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Lumbar Spine Fractures and Dislocations Treatment & Management

  • Author: Federico C Vinas, MD; Chief Editor: Jeffrey A Goldstein, MD  more...
Updated: Oct 29, 2015

Approach Considerations

The decision to perform surgery in the acute setting is determined by the surgeon, depending on the stability of the fracture, the radiologic evidence of spinal cord or cauda equina compression, the patient's neurologic examination, and the overall status of the patient. In general, decompressive surgery is not indicated for patients with complete deficit lasting more than 48 hours and is advocated for patients with partial cord or cauda equina injuries.

The relation between the timing of surgical decompression and the neurologic outcome has been widely debated. Some evidence in the literature suggests that acute surgical therapy decreases the length of hospitalization and related costs, facilitating rehabilitation in many patients with spine injury.

Numerous factors must be considered in the selection of the surgical approach, including the degree of bone destruction, associated ligamentous injury, presence and degree of neurologic deficit, age and medical condition of the patient, and other associated injuries.

With regard to potential future therapeutic developments, the use of growth factors for the induction of spinal fusions is an attractive approach. Some studies have shown that viral vectors can be used to implant osteoinductive growth factor genes directly into the paraspinal muscles or into cells that can be subsequently implanted next to the spine. These osteoinductive factors enhance the activation and differentiation of pluripotent stem cells to produce mature bone.

Numerous studies support the view that morphogenic bone proteins (ie, bone morphogenetic protein type 2 [BMP-2] and other polypeptides, such as transforming growth factor beta), acidic and basic fibroblast growth factors, insulinlike growth factors, and platelet-derived growth factors are effective in promoting bone formation and fusion. Studies performed using BMP-2 have demonstrated the same rate of fusion reported in studies performed using autologous iliac crest bone graft, avoiding the morbidity of harvesting iliac crest bone.[2] However, the high cost of this therapy limits its widespread use.

Studies are being performed on a variety of proteins with a range of physiologic activities in the growth and development of numerous organ systems, including the heart, liver, skeleton, tendons, ligaments, and skin. Their use in humans is currently under investigation.


Medical Therapy

Initial management of lumbar spine injury begins in the field. Any patient in whom a spinal injury is suspected should be placed on a board in a neutral supine position and immobilized in a neck collar for expeditious transportation to a trauma center.[26]

In the emergency department, all patients should be treated as though they have a spinal injury until spinal injury can be ruled out. The Advanced Trauma Life Support (ATLS) guidelines of the American College of Surgeons should be followed. Stabilization of the patient's airway and hemodynamic status should precede any treatment in order to secure an adequate oxygenation and tissue perfusion. A Foley catheter should be inserted. In patients with neurologic deficit, immediate peritoneal lavage often is advocated to rule out intra-abdominal injuries.[27, 28]

Once the patient has been resuscitated, plain films of the cervical, thoracic, and lumbosacral spine should be obtained.

When possible, a detailed history should be obtained to ascertain the mechanism of injury and the relative force sustained. Individuals who sustain falls often have hyperflexion injuries at the thoracolumbar region in association with pelvic and lower-extremity fractures. Persons who wore seat belts during motor vehicle accidents often have distraction injuries or associated cervical spine injuries. In these patients, the vertebrae frequently are compressed or dislocated in the horizontal plane. Crawford et al[29] studied the surgical management of pediatric patients with seat-belt fracture-dislocations of the spine.

Head injuries and extremity fractures commonly accompany vertebral fractures. Abdominal or urological trauma can occur with lumbar fractures, particularly with motor vehicle–type injuries.[7, 30] The possible presence of concurrent direct injuries to adjacent intracavitary soft tissue structures (eg, renal, spleen, or liver lacerations) must be considered. In general, the more caudal the vertebral injury, the greater the biomechanical forces sustained and the greater the propensity for injuries to the pelvis and sacrum.

As early as possible and within 8 hours following injury, all patients with spinal cord injuries should receive intravenous (IV) methylprednisolone at 30 mg/kg in a bolus, followed by infusion at a rate of 5.4 mg/kg/hr for 23 hours. The results of a prospective trial demonstrated significantly better motor function and sensation at 6 months and 1 year in patients treated with this regimen compared with those given placebo.

The major goal of treatment in patients with disruption of the vertebral column who are neurologically intact is the prevention of neurologic deterioration. If a fracture is stable without nerve compression, surgical treatment may not be required. When a fracture is unstable or when neural compression is present, a decompressive procedure with a fusion, usually with instrumentation, becomes necessary. Stabilization is aimed at minimizing pain and subsequent spinal deformity.

Compression fractures have a disrupted anterior and intact middle column. Treatment of these injuries depends on the status of the posterior ligamentous structures, as well as on the integrity of the bony elements. Compression of more than 40% of the anterior vertebral wall or a kyphotic deformity of more than 25° is often associated with posterior ligamentous injury. If the kyphotic angulation is less than 25° and the anterior body compression is less than 40% of the vertebral height, the injury can be treated nonoperatively.

The patient is placed in a thoracolumbar orthosis (TLSO) with restriction of activities.[31] After 3-4 months in the orthosis, flexion extension radiographs should be obtained. If no motion is present and the deformity has not progressed, the patient can be weaned from the TLSO over several weeks and can start physical therapy for muscle strengthening. If abnormal motion is present, the deformity has progressed, or severe pain persists, surgical stabilization may be required. If the anterior column is compressed more than 40% or the kyphosis exceeds 25°, surgical stabilization is indicated.

