Osgood-Schlatter Disease Treatment & Management

  • Author: J Andy Sullivan, MD; Chief Editor: Craig C Young, MD   more...
 
Updated: Dec 1, 2011
 

Medical Care

Therapy for Osgood-Schlatter disease (OSD) is conservative. Initial treatment includes the application of ice for 20 minutes every 2-4 hours.

Analgesics and nonsteroidal anti-inflammatory drugs (NSAIDs) may be given for pain relief and reduction of local inflammation. However, NSAIDs have not been shown to shorten the course of OSD.

Steroid injections should not be used.

Inform the patient to avoid pain-producing activities (eg, sports that involve excess amounts of jumping).

Use of a knee immobilizer for a few days may improve compliance, especially in more severe cases. Pads or braces also can be used for support.

Once the acute symptoms have abated, quadriceps-stretching exercises, including hip extension for a complete stretch of the extensor mechanism, may be performed to reduce tension on the tibial tubercle. Stretching exercises for the hamstrings, which are commonly tight, may also be performed.

Other than the presence of an ossicle that causes pain with kneeling, there are no long-term disabilities or problems associated with this condition.

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Surgical Intervention

If conservative treatment fails, surgical excision of the united painful ossicle is recommended. Removal of ossicle fragmentation in immature patients with an unfused apophysis should be approached with caution, as a resultant recurvatum deformity may occur due to premature fusion of the tibial tubercle.[3]

Tibial tubercle avulsions occasionally can occur due to the contracture of the extensor mechanism. Open reduction and internal fixation (ORIF) usually is recommended, depending on the size and displacement of the fragment as well as the phase of apophyseal closure.

In a study of the surgical treatment of unresolved Osgood-Schlatter disease (OSD), Pihlajamäki et al concluded that in most young adults, good to excellent functional outcomes can be achieved with surgical treatment of unresolved OSD.[12] The investigators examined postsurgical clinical courses, radiographic characteristics, and long-term outcomes of 107 military recruits (117 knees) who were operated on for the condition. Functional outcome data were gathered from medical records, interviews, questionnaires, and physical and radiographic examinations. By the end of a (median) 10-year follow-up period, 93 patients (87%) reported that they could participate without restriction in daily and work activities, and 80 patients (75%) had regained their preoperative sports activity level. In addition, 41 patients (38%) reported the ability to kneel without pain. Minor postoperative complications occurred in 6 patients, and 2 patients required reoperation forOSD.

In a review of a series of patients who were treated operatively, Binazzi et al found that the most widely used procedure was excision of all intratendinous ossicles, with or without removal of a portion of the prominent tibial tubercle.[13] A comparison of 2 groups of individuals, 1 with 15 individuals treated with excision of ossicles and 1 with 11 individuals treated with various methods before 1975, clearly showed that results of simple excision of the ossicles were better.

In another study, patients treated operatively were found to be no more likely than conservatively treated patients to be relieved of pain or to have improvement of cosmetic appearance.

Indications for surgery

Surgery to treat OSD is rarely indicated. Occasionally, adults have a large ossicle and an overlying bursa, which may cause pain with kneeling. If so, treatment consists of excision of the bursa, ossicle, and any prominence.[14] Surgical treatment is rarely, if ever, indicated in children.

Contraindications for surgery

The real question is whether or not surgery is ever indicated in the growing child, as OSD is self-limiting. Trail reviewed 2 groups of symptomatic patients with this condition with 4-5 years of follow-up.[15] One group was treated surgically with tibial sequestrectomy, and the other was managed conservatively. Surgery was found to offer no significant benefit over conservative care. In addition, a significant complication rate was identified with tibial sequestrectomy.

Complications

Following resection of an ossicle, complications include continued pain and poor cosmetic appearance. In the study by Trail, 55% of patients had an obvious bony prominence postoperatively. One third of these prominences were quite marked and troublesome, and 3 required a subsequent shaving.[15] One patient lost 10° of flexion, and another patient had 10° of recurvatum. Other complications that may occur include dehiscence, unsightly scar, anesthesia lateral to the scar, and continued presence of sequestra.

