Mechanical Low Back Pain Clinical Presentation
- Author: Everett C Hills, MD, MS; Chief Editor: Rene Cailliet, MD more...
History
Patients generally present with a history of an inciting event that produced immediate low back pain (LBP). The most commonly reported histories include the following:
- Lifting and/or twisting while holding a heavy object (eg, box, child, nursing home resident, a package on a conveyor)
- Operating a machine that vibrates
- Prolonged sitting (eg, long-distance truck driving, police patrolling)[10]
- Involvement in a motor vehicle collision
- Falls
Commence the history by asking for the patient's age, hand dominance, and occupation. Also ask about the patient's current work status and last day he or she worked. If the back pain is the result of a work-related injury, ask the name of the employer and inquire how long the patient has worked for this particular employer. Sample questions are as follows:
- When did the current symptoms begin and what were you doing?
- What and where are your symptoms now? (A pain diagram is helpful for localizing the symptoms. The patient can draw on a figure and give the clinician an idea of the nature of the pain as neuropathic or nociceptive.)
- Rate the pain on a scale of 0 (none) to 10 (worst imaginable). This is a global pain rating that takes into account physiological and psychological aspects of the LBP.
- What makes the pain better (eg, sitting, standing, laying, medications, physical therapy)?
- What makes the pain worse (eg, sitting, standing, laying, medications, physical therapy)?
- What affect have these symptoms had on sleep, mood, work, activities of daily living, and/or social functioning?
Use open-ended questions to ascertain the maximum information about the patient's history. Establishing a rapport with the patient is essential to detect serious conditions, provide insights into the patient's concerns and expectations, and to achieve the optimum positive response to treatment.
In addition to the history of the present illness, the past medical history should be obtained to rule out infections (eg, septic arthritis), congenital abnormalities (eg, dysplasias, juvenile rheumatoid arthritis), metabolic disorders (eg, Paget disease), or previous traumatic causes (eg, athletic participation, military service).
The review of systems is helpful for relating the current symptoms to any other body parts or systems. Interruption in bowel or bladder function should be a reminder to consider more serious causes of back pain such as a tumor, infection, or fracture. Review of systems also should include a thorough medical history (including history of cancer, arthritis, infection, systemic disease that could increase susceptibility to infection, nocturnal pain, fever, drug use, depression, and symptoms suggestive of metabolic or metastatic disease). Ask for any history of headaches, peptic ulcer disease, prior cancer, or unexplained weight loss.
Assess for any history of previous treatments, such as the following:
- Surgery
- Medications: Obtain as complete a listing as possible, including reasons for discontinuation.
- Physical therapy
- Psychiatric or psychological therapy
Thoroughly screen for anxiety, depression, addiction, somatoform disorders, personality disorders, other prior psychiatric diagnoses, coping styles, and personality traits. Psychosocial factors (eg, depression, hypochondriasis, heavy alcohol consumption, tobacco use, menial work, poor job satisfaction, stressors at home and/or work) may accompany histories involving a work-related injury.
Assess the patient's vocational history. Look for consistency in the type of work and length of service with each employer since high school or college. Ask how many years the patient has been working for his or her current employer. Some cases have involved patients who have worked less than a week on a new job. Some work-related injuries are reported on a Monday or after a vacation. These are important dates for determining if the LBP was indeed work-related.
In a work-related LBP case, ask the patient about pending or planned litigation and related expectations.
Ask the patient what he or she thinks about the cause of the LBP.
Ask the patient what his or her goals are for the evaluation and treatment.
If the patient brought imaging study results (eg, plain radiographs, computed tomography [CT] scans, magnetic resonance imaging [MRI] scans), look for imaging evidence of herniated nucleus pulposus (seen in the images below), spinal stenosis, or other conditions associated with back pain.
Magnetic resonance image of the lumbar spine. This image demonstrates a herniated nucleus pulposus at multiple levels.
Sagittal magnetic resonance image showing loss of intervertebral disk height at L5/S1. Herniations of the nucleus pulposus are noted at L4/5 and L5/S1. Physical
An important part of the physical examination is the general observation of the patient. The patient presents with pain in the low back region and often places his or her whole hand against the skin to indicate a regional pain; however, in some cases the patient may indicate a more precise location.