In burst fractures, both the anterior and the middle column are disrupted[32, 33] ; the posterior column may or may not be affected. In burst fractures, it is important to analyze the percentage of canal compromise, the degree of angulation, and the neurologic status of the patient.[34] If the canal compromise is less than 40%, the patient may require a TLSO brace worn for at least 3 months. Standing lateral radiographs should be obtained on a regular basis to document any interval increase in spinal deformity.

If the canal compromise is more than 40%, the kyphotic deformity is more than 25°, or the patient develops neurologic changes (eg, changes in motor function or bladder control, new sensory deficits), surgical intervention may be required. Burst fractures can be reduced and stabilized from an anterior or a posterior approach.[35, 36] In patients with burst fractures and significant posterior column disruption, anterior and posterior fusion (360°) is indicated.[37, 38]

In patients with fracture-dislocation injuries, all three columns of the spine are disrupted. This type of injury carries a high incidence of spinal cord injury. In general, most fracture-dislocation injuries require surgical treatment. If a patient with a fracture-dislocation has normal neurologic examination findings, the spine must be stabilized to prevent a spinal cord, cauda equina, or nerve root injury.

When the patient has an incomplete spinal cord injury from a fracture-dislocation, the spinal canal should be decompressed and the spine stabilized to prevent neurologic deterioration. Stabilization of the spine in patients with a complete neurologic deficit from a fracture-dislocation may prevent progressive kyphotic deformation, allowing early mobilization and rehabilitation, thereby minimizing the hospital length of stay.


Surgical Therapy

Indications for surgery

Surgical intervention is often necessary for patients with unstable fractures or those with neurologic deficits related to compression of the neural structures by bony elements or hematomas, partial cord injuries, or cauda equina injuries. In patients with fractures and associated spinal cord injury, the efficacy of decompressive surgery varies according on the level and degree of injury.[39, 40]

In general, if a patient has a complete neurologic deficit (paraplegia or tetraplegia) and the neurologic examination findings do not improve within 48 hours, decompressive surgery is not indicated, because it will not produce an improvement in neurologic function.

Patients with cauda equina or incomplete cord lesions, however, have been shown to benefit from decompressive surgery even after long delays. Some studies have failed to demonstrate a correlation between the degree of canal compromise at the thoracolumbar junction and neurologic deficits. In contrast with patients with spinal cord injuries at the cervical and thoracic spine, patients with nerve root compression at the lumbosacral region often achieve better outcomes following surgical decompression.

The effect of the timing of decompressive surgery on the rate of neurologic recovery also has remained unclear.[41, 42] Improved neurologic function has been reported with early and late decompression. Most studies have reported on the neurologic recovery associated with late anterior decompression and have not directly analyzed the significance of the timing of surgery. Several studies showed that early spine fixation (≤48 hours) reduced morbidity and resource utilization.[43] There appears to be a trend toward early surgical intervention in patients with spinal instability or neurologic deficits resulting from compression of the neural structures.

Various operative techniques are used in the treatment of spinal trauma. The surgical approach used is determined by the following factors:

  • Level of injury
  • Characteristics of the fracture
  • Location of the neural compression

Modern surgical techniques allow effective decompression of the neural structures, via using microsurgical approaches. In patients with unstable fractures, the use of segmental instrumental fixation is often necessary in conjunction with a fusion of the spine, via either an anterior or posterior surgical approach. This allows reduction and stabilization of the injured segments. Regardless of the type of instrumentation and surgical approach, a fusion in conjunction with the segmental fixation is of paramount importance because any type of instrumentation will fail if the spine is not supported by a solid bony fusion.

Surgery is contraindicated in moribund patients in very poor medical condition.

Preparation for operation

The surgical procedure usually is performed with the patient under general anesthesia in a prone, lateral, or knee-elbow position. Once general anesthesia has been induced and the endotracheal tube secured, the patient's eyes should be well lubricated and taped shut. For a posterior approach, the author's preference is to perform the procedure with the patient under general anesthesia in the prone position over gel rolls that extend from the shoulders to the lower pelvis; alternatively, a Wilson frame may be used.

The use of a radiolucent Wilson frame on a Jackson table is recommended because it maintains the spine in lordosis and avoids increased intra-abdominal pressure. Increase in intra-abdominal pressure increases venous pressure to the epidural veins, resulting in increased epidural bleeding. The use of intraoperative spinal cord monitoring of the somatosensory evoked potential (SSEP) and electromyography (EMG) is recommended to minimize the risk of further neurologic deficits. The authors prefer the use of the operative microscope, which allows increased illumination and visibility.


Surgical decompression

The preferred posterior skin incision is a midline one. The length of the incision should permit complete visualization of the entire hemilamina rostral and caudad to the appropriate interlaminar space or spaces, as well as the entire facet complex.

The lumbodorsal fascia is incised with the electric knife just lateral to the spinous process and supraspinous ligament. Atraumatic dissection of the muscles off the spinous processes, laminae, and transverse processes is accomplished with a periosteal elevator and electric knife. After exposure of the appropriate interlaminar area, a self-retaining retractor is used to retract the musculofascial layers. Posterior element fractures usually are visualized at this time.