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Approach Considerations

The American Academy of Orthopaedic Surgeons and the American Academy of Family Practice recommend activity limitation, ice, anti-inflammatories, protective padding, quadriceps/hamstring strengthening, and time in the management of Osgood-Schlatter disease (OSD). (See the following Web sites for additional details: (1) American Association of Orthopaedic Surgeons Online Service Fact Sheet, Osgood-Schlatter Disease [Knee Pain], and (2) familydoctor.org, Osgood-Schlatter Disease: A Cause of Knee Pain in Children.)

However, no prospective, interventional studies evaluating the treatment of OSD, including the recommended conservative treatments, are available.

Corticosteroid injections are not recommended because of case reports of complications, primarily related to subcutaneous atrophy.

Long-term immobilization is typically contraindicated, because it may result in increased knee stiffness in mild cases, thus predisposing the athlete to additional sports-related injuries.

However, if a patient is noncompliant, the clinician may recommend immobilization in a knee brace for a minimum of 6 weeks. The brace should be removed daily, but only for stretching and strengthening exercises.

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Physical Therapy

The goal of rehabilitation is for the athlete to be able to return to his or her sport as quickly and safely as possible. The physical therapist and the physician determine when the athlete is ready to resume competition, depending on the findings of the clinical examination and functional testing. The pain may take up to 6-24 months to resolve. If an individual returns to activity too soon, he or she may worsen the condition. Athletes need to work on improving the flexibility and strength of the quadriceps and hamstring muscles throughout the course of rehabilitation to ensure that they are ready to return to sports.

Acute phase

Several techniques may be recommended by the physical therapist to alleviate discomfort and avert recurrence of the disease. Treatment recommendations are dependent upon the severity of the condition.

An infrapatellar strap may be recommended during sports activity.

Resting is recommended when pain arises.

Ice should be applied to the area for 20 minutes following activity.

Stretching of the quadriceps and hamstring muscles helps to prevent the development of OSD.

Short-term rest and knee immobilization may be required.

Knee braces are used for long-term immobilization (6 wk) in severe cases (eg, pain persists longer than 24 h following sports activity and/or limits daily activity) or for noncompliant patients with increasing symptoms.

Recovery phase

The following regimen recommendations for patients with OSD are taken from Meisterling, Wall, and Meisterling.

Straight leg raises can be performed as follows:

  • Lie on the floor with the back propped up a few inches with the elbows
  • Bend the unaffected knee to a comfortable position; using adjustable ankle weights with half-pound increments, determine the weight at which 10 raises can be performed on the affected leg
  • Tighten the thigh muscles and lift the affected leg 12 inches, keeping the leg straight
  • Hold for 5 seconds
  • Slowly lower the leg and relax
  • Start with 10 repetitions for each leg
  • When 15 repetitions have been performed comfortably, increase the weight by half a pound and drop back to 10 repetitions
  • Once 15 repetitions again can be performed comfortably, increase the weight again, to a maximum of 7-12 lb

Short-arc quadriceps exercises can be performed as follows:

  • Lie back with the unaffected knee bent (same as for straight leg raises)
  • Place a few rolled up towels under the affected knee to raise it 6 inches from the floor
  • Tighten the thigh muscles and straighten the leg until it is 12 inches from the floor
  • Hold for 5 seconds
  • Slowly lower the leg and relax
  • Start with 10 repetitions for each leg and increase to 15, using the same ankle weight and repetition progression as for straight leg raises

Wall slides can be performed as follows:

  • To do wall slides or quarter seats, stand about 12 inches from a smooth wall and lean back against it with the feet shoulder width apart
  • Holding a light dumbbell in each hand with the arms straight down, bend the knees and slowly lower the body 4-6 inches
  • If pain is felt, the body has squatted too far
  • Hold for 5 seconds and then rise up quickly
  • Start with 10 repetitions and increase to 15, gradually increasing the dumbbell weight in the same type of progression as for straight leg raises

A good rule of thumb with regard to squats and wall slides for patients with patellar pain of any kind is a relative restriction of not flexing the knee beyond 90°.

