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Abnormal Labor
Updated: Dec 19, 2008
Introduction
Background
To define abnormal labor, a definition of normal labor must be understood and accepted. Normal labor is defined as uterine contractions that result in progressive dilation and effacement of the cervix. By following thousands of labors resulting in uncomplicated vaginal deliveries, time limits and progress milestones have been identified that define normal labor. Failure to meet these milestones defines abnormal labor, which suggests an increased risk of an unfavorable outcome. Thus, abnormal labor alerts the obstetrician to consider alternative methods for a successful delivery that minimize risks to both the mother and the infant.
Dystocia of labor is defined as difficult labor or abnormally slow progress of labor. Other terms that are often used interchangeably with dystocia are dysfunctional labor, failure to progress (lack of progressive cervical dilatation or lack of descent), and cephalopelvic disproportion (CPD).
Friedman's original research in 1955 defined 3 stages of labor.1
- The first stage starts with uterine contractions leading to complete cervical dilation and is divided into latent and active phases. In the latent phase, irregular uterine contractions occur with slow and gradual cervical effacement and dilation. The active phase is demonstrated by an increased rate of cervical dilation and fetal descent. The active phase usually starts at 3-4 cm cervical dilation and is subdivided into the acceleration, maximum slope, and deceleration phases.
- The second stage of labor is defined as complete dilation of the cervix to the delivery of the infant.
- The third stage of labor involves delivery of the placenta.
See Media files 1-2 for the normal labor curves of both nulliparas and multiparas. The following table shows abnormal labor indicators.
Table. Abnormal Labor IndicatorsOpen table in new window
Table
| Indication | Nullipara | Multipara |
| Prolonged latent phase | >20 h | >14 h |
| Average second stage | 50 min | 20 min |
| Prolonged second stage without (with) epidural | >2 h (>3 h) | >1 h (>2 h) |
| Protracted dilation | <1.2 cm/h | <1.5 cm/h |
| Protracted descent | <1 cm/h | <2 cm/h |
| Arrest of dilation* | >2 h | >2 h |
| Arrest of descent* | >2 h | >1 h |
| Prolonged third stage | >30 min | >30 min |
| Indication | Nullipara | Multipara |
| Prolonged latent phase | >20 h | >14 h |
| Average second stage | 50 min | 20 min |
| Prolonged second stage without (with) epidural | >2 h (>3 h) | >1 h (>2 h) |
| Protracted dilation | <1.2 cm/h | <1.5 cm/h |
| Protracted descent | <1 cm/h | <2 cm/h |
| Arrest of dilation* | >2 h | >2 h |
| Arrest of descent* | >2 h | >1 h |
| Prolonged third stage | >30 min | >30 min |
*Adequate contractions >200 Montevideo units [MVU] per 10 minutes for 2 hours. (Please refer to the Pathophysiology for information regarding adequate contractions.)
Abnormal labor constitutes any findings that fall outside the accepted normal labor curve. However, the authors hesitate to apply the diagnosis of abnormal labor during the latent phase because it is easy to confuse prodromal contractions for latent labor. In addition, the original labor curve, as defined by Friedman, may not be completely applicable today.2,3,4,5
First stage of labor
Latent phase: Definitions for prolonged latent phase are outlined in the table above. Diagnosis of abnormal labor during the latent phase is uncommon and likely an incorrect diagnosis.
Active phase: Around the time uterine contractions cause the cervix to become 3-4 cm dilated, the patient usually enters the active phase of the first stage of labor. Abnormalities of cervical dilation (protracted dilation and arrest of dilation) as well as descent abnormalities (protracted descent and arrest of descent) are outlined in the table above.
In general, abnormal labor is the result of problems with one of the 3 P' s.
- Passenger (infant size, fetal presentation [occiput anterior, posterior, or transverse])
- Pelvis or passage (size, shape, and adequacy of the pelvis)
- Power (uterine contractility)
See Causes.