If compression of neural structures has been determined preoperatively, adequate bilateral exposure and decompression of ligaments and bone are then performed with a high-speed drill, rongeurs, and Kerrison punches and carried both rostrad and caudad well beyond the area of neurocompressive pathology. All residual ligamentum flavum is gently microdissected from the dura and nerve root sheath and excised with either a 2- or 3-mm thin-footplate Kerrison rongeur.

Once adequate dorsolateral decompression and exposure of the dural sac and involved nerve root have been accomplished, a generous foraminotomy is accomplished with the thin-plate Kerrison rongeur.

Posterior intertransverse fusion

The spine is exposed through a posterior midline incision and subperiosteal muscle dissection. The incision length must be sufficient to enable full exposure of the transverse processes. The dissection is carried out laterally over the facet joint to expose the transverse processes completely at the levels to be fused. All soft tissue is meticulously removed from the grafting area, including the transverse process, the lateral aspect of the superior facet joints, and the pars interarticularis.

The bone graft can be harvested either through the previously made midline incision or through the separately placed lateral incision. The superior and outer margins of the iliac crest are exposed subperiosteally. Multiple corticocancellous strips are harvested.

After adequate bone harvest, the donor bed is copiously irrigated with antibiotic solution and waxed to reduce blood loss. This wound is closed in layers. Decortication of the graft bed usually is performed with a high-speed drill. The harvested bone then is placed onto the recipient bone bed and packed into the facet joints. The wound is copiously irrigated and closed with an absorbable suture.

The intertransverse process region provides ample surface area for graft contact, which results in a high rate of fusion. Exposure of this region requires substantial paraspinal muscle dissection, which can be bloody and time-consuming. This technique does not decrease immediate motion, correct deformity, or maintain spinal alignment and generally is used in conjunction with pedicle screw placement.

Posterior interbody fusion

Lumbar interbody fusion remains a popular method of arthrodesis because it allows access to the anterior weightbearing spinal column through a standard posterior laminectomy.[44] This technique seems most ideally suited for cases of mechanical instability that require concomitant spinal canal or disk space entry for decompression. Patient position and initial spinal exposure are similar to those described for intertransverse process fusion. The dissection need only be carried out to the lateral aspect of the facet joints.

A standard bilateral laminectomy or bilateral hemilaminectomies are performed. Removal of the medial two thirds of the facet joints adequately exposes the disk space for graft placement. Epidural veins are cauterized and divided. A wide opening is made into the annulus, and the disk is removed. Sharp osteotomes and ring curets are used to remove the cartilaginous endplates. Bone grafts or implants filled with bone graft are impacted into the disk space and levered medially until 60-80% of the central disk space volume has been filled.

The use of this technique in the paramedial space is known as posterior lateral interbody fusion, whereas a more lateral approach is known as transpedicular interbody fusion.[45, 46]

A study by Baumann et al found that demineralized bone matrix was an acceptable alternative to autologous bone graft in posterolateral fusion performed to treat acute traumatic vertebral body fractures of the thoracolumbar spine.[47]

Anterior corpectomy and fusion

Various approaches to the anterior lumbar and lumbosacral spine have been described. Proper exposure of the anterior lumbar spine requires a detailed knowledge of the neurovascular soft tissue surrounding the anterior spine.

After incision of the abdominal wall musculature, the peritoneal sac is bluntly freed from its attachment to the transversalis fascia until the spine and psoas muscle are identified. The ureter usually remains attached to the posterior peritoneum and is elevated away from the spine during the dissection. It must be identified and protected before any sharp dissection is performed.

The aorta overlies the anterior aspect of the spine and bifurcates into common iliac arteries at about the L4-5 disk space. The inferior vena cava is dorsolateral to the aorta on the right side, and the left common iliac vein crosses the midline behind the iliac arteries and may partially overlie the L5-S1 disk space. Segmental arteries and veins run transversely at the midvertebral body level to enter the aorta and vena cava, respectively. These vessels must be suture-ligated to permit reflection of the great vessels for exposure of the spine. The lumbar sympathetic chain descends just medial to the psoas muscle and can be identified easily.

Once adequate exposure has been achieved, self-retaining retractors are used. Injury to the great vessels is a common complication of surgery. Therefore, these vessels must be adequately protected during this dissection.

Localization of the vertebral fracture is performed with a cross-table lateral radiograph. Once the correct level has been exposed, the superior and inferior disks are removed by using a long knife, rongeurs, curets, and osteotomes. The vertebral corpectomy is performed by using a high-speed drill, curettes, osteotomes, and rongeurs, with special care taken in approaching the spinal canal.

Once adequate decompression has been achieved, the cartilaginous endplates are removed down to bleeding subchondral bone. Spinal fixation is placed in the adjacent vertebral bodies, and gentle distraction of the corpectomy is achieved with a distractor.

Various donor bone sources are available. The authors prefer to use humerus, femur, or a tibial strut allograft. This can be combined with local autogenous bone from the corpectomy. Autogenous anterior iliac crest bone graft can also be used (see the image below).

A 37-year-old man who underwent an anterior approa A 37-year-old man who underwent an anterior approach for an unstable L1 burst fracture. A corpectomy was performed, with a vertebral reconstruction with Harms cages and a screw to stabilize the cage. The patient subsequently underwent a posterior arthrodesis with iliac crest bone graft and transpedicular screw placement.