Realize that much of the physical examination is subjective because a patient-generated response or interpretation to the examiner's questions or maneuvers is required. For example, sensory findings observed during the physical examination and reported symptoms in response to provocative testing are reliant on the patient's response and, hence, represent a somewhat subjective portion of the physical examination. A well-performed and well-documented physical examination, with consistent findings from one visit to the next, can yield important information that may be able to stand up to rigorous scrutiny by any involved third parties (eg, insurance company, attorney, workers' compensation judge). These physical examination findings would need to be put into the context of the patient's symptoms and diagnostic test results.
Equipment often used for the examination includes a stethoscope, goniometer, inclinometer, pinwheel or safety pin, tape measure, and reflex hammer.
Testing
Observe the patient walking into the office or examining room. Observe the patient during the history-gathering portion of the visit for development, nutrition, deformities, and attention to grooming.
Measure blood pressure, pulse, respirations, temperature, height, and weight.
Inspect the back for signs of asymmetry, lesions, scars, trauma, or previous surgery.
Note chest expansion. If it is less than 2.5 cm, this finding can be specific, but not sensitive, for ankylosing spondylitis.
Take measurements of the calf circumferences (at midcalf). Differences of less than 2 cm are considered normal variation.
Measure lumbar range of motion (ROM) in forward bending while standing (Schober test).
The neurologic examination should test 2 muscles and 1 reflex representing each lumbar root to accurately distinguish between focal neuropathy and root problems.
Measure leg lengths (anterior superior iliac spine to medial malleolus) if side-to-side discrepancy is suspected.
Using the inclinometer, measure forward, backward, and lateral bending. With the goniometer positioned in a horizontal plane over the axial skeleton (ie, over the head), measure trunk rotation.
The AMA Guides to the Evaluation of Permanent Impairment (5th edition) include reference tables for all motions, but these figures are not based on empiric data, only on consensus.[12] The ROM measurements in the AMA Guides do not correlate with disability and are not consistent within the document itself.
Palpate the entire spine to identify vertebral tenderness that may be a nonspecific finding of fracture or other cause of low back pain (LBP). Note any asymmetry, misalignment, or step-off between vertebral bodies. Remember also to palpate the sacroiliac joints.
Test for manual muscle strength in both lower extremities. The Medical Research Council rating is an ordinal scale used for this purpose (0 = absent strength, 1 = trace muscle movement, 2 = poor muscle strength [less than antigravity], 3 = fair muscle strength [antigravity strength through normal arc of motion], 4 = good strength, and 5 = normal strength).
Table 1. Functional Muscle Testing (Open Table in a new window)
| Nerve Root | Motor Examination | Functional Test |
| L3 | Extend quadriceps | Squat down and rise |
| L4 | Dorsiflex ankle | Walk on heels |
| L5 | Dorsiflex great toe | Walk on heels |
| S1 | Stand on toes* | Walk on toes (plantarflex ankle) |
| *When testing the S1 innervated gastrocnemius muscle, the ability to stand on the toes once represents fair (3/5) strength. The patient must stand on his or her toes 5 times in a row to be rated normal (5/5) strength. Note that this approach should allow the physician to detect weakness at a much milder stage than if gastrocnemius strength were assessed only by using the examiner's hand to apply resistance to ankle plantar flexion. | ||
Test for sensation and reflexes using 0-2 ordinal scale for pinprick sensation (0 = no sensation, 1 = diminished sensation, and 2 = normal sensation), and 0-4 ordinal scale to rate reflexes (0 = no reflex, 1 = hyporeflexic, 2 = normal reflex, 3 = hyperreflexic, and 4 = hyperreflexic with clonus).
Table 2. Dermatomal Sensory and Reflex Testing (Open Table in a new window)
| Nerve Root | Pin-Prick Sensation | Reflex |
| L3 | Lateral thigh and medial femoral condyle | Patellar tendon reflex |
| L4 | Medial leg and medial ankle | Patellar tendon reflex |
| L5 | Lateral leg and dorsum of foot | Medial hamstring |
| S1 | Sole of foot and lateral ankle | Achilles tendon reflex |
Clinical tests for signs of sciatic nerve tension are as follows:
- Supine straight leg raising (SLR) test - Reproduction of pain caused by elevation of the contralateral limb raises the probability of a disk herniation to 98%. Remember that the SLR test result can be negative in persons with spinal stenosis.