Pathophysiology
A prolonged latent phase may result from oversedation or from entering labor early with a thickened or uneffaced cervix. It may be misdiagnosed in the face of frequent prodromal contractions. Protraction of active labor is more easily diagnosed and is dependent upon the 3 P' s.
The first P, the passenger, may produce abnormal labor because of the infant's size (eg, macrosomia) or from malpresentation.
The second P, the pelvis, can cause abnormal labor because its contours may be too small or narrow to allow passage of the infant. Both the passenger and pelvis cause abnormal labor by a mechanical obstruction, referred to as mechanical dystocia.
With the third P, the power component, the frequency of uterine contraction may be adequate, but the intensity may be inadequate. Disruption of communication between adjacent segments of the uterus may also exist, resulting from surgical scarring, fibroids, or other conduction disruption. Whatever the cause, the contraction pattern fails to result in cervical effacement and dilation. This is called functional dystocia. Uterine contractile force can be quantified by the use of an intra-uterine pressure catheter. Use of this device allows for direct measurement and calculation of uterine contractility per each contraction and is reported in Montevideo units (MVUs). For uterine contractile force to be considered adequate, the force produced must exceed 200 MVUs during a 10-minute contraction period. Arrest disorders cannot be properly diagnosed until the patient is in the active phase and had no cervical change for 2 or more hours with the contraction pattern exceeding 200 MVUs. Uterine contractions must be considered adequate to correctly diagnose arrest of dilation.6
Frequency
United States
Of all cephalic deliveries, 8-11% are complicated by an abnormal first stage of labor. Dystocia occurs in 12% of deliveries in women without a history of prior cesarean delivery. Dystocia may account for as many as 60% of cesarean deliveries.
Mortality/Morbidity
Both maternal and fetal mortality and morbidity rates increase with abnormal labor. This is probably an effect-effect relationship rather than a cause-effect relationship. Nonetheless, identification of abnormal labor and initiation of appropriate actions to reduce the risks are matters of some urgency.
Clinical
History
- Evaluate every pregnant patient who presents with contractions in the labor and delivery unit.
- Any patient in labor is at risk for abnormal labor regardless of the number of previous pregnancies or the seemingly adequate dimensions of the pelvis.
- Plot the progress of any patient in labor, and evaluate it on a labor curve (see Media files 1-2).
Physical
- Upon admission to the labor and delivery unit, determine and document clinical findings.
- Clinical pelvimetry, which is best performed at the first prenatal care visit, is important in order to assess the pelvic type (eg, android, gynecoid, platypelloid, anthropoid).
- Evaluate the position of the fetal head in early labor because caput and moulding complicate correct assessment as labor progresses.
- Establish and document an estimated fetal weight.
- Monitor fetal heart rate and uterine contraction patterns to assess fetal well-being and adequacy of labor.
- Perform a cervical examination to determine whether the patient is in the latent or active phase of labor.
- Addressing these issues allows for an assessment of the current phase of labor and anticipation of whether abnormal labor from any of the 3 P' s may be encountered.
Causes
- Prolonged latent phase: The latent phase of labor is defined as the period of time starting with the onset of regular uterine contractions and ending with the onset of the active phase (usually 3-4 cm cervical dilation).
- A prolonged latent phase is defined as exceeding 20 hours in patients who are nulliparas or 14 hours in patients who are multiparas.
- The most common reason for prolonged latent phase is entering labor without substantial cervical effacement.
- Power: Power is defined as uterine contractility multiplied by the frequency of contractions.
- Montevideo units (MVUs) refer to the strength of contractions in millimeters of mercury multiplied by the frequency per 10 minutes as measured by intrauterine pressure transducer.
- The uterine contraction pattern should repeat every 2-3 minutes.
- The uterine contractile force produced must exceed 200 MVUs/10 min for active labor to be considered adequate. For example, 3 contractions in 10 minutes that each reach a peak of 60 mm Hg are 60 X 3 = 180 MVUs.