The bone graft is carefully shaped to maximize bone contact area and is impacted into the space provided by the corpectomy. The distraction then is removed, further securing the bone graft. A titanium plate or rods are placed on the bolts, securing the graft in position. The retractors are removed, and the wound is then closed in layers.[44]

Reported fusion rates and clinical success with anterior interbody techniques are widely variable. Differences probably are related to surgical technique, the source of donor bone, patient selection, and the method by which determination of fusion was evaluated. Internal fixation and direct current electrical stimulation probably enhance fusion rates.[48]

Posterior internal fixation with pedicle screws

Internal fixation as an adjunct to spinal fusion has become increasingly popular. Titanium rods are longitudinally anchored to the spine by hooks or transpedicular screws. Powerful forces can be applied to the spine through these implants to correct deformity. Implants provide immediate rigid spinal immobilization, which allows early patient mobilization and affords a more optimal environment for bone graft incorporation. Numerous clinical and experimental studies demonstrate higher fusion rates in patients with rigid internal fixation than in controls without instrumentation. (See the image below.)

Postoperative lateral radiograph; although the pat Postoperative lateral radiograph; although the patient was paraplegic, in order to prevent severe kyphotic deformity of the spine and to allow a rapid mobilization, a posterior arthrodesis was performed with pedicle screws, hooks, and rods.

Although various implants are available, pedicle fixation systems are the most commonly used implant type in the lumbosacral spine. The large size of the lumbar pedicles minimizes the number of instrumented motion segments required to achieve adequate stabilization. The technique of pedicle fixation requires a thorough knowledge of the pedicle anatomy.[49]

Several techniques are available for screw placement, but the authors prefer an entry point into the pedicle at the intersection of the middle of the transverse process, the facet joint, and the pars.[50] Once a screw trajectory has been achieved within a pedicle finder, palpation of the cortical margins of the screw tract with a ball tip finder minimizes the penetration of the screw into the spinal canal. A tap is then used to create the threads for the screws.

Finally, the screws can be placed under continuous fluoroscopic guidance. A depth of 50-75% of the anteroposterior (AP) vertebral body diameter is usually recommended for lumbar fixation, while bicortical screw purchase is recommended for sacral fixation. The position of the screws is then assessed electrophysiologically with a nerve stimulator and radiologically with an AP, lateral, and two-dimensional scan of the spine performed with an isocentric C-arm.


Compression fractures with an intact posterior cortical wall can be treated by means of a kyphoplasty. This procedure involves transpedicular placement of a balloon through a bone biopsy needle and cannula into the compressed vertebral body under fluoroscopic guidance. The balloon is then inflated under controlled pressure, resulting in expansion of the vertebral body and creation of a cavity. This cavity is then filled with bone cement. This results in elevation of the endplate and stabilization of the fracture fragments, with a consequent reduction of pain.[51, 52] A posterior cortical defect in a burst fracture is considered a contraindication for kyphoplasty. (See the images below.)

Patients with compression fractures not compromisi Patients with compression fractures not compromising the spinal canal can be treated by means of kyphoplasty. Use of a percutaneous balloon allows for expansion of the fractured vertebrae. Then, the void created by the balloon is filled with bone cement.
Patients with an acute compression fracture treate Patients with an acute compression fracture treated with kyphoplasty. AP and lateral views demonstrate a good expansion of the compressed vertebral body and good filling with cement.
A 47-year-old man was involved in a motor vehicle A 47-year-old man was involved in a motor vehicle accident. He arrived at the hospital with paraplegia but preserved sensation in both lower extremities. He was immediately taken to surgery for an open reduction of the fracture, decompression of the cauda equina, and arthrodesis of the spine. He regained motor function following the surgery.

 For a comparison of vertebroplasty and kyphoplasty, see Hiwatashi et al[53] and Karlsson et al.[54]

Postoperative care

Significant postoperative discomfort limits activity for several days in most patients. A morphine patient-controlled analgesia pump usually is employed during the first 36-48 hours. A molded lumbar or thoracolumbar orthosis is often worn for 3 months.

One study evaluated the effects of two different doses of perioperative pregabalin administration in patients undergoing spinal fusion surgery.[55] The study found that pregabalin 150 mg, but not 75 mg, administered prior to and 12 hours after surgery significantly reduced the use of postoperative opioid consumption for 48 hours without significant side effects.

During the postoperative period, patients with fractures that have resulted in neurologic deficits are prone to multiple complications, including skin decubitus, pulmonary problems, and urinary sepsis (see below).

Nursing care should include frequent repositioning, vigorous pulmonary toilet, and deep venous thrombosis (DVT) prophylaxis. Intermittent pneumatic compression stockings are indicated for all patients with spinal injuries. If the patient is neurologically intact, pulsatile stockings alone suffice. However, if the patient has neurologic compromise, pulsatile stockings and low-dose subcutaneous heparin are used in combination to prevent DVT. If the patient is immobilized from multiple injuries, heparin should be started after postoperative day 2, even if he or she is neurologically intact.

Intermittent catheterization should be performed in patients with spinal cord injuries and urinary retention. A bowel regimen consisting of stool softeners and suppositories always should be instituted in these patients.



Patients with spinal cord injuries are prone to multiple complications, including decubitus ulcers, pulmonary problems, urinary sepsis, and new fractures[56] . Occasionally, patients develop delayed progressive neurologic deterioration months to years after sustaining spinal trauma as a result of instability and progressive spinal deformation.