- Sitting SLR (knee extension) test (for lower roots) - The patient should sit on the table edge with both hips and knees flexed at 90° and extend the knee slowly. This maneuver stretches the nerve roots as much as a moderate degree of supine SLR. The SLR test result, if positive, reproduces symptoms of sciatica with pain that radiates below the knee.
- The prone SLR test (also called the reverse SLR test or the femoral nerve stretch test) assesses the upper lumbar roots, a less common site of radiculopathy worth remembering.
Nonphysiologic testing (Waddell signs) should be performed. The presence of 3 or more positive findings out of the 5 types may be clinically significant in terms of psychosocial issues or poor surgical outcome. Isolated positive signs are of limited value.
Nonorganic tenderness consists of the following:
- Superficial - Skin tenderness to light pinch over a wide area of lumbar surface
- Nonanatomic - Deep tenderness over a wide area, often extending cephalad to the thoracic spine or caudad to the sacrum
Simulation tests give the patient the impression that a particular examination is being conducted, including the following:
- Axial loading - Vertical loading over the patient's head while he or she is standing, producing LBP
- Rotation - Back pain when the shoulders and pelvis are rotated passively in the same plane with the feet together
Distraction tests indicate a positive finding when the patient's attention is distracted.
- SLR - Observing an improvement of 30-40° when the patient is distracted, compared with formal testing.
- Flip test - The patient is seated with the legs dangling over the examination table. Instruct the patient to steady himself or herself by holding the edge of the table. When the affected leg is flipped up quickly, the patient falls back and lets go, placing both hands behind him or her on the table.
Regional disturbances that do not correlate with anatomy include the following:
- Weakness - Cogwheeling (giving way) of many muscle groups upon manual muscle testing of strength
- Sensory - Diminished light touch or pinprick sensation in a stocking pattern, rather than a dermatomal pattern, in an individual who is not diabetic
- Nonanatomic sensory loss
Overreaction during the examination may be observed in several manifestations (eg, disproportionate verbalization, facial grimacing, muscle tension and tremor, collapsing, sweating). Care must be taken to account for cultural variations.
In addition, evaluate the patient's function. Observe ROM and flexibility, ability to dress and undress, and ability to rise from a chair or the examination table.
Institute for Clinical Systems Improvement. Adult low back pain. Bloomington, Minn: Institute for Clinical Systems Improvement;. Sept 2005.
National Guideline Clearinghouse. Listing of Guidelines for Low Back Pain. Accessed December 29, 2005. Available at: http://www.guidelines.gov. [Full Text].
Philadelphia Panel evidence-based clinical practice guidelines on selected rehabilitation interventions for low back pain. Phys Ther. Oct 2001;81(10):1641-74. [Medline]. [Full Text].
Bigos SJ, Boyer OR, Braen GR. Acute Low Back Problems in Adults. Clinical Practice Guideline, Quick Reference Guide Number 14. Public Health Agency, Agency for Health Care Policy and Research. Rockville, Md:. Department of Health and Human Services;1994.
Bone and Joint Decade. US Bone and Joint Decade Web site. Accessed December 28, 2005. Available at: http://www.usbjd.org/index.cfm. [Full Text].
Datta S, Lee M, Falco FJ, et al. Systematic assessment of diagnostic accuracy and therapeutic utility of lumbar facet joint interventions. Pain Physician. Mar-Apr 2009;12(2):437-60. [Medline]. [Full Text].
Rupert MP, Lee M, Manchikanti L, et al. Evaluation of sacroiliac joint interventions: a systematic appraisal of the literature. Pain Physician. Mar-Apr 2009;12(2):399-418. [Medline].
Lambeek LC, van Mechelen W, Knol DL, et al. Randomised controlled trial of integrated care to reduce disability from chronic low back pain in working and private life. BMJ. Mar 16 2010;340:c1035. [Medline]. [Full Text].
Heuch I, Hagen K, Heuch I, et al. The Impact of Body Mass Index on the Prevalence of Low Back Pain: The HUNT Study. Spine (Phila Pa 1976). Mar 11 2010;[Medline].
Chen SM, Liu MF, Cook J, et al. Sedentary lifestyle as a risk factor for low back pain: a systematic review. Int Arch Occup Environ Health. Mar 20 2009;[Medline].