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Contributor Information and Disclosures
Author

J Andy Sullivan, MD  Clinical Professor of Pediatric Orthopedics, Department of Orthopedic Surgery, University of Oklahoma College of Medicine

J Andy Sullivan, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, American Medical Association, Oklahoma State Medical Association, and Pediatric Orthopaedic Society of North America

Disclosure: Nothing to disclose.

Chief Editor

Craig C Young, MD  Professor, Departments of Orthopedic Surgery and Community and Family Medicine, Medical Director of Sports Medicine, Director of Primary Care Sports Medicine Fellowship, Medical College of Wisconsin

Craig C Young, MD is a member of the following medical societies: American Academy of Family Physicians, American College of Sports Medicine, American Medical Society for Sports Medicine, and Phi Beta Kappa

Disclosure: Nothing to disclose.

Additional Contributors

Andrew K Chang, MD Associate Professor, Department of Emergency Medicine, Albert Einstein College of Medicine, Montefiore Medical Center

Andrew K Chang, MD is a member of the following medical societies: American Academy of Emergency Medicine, American Academy of Neurology, American College of Emergency Physicians, and Society for Academic Emergency Medicine

Disclosure: Nothing to disclose.

Thomas M DeBerardino, MD Associate Professor, Department of Orthopedic Surgery, Consulting Surgeon, Sports Medicine, Arthroscopy and Reconstruction of the Knee, Hip and Shoulder, Team Physician, Orthopedic Consultant to UConn Department of Athletics, University of Connecticut Health Center

Thomas M DeBerardino, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, American Orthopaedic Association, and American Orthopaedic Society for Sports Medicine

Disclosure: Arthrex, Inc. Grant/research funds Other; Arthrex, Inc. Consulting fee Speaking and teaching; Genzyme Biosurgery. Inc. Grant/research funds Other; Musculoskeletal Transplant Foundation Grant/research funds Other; Histogenics Grant/research funds None

Janos P Ertl, MD Assistant Professor, Department of Orthopedic Surgery, Indiana University School of Medicine; Chief of Orthopedic Surgery, Wishard Hospital

Janos P Ertl, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, American Orthopaedic Association, Hungarian Medical Association of America, and Sierra Sacramento Valley Medical Society

Disclosure: Nothing to disclose.

Gyorgy Kovacs, MD Consulting Surgeon, Department of Orthopedic Surgery, GOC Clinic

Disclosure: Nothing to disclose.

David B Levy, DO, FACEP, FAAEM Chairman, Department of Emergency Medicine, St Elizabeth Health Center; Associate Professor of Emergency Medicine, Northeastern Ohio Universities College of Medicine

David B Levy, DO, FACEP, FAAEM is a member of the following medical societies: American Academy of Emergency Medicine, American Medical Informatics Association, and Society for Academic Emergency Medicine

Disclosure: Nothing to disclose.

Munisha Mehra Bhatia, MD General Academic Pediatrics, Faculty Development Fellow, Children's Memorial Hospital of Northwestern University

Munisha Mehra Bhatia, MD is a member of the following medical societies: Academic Pediatric Association and American Academy of Pediatrics

Disclosure: Nothing to disclose.

Albert W Pearsall IV, MD Associate Professor, Department of Orthopedic Surgery, University of South Alabama College of Medicine; Director, Section of Sports Medicine and Shoulder Service, Department of Orthopedic Surgery, University of South Alabama Medical Center

Disclosure: Nothing to disclose.

Andrew L Sherman, MD, MS Associate Professor of Clinical Rehabilitation Medicine, Vice Chairman, Chief of Spine and Musculoskeletal Services, Program Director, SCI Fellowship and PMR Residency Programs, Department of Rehabilitation Medicine, University of Miami, Leonard A Miller School of Medicine

Andrew L Sherman, MD, MS is a member of the following medical societies: American Academy of Physical Medicine and Rehabilitation, American Association of Neuromuscular and Electrodiagnostic Medicine, American Medical Association, and Association of Academic Physiatrists

Disclosure: Pfizer Honoraria Speaking and teaching

Mark S Slabinski, MD, FACEP, FAAEM Vice President, EMP Medical Group

Mark S Slabinski, MD, FACEP, FAAEM is a member of the following medical societies: Alpha Omega Alpha, American Academy of Emergency Medicine, American College of Emergency Physicians, American Medical Association, and Ohio State Medical Association

Disclosure: Nothing to disclose.