Intraoperative complications

Neurologic deterioration can occur from neural traction, compression, or interruption of the vascular supply to the neural elements. The overall risk of neurologic injury from posterior instrumentation is 1-3%. In addition, postoperative neurologic deterioration may occur from graft dislodgment, displacement of the hardware, or hematomas.

Intraoperative injury to major vessels and viscera may occur during vertebral exposure and reconstruction. Dural tears, which may be the result of bone fragments or may occur during the surgical approach, can result in cerebrospinal fluid leaks.

Failure of the fusion

Pseudarthrosis is a cause of chronic pain as result of the malunion of the fusion. It may lead to progressive deformity, neural compromise, and pain. Failure of the instrumentation, such as dislodgment or breakage, is usually related to pseudarthrosis.


Infections can occur after spine surgery, especially after a long surgical procedure for a complicated instrumentation placement. Superficial infections should be opened and debrided. The wound may be packed open or closed using retention sutures. Appropriate antibiotics should be employed, starting with coverage against gram-positive cocci and adjusting in accordance with culture results. All attempts should be made to keep the instrumentation and graft in place until the fusion is solid.

Thromboembolic disease

DVT is a significant potential complication in patients with spinal fractures. Thromboembolism has been reported to occur in as many as 70% of patients with complete motor paralysis. Pulmonary embolism (PE) significantly affects the probability of survival after a spinal fracture. Mortality figures for patients with PE have not decreased significantly in the last 30 years, emphasizing the need for more effective preventive measures.

Recommendations for prophylaxis are varied and usually include subcutaneous or low-molecular-weight heparin, sequential compression stockings, and elastic hose placed on the lower extremities. Patients who develop DVT should be treated aggressively with anticoagulation. If the risk for systemic anticoagulation is prohibitive, thrombectomy or placement of a vena caval filter is an option.

Stress ulcers

The stress resulting from a traumatic injury, a complicated surgery, and mechanical ventilation can predispose a patient to gastric ulcers. However, the widespread use of prophylactic agents, such as H2-receptor blockers, sucralfate, and proton pump inhibitors, has reduced the incidence of severe bleeding from stress ulcers.

Adynamic ileus and Ogilvie syndrome

Ogilvie syndrome, also known as pseudo-obstruction of the colon, is characterized by massive abdominal distention with a cecal diameter of more than 9 cm. Nausea, vomiting, diarrhea, and severe abdominal distention are common symptoms.

Preventive measures for both Ogilvie syndrome and adynamic ileus include minimizing bed rest, returning to ambulation as early as possible, and limiting the use of narcotics. Early recognition and treatment of these conditions are essential to reduce morbidity and mortality. Initial treatment includes cessation of oral intake, nasogastric suction, insertion of rectal tubes, and cessation of narcotics.

Genitourinary complications

Urinary complications continue to be significant sources of morbidity after spinal injuries. In patients with spinal cord injuries, distention of the bladder can lead to autonomic dysreflexia, impairment of bladder sensation, detrusor hyperreflexia, and sphincter dyssynergia, which can lead to renal damage from hydronephrosis or vesicourethral reflux. These complications are decreased with indwelling Foley catheters. In patients with spinal cord injury, the most frequent source of morbidity is sepsis related to urinary tract infections.


Long-Term Monitoring

The fusion is evaluated by means of plain radiography, including flexion and extension views, at 6 weeks, 3 months, and 6 months postoperatively. If there is any doubt, computed tomography (CT) is performed.

Contributor Information and Disclosures

Federico C Vinas, MD Consulting Neurosurgeon, Department of Neurological Surgery, Halifax Medical Center

Federico C Vinas, MD is a member of the following medical societies: American Association of Neurological Surgeons, American College of Surgeons, American Medical Association, Florida Medical Association, North American Spine Society, Congress of Neurological Surgeons

Disclosure: Nothing to disclose.

Specialty Editor Board

Francisco Talavera, PharmD, PhD Adjunct Assistant Professor, University of Nebraska Medical Center College of Pharmacy; Editor-in-Chief, Medscape Drug Reference

Disclosure: Received salary from Medscape for employment. for: Medscape.

William O Shaffer, MD Orthopedic Spine Surgeon, Northwest Iowa Bone, Joint, and Sports Surgeons

William O Shaffer, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, American Orthopaedic Association, Kentucky Medical Association, North American Spine Society, Kentucky Orthopaedic Society, International Society for the Study of the Lumbar Spine, Southern Medical Association, Southern Orthopaedic Association

Disclosure: Received royalty from DePuySpine 1997-2007 (not presently) for consulting; Received grant/research funds from DePuySpine 2002-2007 (closed) for sacropelvic instrumentation biomechanical study; Received grant/research funds from DePuyBiologics 2005-2008 (closed) for healos study just closed; Received consulting fee from DePuySpine 2009 for design of offset modification of expedium.

Chief Editor

Jeffrey A Goldstein, MD Clinical Professor of Orthopedic Surgery, New York University School of Medicine; Director of Spine Service, Director of Spine Fellowship, Department of Orthopedic Surgery, NYU Hospital for Joint Diseases, NYU Langone Medical Center

Jeffrey A Goldstein, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, American College of Surgeons, American Orthopaedic Association, North American Spine Society, Scoliosis Research Society, Cervical Spine Research Society, International Society for the Study of the Lumbar Spine, AOSpine, Society of Lateral Access Surgery, International Society for the Advancement of Spine Surgery, Lumbar Spine Research Society

Disclosure: Received consulting fee from Medtronic for consulting; Received consulting fee from NuVasive for consulting; Received royalty from Nuvasive for consulting; Received consulting fee from K2M for consulting; Received ownership interest from NuVasive for none.