- An arrest disorder of labor cannot be diagnosed until the patient is in the active phase and the contraction pattern exceeds 200 MVUs for 2 or more hours with no cervical change. Extending the minimum period of oxytocin augmentation for active-phase arrest from 2 up to 4 hours may be considered as long as fetal reassurance is noted with fetal heart rate monitoring.
- Pelvis or the size of the passageway inhibiting delivery
- The shape of the bony pelvis (eg, anthropoid or platypelloid) can result in abnormal labor.
- A patient who is extremely short or obese, or who has had prior severe trauma to the bony pelvis, may also be at increased risk of abnormal labor.
- Abnormal labor could also be secondary to the passenger, the size of the infant, and/or the presentation of the infant.
- In addition to problems caused by the differential in size between the fetal head and the maternal bony pelvis, the fetal presentation may include asynclitism or head extension. Asynclitism is malposition of the fetal head within the pelvis, which compromises the narrowest diameter through the pelvis.
- Fetal macrosomia and other anomalies (including hydrocephalus, encephalocele, fetal goiter, cystic hygroma, hydrops, or any other abnormality that increases the size of the infant) are likely to cause deviation from the normal labor curve.
- Other factors include either a low-dose epidural or combined spinal-epidural anesthetics that minimize motor block and may contribute to a prolonged second stage. These have also been associated with an increase in oxytocin use and operative vaginal delivery. However, use of epidural for analgesia during labor does not result in a statistically significant increase in cesarean delivery.7 Intravenous oversedation has also been implicated as prolonging labor in both the latent and active phases.
More on Abnormal Labor |
Overview: Abnormal Labor |
| Differential Diagnoses & Workup: Abnormal Labor |
| Treatment & Medication: Abnormal Labor |
| Follow-up: Abnormal Labor |
| Multimedia: Abnormal Labor |
| References |
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References
Friedman EA. Primigravid labor; a graphicostatistical analysis. Obstet Gynecol. Dec 1955;6(6):567-89. [Medline].
Zhang J, Troendle JF, Yancey MK. Reassessing the labor curve in nulliparous women. Am J Obstet Gynecol. Oct 2002;187(4):824-8. [Medline].
Rouse DJ, Owen J, Hauth JC. Criteria for failed labor induction: prospective evaluation of a standardized protocol. Obstet Gynecol. Nov 2000;96(5 Pt 1):671-7. [Medline].
Cheng YW, Hopkins LM, Caughey AB. How long is too long: Does a prolonged second stage of labor in nulliparous women affect maternal and neonatal outcomes?. Am J Obstet Gynecol. Sep 2004;191(3):933-8. [Medline].
Rinehart BK, Terrone DA, Hudson C, et al. Lack of utility of standard labor curves in the prediction of progression during labor induction. Am J Obstet Gynecol. Jun 2000;182(6):1520-6. [Medline].
Cunningham FG, Leveno KL, Bloom SL, et al. Abnormal labor. In: Williams Obstetrics. 22nd ed. Appleton & Lange; 2007:415-434.
Anim-Somuah M, Smyth R, Howell C. Epidural versus non-epidural or no analgesia in labour. Cochrane Database Syst Rev. Oct 19 2005;CD000331. [Medline].
Gabbe SJ, O'Brien WF, Cefalo RC. Labor and delivery. In: Obstetrics: Normal and Problem Pregnancies. 5th ed. 2007:322-326.
Sanchez-Ramos L, Quillen MJ, Kaunitz AM. Randomized trial of oxytocin alone and with propranolol in the management of dysfunctional labor. Obstet Gynecol. Oct 1996;88(4 Pt 1):517-20. [Medline].
Mitrani A, Oettinger M, Abinader EG, et al. Use of propranolol in dysfunctional labour. Br J Obstet Gynaecol. Aug 1975;82(8):651-5. [Medline].
Smyth RM, Alldred SK, Markham C. Amniotomy for shortening spontaneous labour. Cochrane Database Syst Rev. Oct 17 2007;CD006167. [Medline].
