eMedicine Specialties > Orthopedic Surgery > Foot & Ankle
Plantar Heel Pain: Treatment & Medication
Updated: Jun 17, 2008
- Overview
- Differential Diagnoses & Workup
- Treatment & Medication
- Follow-up
- Multimedia
Treatment
Medical Care
Proximal plantar fasciitis is successfully managed with conservative care in approximately 90% of cases. In general, the longer the duration of symptoms, the longer it takes for the patient to obtain complete pain relief. Various modalities of treatment are available, and patient education is important to improve the understanding of the condition and to obtain compliance with various treatment regimens. The important aims of the treatment are to limit impact stresses on the heel, to alleviate inflammation, and to stretch the triceps surae muscle.
- Reducing impact
- Activity modification: Avoiding impact activities is especially important in athletes, who can cross-train with nonimpact sports such as cycling or swimming.
- Reduction of body weight
- Use of soft cushions or insoles and soft-heeled footwear (see Images 1-2)
- Taping, arch supports, and custom-molded orthotics (see Image 3)14,15
- Reducing inflammation
- Application of ice and/or iontophoresis
- Anti-inflammatory medication: Medication is useful in the early stages, especially if the patient has begun stretching exercises because, initially, these can worsen the pain (see Medication).
- Stretching and strengthening
- Exercises: A variety of exercises can help the patient to achieve active and passive ankle dorsiflexion with the knee kept straight and the subtalar joint in inversion, which helps achieve maximum stretch of the triceps surae muscle. The foot can be rolled over a tennis ball or a can to massage and stretch the plantar fascia. The exercises can be performed at home or can be guided by a physical therapist (see Images 4-6).
- Plantar fascia–specific stretching exercises: A randomized, prospective study with 2-year follow up compared Achilles tendon stretching with plantar fascia tissue—specific exercises.16 The authors found a plantar fascia–specific stretching exercises was better. To perform the exercise, the patient crosses the affected leg over the contralateral leg. While placing the fingers across the base of the toes, the patient pulls the toes back toward the shin until he or she feels a stretch in the arch or plantar fascia. The patient confirms that the stretch was correct by palpating tension in the plantar fascia. (see Image 10).
- Intrinsic muscle strengthening: Exercises include toe curls or other activities, such as picking up marbles with the toes.
- Resting splints: During the night, the relaxed posture of plantar flexion at the ankle tends to favor contracture of the triceps surae. To prevent this, night splints that hold the ankle in dorsiflexion can be worn.17 Patients who wear a posterior night splint should be warned to take it off before getting out of bed. As an anecdotal example, one patient walked to the toilet while wearing the splint, slipped, and sustained a humeral fracture. However, a dorsally applied splint, as opposed to a posterior splint, does not need to be taken off before getting out of bed (see Images 7-8).
- Treating recalcitrant pain: If the pain persists for longer than 2 months despite the above treatment, then the following modalities can be offered:
- Cast: A short leg walking cast for 6 weeks is generally effective in relieving pain, but the pain can recur after the cast is removed.18 To prevent this, the patient should use the previously mentioned treatment modalities, such as activity modification, stretching exercises, and insoles, until recovery is complete.
- Corticosteroids: Iontophoresis is administered by a physical therapist and uses low-voltage galvanic current stimulation to distribute topical corticosteroids. It is performed 2-3 times a week. This therapy can provide short-term relief, but it is usually reserved for patients in whom other therapies are unsuccessful or who have occupations that involve spending most of their time on their feet. Depot injections can provide good short-term relief, but multiple injections can cause plantar fascia rupture and fat pad atrophy—and, later, a flat-foot deformity — especially if the injection is not administered deep into the fascia.
- Extracorporeal shockwave therapy (ESWT)5,19,20 : This therapy was approved by the US Food and Drug Administration (FDA) in 2005 (see New Device Approval: Orthospec Extracorporeal Shock Wave Therapy – P040026), although the treatment has been used in Europe for more than a decade. Animal study data suggest that this modality creates microdisruption and stimulates new bone and tissue formation. Shock waves may be delivered in 3 ways: (1) electrohydraulically (high power), (2) electromagnetically, and (3) piezoelectrically. The FDA approved electrohydraulic and electromagnetic devices for the treatment of chronic plantar heel pain that has persisted for longer than 6 months despite other treatment.
Fat pad atrophy is managed conservatively with the use of heel cups, soft insoles, and soft-soled footwear. The heel cup helps to centralize and increase the bulk of the soft tissue under the calcaneus.