Additional Contributors

Lee H Riley III, MD Chief, Division of Orthopedic Spine Surgery, Associate Professor, Departments of Orthopedic Surgery and Neurosurgery, Johns Hopkins University School of Medicine

Disclosure: Nothing to disclose.

  1. Fontaine MA, Albert A, Dubois B, et al. Fracture and bone mineral density in hemodialysis patients. Clin Nephrol. 2000 Sep. 54(3):218-26. [Medline].

  2. Ichikawa S, Johnson ML, Koller DL, et al. Polymorphisms in the bone morphogenetic protein 2 (BMP2) gene do not affect bone mineral density in white men or women. Osteoporos Int. 2006 Jan 24. 1-6. [Medline].

  3. Alanay A. Re: post-traumatic findings of the spine after earlier vertebral fracture in young patients. Spine. 2000 Nov 1. 25(21):2847-8. [Medline].

  4. Dogan S, Safavi-Abbasi S, Theodore N, Chang SW, Horn EM, Mariwalla NR, et al. Thoracolumbar and sacral spinal injuries in children and adolescents: a review of 89 cases. J Neurosurg. 2007 Jun. 106(6 Suppl):426-33. [Medline].

  5. El Assuity WI, El Masry MA, Chan D. Acute traumatic spondylolisthesis at the lumbosacral junction. J Trauma. 2007 Jun. 62(6):1514-6; discussion 1516-7. [Medline].

  6. Smith JA, Siegel JH, Siddiqi SQ. Spine and spinal cord injury in motor vehicle crashes: a function of change in velocity and energy dissipation on impact with respect to the direction of crash. J Trauma. 2005 Jul. 59(1):117-31. [Medline].

  7. Beaunoyer M, St-Vil D, Lallier M, Blanchard H. Abdominal injuries associated with thoraco-lumbar fractures after motor vehicle collision. J Pediatr Surg. 2001 May. 36(5):760-2. [Medline].

  8. le Roux JC, Dunn RN. Gunshot injuries of the spine--a review of 49 cases managed at the Groote Schuur Acute Spinal Cord Injury Unit. S Afr J Surg. 2005 Nov. 43(4):165-8. [Medline].

  9. Franz T, Hasler RM, Benneker L, Zimmermann H, Siebenrock KA, Exadaktylos AK. Severe spinal injuries in alpine skiing and snowboarding: a 6-year review of a tertiary trauma centre for the Bernese Alps ski resorts, Switzerland. Br J Sports Med. 2008 Jan. 42(1):55-8. [Medline].

  10. Sieradzki JP, Sarwark JF. Thoracolumbar fracture-dislocation in child abuse: case report, closed reduction technique and review of the literature. Pediatr Neurosurg. 2008. 44(3):253-7. [Medline].

  11. Schoenfeld AJ, Newcomb RL, Pallis MP, Cleveland AW 3rd, Serrano JA, Bader JO, et al. Characterization of spinal injuries sustained by American service members killed in Iraq and Afghanistan: a study of 2,089 instances of spine trauma. J Trauma Acute Care Surg. 2013 Apr. 74(4):1112-8. [Medline].

  12. Patten RM, Gunberg SR, Brandenburger DK. Frequency and importance of transverse process fractures in the lumbar vertebrae at helical abdominal CT in patients with trauma. Radiology. 2000 Jun. 215(3):831-4. [Medline].

  13. Hsieh CT, Chen GJ, Wu CC, Su YH. Complete fracture-dislocation of the thoracolumbar spine without paraplegia. Am J Emerg Med. 2008 Jun. 26(5):633.e5-7. [Medline].

  14. Wood KB, Li W, Lebl DR, Ploumis A. Management of thoracolumbar spine fractures. Spine J. 2014 Jan. 14 (1):145-64. [Medline].

  15. Levi AD, Hurlbert RJ, Anderson P, et al. Neurologic deterioration secondary to unrecognized spinal instability following trauma--a multicenter study. Spine. 2006 Feb 15. 31(4):451-8. [Medline].

  16. Kinoshita T, Ebara S, Kamimura M, et al. Nontraumatic lumbar vertebral compression fracture as a risk factor for femoral neck fractures in involutional osteoporotic patients. J Bone Miner Metab. 1999. 17(3):201-5. [Medline].

  17. Castaño-Betancourt MC, Oei L, Rivadeneira F, de Schepper EI, Hofman A, Bierma-Zeinstra S, et al. Association of lumbar disc degeneration with osteoporotic fractures; the Rotterdam study and meta-analysis from systematic review. Bone. 2013 Aug 17. [Medline].

  18. Kudlacek S, Schneider B, Resch H, et al. Gender differences in fracture risk and bone mineral density. Maturitas. 2000 Oct 31. 36(3):173-80. [Medline].

  19. Wu CT, Lee SC, Lee ST, Chen JF. Classification of symptomatic osteoporotic compression fractures of the thoracic and lumbar spine. J Clin Neurosci. 2006 Jan. 13(1):31-8. [Medline].