Rivinoja AE, Paananen MV, Taimela SP, Solovieva S, Okuloff A, Zitting P, et al. Sports, Smoking, and Overweight During Adolescence as Predictors of Sciatica in Adulthood: A 28-Year Follow-up Study of a Birth Cohort. Am J Epidemiol. Apr 15 2011;173(8):890-7. [Medline].
Cocchiarella L, Andersson GBJ. AMA Guides to the Evaluation of Permanent Impairment. 5th ed. 2000.
[Best Evidence] Sertpoyraz F, Eyigor S, Karapolat H, et al. Comparison of isokinetic exercise versus standard exercise training in patients with chronic low back pain: a randomized controlled study. Clin Rehabil. Mar 2009;23(3):238-47. [Medline].
Kumar S, Sharma VP, Shukla R, et al. Comparative efficacy of two multimodal treatments on male and female sub-groups with low back pain (part II). J Back Musculoskelet Rehabil. Jan 1 2010;23(1):1-9. [Medline].
van Middelkoop M, Rubinstein SM, Verhagen AP, et al. Exercise therapy for chronic nonspecific low-back pain. Best Pract Res Clin Rheumatol. Apr 2010;24(2):193-204. [Medline].
Long A, Donelson R, Fung T. Does it matter which exercise? A randomized control trial of exercise for low back pain. Spine. Dec 1 2004;29(23):2593-602. [Medline].
[Best Evidence] Juni P, Battaglia M, Nuesch E, et al. A randomised controlled trial of spinal manipulative therapy in acute low back pain. Ann Rheum Dis. Sep 2009;68(9):1420-7. [Medline].
Hill JC, Whitehurst DG, Lewis M, et al. Comparison of stratified primary care management for low back pain with current best practice (STarT Back): a randomised controlled trial. Lancet. Oct 29 2011;378(9802):1560-71. [Medline].
Tilbrook HE, Cox H, Hewitt CE, et al. Yoga for chronic low back pain: a randomized trial. Ann Intern Med. Nov 1 2011;155(9):569-78. [Medline].
Sherman KJ, Cherkin DC, Wellman RD, Cook AJ, Hawkes RJ, Delaney K, et al. A Randomized Trial Comparing Yoga, Stretching, and a Self-care Book for Chronic Low Back Pain. Arch Intern Med. Dec 12 2011;171(22):2019-26. [Medline].
Epter RS, Helm S, Hayek SM, et al. Systematic review of percutaneous adhesiolysis and management of chronic low back pain in post lumbar surgery syndrome. Pain Physician. Mar-Apr 2009;12(2):361-378. [Medline].
Veresciagina K, Ambrozaitis KV, Spakauskas B. The measurements of health-related quality-of-life and pain assessment in the preoperative patients with low back pain. Medicina (Kaunas). 2009;45(2):111-22. [Medline].
[Guideline] Chou R, Loeser JD, Owens DK, et al. Interventional therapies, surgery, and interdisciplinary rehabilitation for low back pain: an evidence-based clinical practice guideline from the American Pain Society. Spine. May 1 2009;34(10):1066-77. [Medline].
Chen CP, Wong AM, Hsu CC, et al. Ultrasound as a Screening Tool for Proceeding With Caudal Epidural Injections. Arch Phys Med Rehabil. Mar 2010;91(3):358-63. [Medline].
van Tulder MW, Scholten RJ, Koes BW, et al. Non-steroidal anti-inflammatory drugs for low back pain. Cochrane Database Syst Rev. 2000;CD000396.
US Food and Drug Administration. FDA News Release. FDA clears Cymbalta to treat chronic musculoskeletal pain. Available at. Available at http://www.fda.gov/NewsEvents/Newsroom/PressAnnouncements/ucm232708.htm.. Accessed November 5, 2010.
Mehling WE, Gopisetty V, Acree M, Pressman A, Carey T, Goldberg H, et al. Acute low back pain and primary care: how to define recovery and chronification?. Spine (Phila Pa 1976). Dec 15 2011;36(26):2316-23. [Medline]. [Full Text].
Adams MA, May S, Freeman BJ, et al. Effects of backward bending on lumbar intervertebral discs. Relevance to physical therapy treatments for low back pain. Spine. Feb 15 2000;25(4):431-7; discussion 438. [Medline].
Atkinson JH, Slater MA, Wahlgren DR, et al. Effects of noradrenergic and serotonergic antidepressants on chronic low back pain intensity. Pain. Nov 1999;83(2):137-45. [Medline].