Mollberg M, Hagberg H, Bager B, et al. Risk factors for obstetric brachial plexus palsy among neonates delivered by vacuum extraction. Obstet Gynecol. Nov 2005;106(5 Pt 1):913-8. [Medline].
Mehta SH, Bujold E, Blackwell SC, et al. Is abnormal labor associated with shoulder dystocia in nulliparous women?. Am J Obstet Gynecol. Jun 2004;190(6):1604-7; discussion 1607-9. [Medline].
Shields SG, Ratcliffe SD, Fontaine P, et al. Dystocia in nulliparous women. Am Fam Physician. Jun 1 2007;75(11):1671-8. [Medline].
Oppenheimer LW, Labrecque M, Wells G, Bland ES, Fraser WD, Eason E. Prostaglandin E vaginal gel to treat dystocia in spontaneous labour: a multicentre randomised placebo-controlled trial. BJOG. May 2005;112(5):612-8. [Medline].
Treacy A, Robson M, O'Herlihy C. Dystocia increases with advancing maternal age. Am J Obstet Gynecol. Sep 2006;195(3):760-3. [Medline].
Zhu BP, Grigorescu V, Le T, et al. Labor dystocia and its association with interpregnancy interval. Am J Obstet Gynecol. Jul 2006;195(1):121-8. [Medline].
American College of Obstetricians and Gynecologists. ACOG technical bulletin. Dystocia and the augmentation of labor. Number 218--December 1995 (replaces no. 137, December 1989, and no. 157, July 1991). American College of Obstetricians and Gynecologists. Int J Gynaecol Obstet. Apr 1996;53(1):73-80. [Medline].
American College of Obstetrics and Gynecology Committee on Practice Bulletins-Obstetrics. ACOG Practice Bulletin Number 49, December 2003: Dystocia and augmentation of labor. Obstet Gynecol. Dec 2003;102(6):1445-54. [Medline].
Cheng YW, Hopkins LM, Laros RK Jr, et al. Duration of the second stage of labor in multiparous women: maternal and neonatal outcomes. Am J Obstet Gynecol. Jun 2007;196(6):585.e1-6. [Medline].
Creasy RK, Resnik R, Iams J. Clinical aspects of normal and abnormal labor. In: Maternal-Fetal Medicine. 5th ed. 2003:543-549.
Friedman EA. Labor in multiparas; a graphicostatistical analysis. Obstet Gynecol. Dec 1956;8(6):691-703. [Medline].
Gardberg M, Stenwall O, Laakkonen E. Recurrent persistent occipito-posterior position in subsequent deliveries. BJOG. Feb 2004;111(2):170-1. [Medline].
Gifford DS, Morton SC, Fiske M, et al. Lack of progress in labor as a reason for cesarean. Obstet Gynecol. Apr 2000;95(4):589-95. [Medline].
Hoffman MK, Vahratian A, Sciscione AC, et al. Comparison of labor progression between induced and noninduced multiparous women. Obstet Gynecol. May 2006;107(5):1029-34. [Medline].
Saito M, Kozuma S, Kikuchi A, et al. Sonographic assessment of the cervix before, during and after a uterine contraction is effective in predicting the course of labor. Ultrasound Obstet Gynecol. Dec 2003;22(6):604-8. [Medline].
Socol ML, Peaceman AM. Active management of labor. Obstet Gynecol Clin North Am. Jun 1999;26(2):287-94. [Medline].
Vahratian A, Hoffman MK, Troendle JF, et al. The impact of parity on course of labor in a contemporary population. Birth. Mar 2006;33(1):12-7. [Medline].
Further Reading
Keywords
dystocia, prolonged labor, arrest of dilation, arrest of descent, cephalopelvic disproportion, protraction disorder, primary dysfunctional labor, failure to progress, lack of progressive cervical dilatation, lack of descent, cephalopelvic disproportion, CPD, pelvimetry, Montevideo units, MVUs, cesarean section, c-section, cesarean birth, cesarean delivery, mechanical dystocia, functional dystocia
Overview: Abnormal Labor