  20. van der Roer N, de Bruyne MC, Bakker FC, et al. Direct medical costs of traumatic thoracolumbar spine fractures. Acta Orthop. 2005 Oct. 76(5):662-6. [Medline].

  21. Heyde CE, Ertel W, Kayser R. [Management of spine injuries in polytraumatized patients]. Orthopade. 2005 Sep. 34(9):889-905. [Medline].

  22. Vaccaro AR, Oner C, Kepler CK, Dvorak M, Schnake K, Bellabarba C, et al. AOSpine thoracolumbar spine injury classification system: fracture description, neurological status, and key modifiers. Spine (Phila Pa 1976). 2013 Nov 1. 38 (23):2028-37. [Medline].

  23. Nelson DW, Martin MJ, Martin ND, Beekley A. Evaluation of the risk of noncontiguous fractures of the spine in blunt trauma. J Trauma Acute Care Surg. 2013 Jul. 75(1):135-9. [Medline].

  24. Dosch JC, Moser T, Dupuis MG, Dietemann JL. [How to read radiography of the traumatic spine?]. J Radiol. 2007 May. 88(5 Pt 2):802-16. [Medline].

  25. Groves CJ, Cassar-Pullicino VN, Tins BJ, et al. Chance-type flexion-distraction injuries in the thoracolumbar spine: MR imaging characteristics. Radiology. 2005 Aug. 236(2):601-8. [Medline].

  26. Karaikovic EE, Pacheco HO. Treatment options for thoracolumbar spine fractures. Bosn J Basic Med Sci. 2005 May. 5(2):20-6. [Medline].

  27. Inaba K, Kirkpatrick AW, Finkelstein J, et al. Blunt abdominal aortic trauma in association with thoracolumbar spine fractures. Injury. 2001 Apr. 32(3):201-7. [Medline].

  28. Tyroch AH, McGuire EL, McLean SF, et al. The association between Chance fractures and intra-abdominal injuries revisited: a multicenter review. Am Surg. 2005 May. 71(5):434-8. [Medline].

  29. Crawford CH 3rd, Puno RM, Campbell MJ, Carreon LY. Surgical management of severely displaced pediatric seat-belt fracture-dislocations of the lumbar spine associated with occlusion of the abdominal aorta and avulsion of the cauda equina: a report of two cases. Spine. 2008 May 1. 33(10):E325-8. [Medline].

  30. Ball ST, Vaccaro AR, Albert TJ, Cotler JM. Injuries of the thoracolumbar spine associated with restraint use in head-on motor vehicle accidents. J Spinal Disord. 2000 Aug. 13(4):297-304. [Medline].

  31. Ohana N, Sheinis D, Rath E, et al. Is there a need for lumbar orthosis in mild compression fractures of the thoracolumbar spine? A retrospective study comparing the radiographic results between early ambulation with and without lumbar orthosis. J Spinal Disord. 2000 Aug. 13(4):305-8. [Medline].

  32. Al-Khalifa FK, Adjei N, Yee AJ, Finkelstein JA. Patterns of collapse in thoracolumbar burst fractures. J Spinal Disord Tech. 2005 Oct. 18(5):410-2. [Medline].

  33. Lalonde F, Letts M, Yang JP, Thomas K. An analysis of burst fractures of the spine in adolescents. Am J Orthop. 2001 Feb. 30(2):115-20. [Medline].

  34. Sanderson PL, Fraser RD, Hall DJ, et al. Short segment fixation of thoracolumbar burst fractures without fusion. Eur Spine J. 1999. 8(6):495-500. [Medline].

  35. Blanco JF, De Pedro JA, Hernández PJ, et al. Conservative management of burst fractures of the fifth lumbar vertebra. J Spinal Disord Tech. 2005 Jun. 18(3):229-31. [Medline].

  36. Carl AL, Matsumoto M, Whalen JT. Anterior dural laceration caused by thoracolumbar and lumbar burst fractures. J Spinal Disord. 2000 Oct. 13(5):399-403. [Medline].

  37. Dai LY. Remodeling of the spinal canal after thoracolumbar burst fractures. Clin Orthop. 2001 Jan. (382):119-23. [Medline].

  38. Razak M, Mahmud MM, Hyzan MY, Omar A. Short segment posterior instrumentation, reduction and fusion of unstable thoracolumbar burst fractures--a review of 26 cases. Med J Malaysia. 2000 Sep. 55 Suppl C:9-13. [Medline].

  39. Marczynski W, Kroczak S, Baranski M. Fractures of thoracic and lumbar spine; treatment and follow up. Ann Transplant. 1999. 4(3-4):46-8. [Medline].

  40. Woolard A, Oussedik S. Injuries to the lumbar spine: identification and management. Hosp Med. 2005 Jul. 66(7):384-8. [Medline].

  41. Berry GE, Adams S, Harris MB, et al. Are plain radiographs of the spine necessary during evaluation after blunt trauma? Accuracy of screening torso computed tomography in thoracic/lumbar spine fracture diagnosis. J Trauma. 2005 Dec. 59(6):1410-3; discussion 1413. [Medline].

  42. Croce MA, Bee TK, Pritchard E, et al. Does optimal timing for spine fracture fixation exist?. Ann Surg. 2001 Jun. 233(6):851-8. [Medline].

  43. Kerwin AJ, Frykberg ER, Schinco MA, Griffen MM, Arce CA, Nguyen TQ, et al. The effect of early surgical treatment of traumatic spine injuries on patient mortality. J Trauma. 2007 Dec. 63(6):1308-13. [Medline].