In patients with planovalgus deformity, if the valgus hindfoot is thought to be the cause of tarsal tunnel syndrome due to traction on the tibial nerve, the initial treatment can be placement of a medial longitudinal arch support and a medial lift.
Stress fractures of the calcaneus and traumatic rupture of the plantar fascia are managed with conservative measures. Avoiding the offending activity and a 6- to 8-week period in a cast may be required to alleviate the symptoms.
Surgical Care
The guidelines developed by the American Orthopaedic Foot and Ankle Society on the use of endoscopic and open heel surgery to treat plantar heel pain are widely accepted.
Because 90% of patients with plantar fasciitis respond favorably to conservative care, conservative methods should be tried for at least 6, or, preferably 12, months before surgery is considered. Furthermore, full counseling regarding the risks and benefits must be administered because complete satisfaction after surgery is observed in only 50% of patients.
The surgery can be performed by open or endoscopic methods. However, if plantar fasciitis is suspected to coexist with compression of the first branch of the lateral plantar nerve, then the endoscopic method is not recommended. (Electromyography and nerve conduction studies are not necessary to diagnose compressive neuropathy of the first branch of the lateral plantar nerve; rather, the diagnosis of entrapment of the first branch of the lateral plantar nerve is made on a clinical basis. Testing nerve conduction across the site of entrapment in the heel is technically demanding. Motor weakness in the abductor digiti quinti may not be detected because of the dynamic nature of the compression.)
- By either the open or endoscopic method, only 50% of the plantar fascia is released because a complete release can lead to collapse of the medial and lateral longitudinal arches.
- Excision of a plantar heel spur is performed only if it is significantly large and it is compressing the first branch of the lateral plantar nerve.
- Surgery for tarsal tunnel syndrome or for decompression of the first branch of lateral plantar nerve requires release of the tibial nerve and its branches and overlying fascia, including the deep fascia of the abductor hallucis.
Medication
Medication is useful in the early stages, especially if the patient has begun stretching exercises, because, initially, these can worsen the pain.
Analgesics
Pain control is essential to quality patient care. Analgesics ensure patient comfort, promote pulmonary toilet, and have sedating properties, which are beneficial for patients who have sustained trauma or who have sustained injuries.
Acetaminophen (Aspirin Free Anacin, Feverall, Tylenol)
DOC for pain in patients with documented hypersensitivity to aspirin or NSAIDs, with upper GI disease, or who are taking PO anticoagulants.
Effective in relieving mild to moderate acute pain; however, it has no peripheral anti-inflammatory effects. May be preferred in elderly patients because of fewer adverse GI and renal effects.
Adult
325-650 mg PO/PR q4-6h or 1000 mg tid/qid; not to exceed 4 g/d
Pediatric
<12 years: 10-15 mg/kg/dose PO q4-6h prn; not to exceed 2.6 g/d
>12 years: 325-650 mg PO q4h; not to exceed 4 g/d
Rifampin can reduce the analgesic effects; coadministration with barbiturates, carbamazepine, hydantoins, and isoniazid may increase hepatotoxicity.
Documented hypersensitivity; known G6PD deficiency
Pregnancy
B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals
Precautions
Hepatotoxicity is possible in people with long-term alcoholism following various dose levels; severe or recurrent pain or high or continued fever may indicate a serious illness; acetaminophen is contained in many OTC products, and combined use with these products may result in cumulative doses that exceed the recommended maximum dose.
Nonsteroidal Anti-inflammatory Drugs
NSAIDs have analgesic and antipyretic activities. The mechanism of action of these agents is not known, but NSAIDs may inhibit cyclooxygenase activity and prostaglandin synthesis. Other mechanisms may exist as well, such as inhibition of leukotriene synthesis, lysosomal enzyme release, lipoxygenase activity, neutrophil aggregation and various cell membrane functions. Treatment of pain tends to be patient specific.
Ibuprofen (Advil, Excedrin IB, Motrin, Ibuprin)
DOC for patients with mild to moderate pain. Inhibits inflammatory reactions and pain by decreasing prostaglandin synthesis.