Francisco Talavera, PharmD, PhD Adjunct Assistant Professor, University of Nebraska Medical Center College of Pharmacy; Editor-in-Chief, Medscape Drug Reference

Disclosure: Medscape Salary Employment

References
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  2. Krause BL, Williams JP, Catterall A. Natural history of Osgood-Schlatter disease. J Pediatr Orthop. Jan-Feb 1990;10(1):65-8. [Medline].

  3. Weiss JM, Jordan SS, Andersen JS, Lee BM, Kocher M. Surgical treatment of unresolved Osgood-Schlatter disease: ossicle resection with tibial tubercleplasty. J Pediatr Orthop. Oct-Nov 2007;27(7):844-7. [Medline].

  4. Demirag B, Ozturk C, Yazici Z, Sarisozen B. The pathophysiology of Osgood-Schlatter disease: a magnetic resonance investigation. J Pediatr Orthop B. Nov 2004;13(6):379-82. [Medline].

  5. Kujala UM, Kvist M, Heinonen O. Osgood-Schlatter's disease in adolescent athletes. Retrospective study of incidence and duration. Am J Sports Med. Jul-Aug 1985;13(4):236-41. [Medline].

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  7. Ducher G, Cook J, Spurrier D, Coombs P, Ptasznik R, Black J, et al. Ultrasound imaging of the patellar tendon attachment to the tibia during puberty: a 12-month follow-up in tennis players. Scand J Med Sci Sports. Feb 2010;20(1):e35-40. [Medline].

  8. Ducher G, Cook J, Lammers G, Coombs P, Ptazsnik R, Black J, et al. The ultrasound appearance of the patellar tendon attachment to the tibia in young athletes is conditional on gender and pubertal stage. J Sci Med Sport. Jan 2010;13(1):20-3. [Medline].

  9. LAZERTE GD, RAPP IH. Pathogenesis of Osgood-Schlatter's disease. Am J Pathol. Jul-Aug 1958;34(4):803-15. [Medline]. [Full Text].

  10. EHRENBORG G. The Osgood-Schlatter lesion. A clinical study of 170 cases. Acta Chir Scand. Aug 1962;124:89-105. [Medline].

  11. EHRENBORG G. The Osgood-Schlatter lesion. A clinical and experimental study. Acta Chir Scand Suppl. 1962;Suppl 288:1-36. [Medline].

  12. Pihlajamäki HK, Mattila VM, Parviainen M, Kiuru MJ, Visuri TI. Long-term outcome after surgical treatment of unresolved Osgood-Schlatter disease in young men. J Bone Joint Surg Am. Oct 2009;91(10):2350-8. [Medline].

  13. Binazzi R, Felli L, Vaccari V, Borelli P. Surgical treatment of unresolved Osgood-Schlatter lesion. Clin Orthop Relat Res. Apr 1993;202-4. [Medline].

  14. Orava S, Malinen L, Karpakka J, Kvist M, Leppilahti J, Rantanen J, et al. Results of surgical treatment of unresolved Osgood-Schlatter lesion. Ann Chir Gynaecol. 2000;89(4):298-302. [Medline].

  15. Trail IA. Tibial sequestrectomy in the management of Osgood-Schlatter disease. J Pediatr Orthop. Sep-Oct 1988;8(5):554-7. [Medline].

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Radiograph of a patient who is skeletally mature. Note that the tibial tubercle is enlarged and there is an ossicle. A bursa was overlying this.
Radiograph of a patient who is skeletally immature. The tubercle is elongated and fragmented
Image courtesy of John T. Killion, MD; OSA Pediatric Orthopaedics
Image courtesy of John T. Killion, MD; OSA Pediatric Orthopaedics
 
 
 
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