansas for Medical Sciences

Hampton Roy Sr, MD is a member of the following medical societies: American Academy of Ophthalmology, American College of Surgeons, and Pan-American Association of Ophthalmology

Disclosure: Nothing to disclose.

References

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Kanski JJ. Clinical Ophthalmology: A Systematic Approach. 1989:257-8.
Schwab IR, et al. Anterior segment trauma. In: AAO Basic and Clinical Science Course. Section 8. 1997:285-6.
Witherspoon CD, Kunh F, Morris R, et al. Anterior and posterior segment trauma. Master Techniques in Ophthalmic Surgery. 1995;538-47.
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