Adult
200-400 mg PO q4-6h while symptoms persist; not to exceed 3.2 g/d
Pediatric
<6 months: Not established
6 months to 12 years: 4-10 mg/kg/dose PO tid/qid
>12 years: Administer as in adults
Coadministration with aspirin increases the risk of inducing serious NSAID-related adverse effects; probenecid may increase the concentrations and, possibly, the toxicity of NSAIDs; may decrease the effect of hydralazine, captopril, and beta-blockers; may decrease the diuretic effects of furosemide and thiazides; may increase PT duration when taking anticoagulants (instruct patients to watch for signs of bleeding); may increase the risk of methotrexate toxicity; phenytoin levels may be increased when administered concurrently
Documented hypersensitivity; peptic ulcer disease; recent GI bleeding or perforation; renal insufficiency; high risk of bleeding
Pregnancy
B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals
D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus
Precautions
Caution in patients with congestive heart failure, hypertension, and decreased renal and hepatic function; caution in the presence of coagulation abnormalities or during anticoagulant therapy
Ketoprofen (Actron, Orudis, Oruvail)
For the relief of mild to moderate pain and inflammation. Small initial dosages are indicated in small and elderly patients and in those with renal or liver disease.
Doses >75 mg do not increase therapeutic effects.
Administer high doses with caution and closely observe patient for response.
Adult
25-50 mg PO q6-8h prn; not to exceed 300 mg/d
Pediatric
<3 months: Not established
3 months to 12 years: 0.1-1 mg/kg PO q6-8h
>12 years: Administer as in adults
Coadministration with aspirin increases the risk of inducing serious NSAID-related adverse effects; probenecid may increase the concentrations and, possibly, the toxicity of NSAIDs; may decrease the effect of hydralazine, captopril, and beta-blockers; may decrease the diuretic effects of furosemide and thiazides; may increase PT duration when taking anticoagulants (instruct patients to watch for signs of bleeding); may increase the risk of methotrexate toxicity; phenytoin levels may be increased when administered concurrently
Documented hypersensitivity
Pregnancy
C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus
D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus
Precautions
Caution in patients with congestive heart failure, hypertension, and decreased renal and hepatic function; caution in the presence of coagulation abnormalities or during anticoagulant therapy
Naproxen (Aleve, Anaprox, Naprelan, Naprosyn)
For the relief of mild to moderate pain; inhibits inflammatory reactions and pain by decreasing the activity of cyclooxygenase, which is responsible for prostaglandin synthesis. NSAIDs decrease intraglomerular pressure and decrease proteinuria.
Adult
250-500 mg PO bid; may increase to 1.5 g/d for limited periods
Pediatric
<2 years: Not established
>2 years: 2.5 mg/kg/dose PO; not to exceed 10 mg/kg/d
Coadministration with aspirin increases the risk of inducing serious NSAID-related adverse effects; probenecid may increase the concentrations and, possibly, the toxicity of NSAIDs; may decrease the effect of hydralazine, captopril, and beta-blockers; may decrease the diuretic effects of furosemide and thiazides; may increase PT duration when taking anticoagulants (instruct patients to watch for signs of bleeding); may increase the risk of methotrexate toxicity; phenytoin levels may be increased when administered concurrently
Documented hypersensitivity; peptic ulcer disease; recent GI bleeding or perforation; renal insufficiency
Pregnancy
C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus
D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus
Precautions
Acute renal insufficiency, interstitial nephritis, hyperkalemia, hyponatremia, and renal papillary necrosis may occur; patients with preexisting renal disease or compromised renal perfusion risk acute renal failure; leukopenia occurs rarely, is transient, and usually returns to normal during therapy; persistent leukopenia, granulocytopenia, or thrombocytopenia warrants further evaluation and may require discontinuation of the drug.
More on Plantar Heel Pain |
| Overview: Plantar Heel Pain |
| Differential Diagnoses & Workup: Plantar Heel Pain |
Treatment & Medication: Plantar Heel Pain |
| Follow-up: Plantar Heel Pain |
| Multimedia: Plantar Heel Pain |
| References |
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References
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Further Reading
Keywords
heel pain syndrome, plantar fasciitis, proximal plantar fasciitis, heel spur, plantar heel pain, tarsal tunnel syndrome, fat pad atrophy, heel pain, foot pain, plantar fascia rupture, lateral plantar nerve compression, calcaneal stress fracture, stress fracture of the calcaneus, bone tumor, bone cyst, osteomyelitis, spinal stenosis, prolapsed intervertebral disk, prolapsed intervertebral disc, arthritic inflammatory bowel disease, seronegative spondyloarthropathy, inflammatory arthritis, rheumatoid arthritis
Treatment & Medication: Plantar Heel Pain