  44. Elias WJ, Simmons NE, Kaptain GJ, et al. Complications of posterior lumbar interbody fusion when using a titanium threaded cage device. J Neurosurg. 2000 Jul. 93(1 Suppl):45-52. [Medline].

  45. Godlewski P, Mazurkiewicz T. [Use of transpedicular fixation in treatment of thoraco-lumbar spinal injuries]. Neurol Neurochir Pol. 2000 Nov-Dec. 34(6):1187-95. [Medline].

  46. Knop C, Fabian HF, Bastian L, Blauth M. Late results of thoracolumbar fractures after posterior instrumentation and transpedicular bone grafting. Spine. 2001 Jan 1. 26(1):88-99. [Medline].

  47. Baumann F, Krutsch W, Pfeifer C, Neumann C, Nerlich M, Loibl M. Posterolateral Fusion in Acute Traumatic Thoracolumbar Fractures: A Comparison of Demineralized Bone Matrix and Autologous Bone Graft. Acta Chir Orthop Traumatol Cech. 2015. 82 (2):119-125. [Medline].

  48. Oishi M, Onesti ST. Electrical bone graft stimulation for spinal fusion: a review. Neurosurgery. 2000 Nov. 47(5):1041-55; discussion 1055-6. [Medline].

  49. Tezeren G, Kuru I. Posterior fixation of thoracolumbar burst fracture: short-segment pedicle fixation versus long-segment instrumentation. J Spinal Disord Tech. 2005 Dec. 18(6):485-8. [Medline].

  50. Kim TK, Kim KH, Kim CH, et al. Percutaneous vertebroplasty and facet joint block. J Korean Med Sci. 2005 Dec. 20(6):1023-8. [Medline].

  51. Pradhan BB, Bae HW, Kropf MA, et al. Kyphoplasty reduction of osteoporotic vertebral compression fractures: correction of local kyphosis versus overall sagittal alignment. Spine. 2006 Feb 15. 31(4):435-41. [Medline].

  52. Verlaan JJ, van de Kraats EB, Oner FC, et al. The reduction of endplate fractures during balloon vertebroplasty: a detailed radiological analysis of the treatment of burst fractures using pedicle screws, balloon vertebroplasty, and calcium phosphate cement. Spine. 2005 Aug 15. 30(16):1840-5. [Medline].

  53. Hiwatashi A, Sidhu R, Lee RK, et al. Kyphoplasty versus vertebroplasty to increase vertebral body height: a cadaveric study. Radiology. 2005 Dec. 237(3):1115-9. [Medline].

  54. Karlsson MK, Hasserius R, Gerdhem P, et al. Vertebroplasty and kyphoplasty: New treatment strategies for fractures in the osteoporotic spine. Acta Orthop. 2005 Oct. 76(5):620-7. [Medline].

  55. Kim JC, Choi YS, Kim KN, Shim JK, Lee JY, Kwak YL. Effective dose of peri-operative oral pregabalin as an adjunct to multimodal analgesic regimen in lumbar spinal fusion surgery. Spine (Phila Pa 1976). 2011 Mar 15. 36(6):428-33. [Medline].

  56. Trout AT, Kallmes DF, Kaufmann TJ. New fractures after vertebroplasty: adjacent fractures occur significantly sooner. AJNR Am J Neuroradiol. 2006 Jan. 27(1):217-23. [Medline].

A 42-year-old man fell from a tree. He arrived at the hospital with a complete paraplegia. Plain radiographs reveal a fracture of L2 with L2-L3 subluxation.
CT scan of a 42-year-old man who fell from a tree. He arrived at the hospital with a complete paraplegia (same patient as in Image above). Note the large amount of bone retropulsed inside the spinal canal.
CT scan showing a burst of the L2 vertebral body.
Postoperative lateral radiograph; although the patient was paraplegic, in order to prevent severe kyphotic deformity of the spine and to allow a rapid mobilization, a posterior arthrodesis was performed with pedicle screws, hooks, and rods.
A 37-year-old man who underwent an anterior approach for an unstable L1 burst fracture. A corpectomy was performed, with a vertebral reconstruction with Harms cages and a screw to stabilize the cage. The patient subsequently underwent a posterior arthrodesis with iliac crest bone graft and transpedicular screw placement.
A 52-year-old man was involved in a severe motor vehicle collision. He arrived at the hospital with severe pain but no neurologic deficit. Lateral plain radiographs show a fracture at T12.
Sagittal T2-weighted image of a 52-year-old man who was involved in a severe motor vehicle collision. He arrived at the hospital with severe pain but no neurologic deficit (same patient as in the previous image). Image reveals a significant mass effect within the spinal canal.
Patients with compression fractures not compromising the spinal canal can be treated by means of kyphoplasty. Use of a percutaneous balloon allows for expansion of the fractured vertebrae. Then, the void created by the balloon is filled with bone cement.
Patients with an acute compression fracture treated with kyphoplasty. AP and lateral views demonstrate a good expansion of the compressed vertebral body and good filling with cement.
A 47-year-old man was involved in a motor vehicle accident. He arrived at the hospital with paraplegia but preserved sensation in both lower extremities. He was immediately taken to surgery for an open reduction of the fracture, decompression of the cauda equina, and arthrodesis of the spine. He regained motor function following the surgery.
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