Batt ME, Todd C. Five facts and five concepts for rehabilitation of mechanical low back pain. Br J Sports Med. Aug 2000;34(4):261. [Medline].
Borenstein D. Epidemiology, etiology, diagnostic evaluation, and treatment of low back pain. Curr Opin Rheumatol. Mar 1996;8(2):124-9. [Medline].
Brigham CR, Babitsky S, Mangraviti JJ. The Independent Medical Evaluation Report: A Step-by-Step Guide with Models. SEAK Inc;1996.
Cherkin DC, Wheeler KJ, Barlow W, et al. Medication use for low back pain in primary care. Spine. Mar 1 1998;23(5):607-14. [Medline].
Chrubasik S, Eisenberg E, Balan E, et al. Treatment of low back pain exacerbations with willow bark extract: a randomized double-blind study. Am J Med. Jul 2000;109(1):9-14. [Medline].
Consumer Reports. Back pain: the best treatment is surprisingly simple. Consumer Reports;1995:620-621.
Cox ME, Asselin S, Gracovetsky SA, et al. Relationship between functional evaluation measures and self-assessment in nonacute low back pain. Spine. Jul 15 2000;25(14):1817-26. [Medline].
Derby R, Bogduk N, Anat D, et al. Precision percutaneous blocking procedures for localizing spine pain. Part 1: the posterior lumbar compartment. Pain Digest. 1993;3:89-100.
Deyo RA, Rainville J, Kent DL. What can the history and physical examination tell us about low back pain?. JAMA. Aug 12 1992;268(6):760-5. [Medline].
Di Fabio RP, Mackey G, Holte JB. Physical therapy outcomes for patients receiving worker's compensation following treatment for herniated lumbar disc and mechanical low back pain syndrome. J Orthop Sports Phys Ther. Mar 1996;23(3):180-7. [Medline].
Dishman JD, Bulbulian R. Spinal reflex attenuation associated with spinal manipulation [In Process Citation]. Spine. Oct 1 2000;25(19):2519-25. [Medline].
Drugs for pain. Med Lett Drugs Ther. Aug 21 2000;42(1085):73-8. [Medline].
Duance VC, Crean JK, Sims TJ, et al. Changes in collagen cross-linking in degenerative disc disease and scoliosis. Spine. Dec 1 1998;23(23):2545-51. [Medline].
Ferguson SA, Marras WS, Gupta P. Longitudinal quantitative measures of the natural course of low back pain recovery. Spine. Aug 1 2000;25(15):1950-6. [Medline].
Frymoyer JW. Back pain and sciatica. N Engl J Med. Feb 4 1988;318(5):291-300. [Medline].
Furlan AD, Brosseau L, Welch V, Wong J. Massage for low back pain. Cochrane Database Syst Rev. 2000;CD001929.
Guidelines for the assessment and management of chronic pain. WMJ. 2004;103(3):13-42. [Medline].
Handa N, Yamamoto H, Tani T, et al. The effect of trunk muscle exercises in patients over 40 years of age with chronic low back pain. J Orthop Sci. 2000;5(3):210-6. [Medline].
Hanson P, Qvortrup K, Magnusson SP. The superficial annulus fibrosus ligament. an incipient description of a separate ligament between the lumbar anterior longitudinal ligament and the intervertebral disc [In Process Citation]. Cells Tissues Organs. 2000;167(4):259-65. [Medline].
Harrington JF, Messier AA, Bereiter D, et al. Herniated lumbar disc material as a source of free glutamate available to affect pain signals through the dorsal root ganglion. Spine. Apr 15 2000;25(8):929-36. [Medline].
Hart LG, Deyo RA, Cherkin DC. Physician office visits for low back pain. Frequency, clinical evaluation, and treatment patterns from a U.S. national survey. Spine. Jan 1 1995;20(1):11-9. [Medline].
Hoogendoorn WE, van Poppel MN, Bongers PM, et al. Systematic review of psychosocial factors at work and private life as risk factors for back pain. Spine. Aug 15 2000;25(16):2114-25. [Medline].
McMorland G, Suter E. Chiropractic management of mechanical neck and low-back pain: a retrospective, outcome-based analysis. J Manipulative Physiol Ther. Jun 2000;23(5):307-11. [Medline].
North American Spine Society. Orthopedic Knowledge Update: Spine. NASS/AAOS;1997:113-119.
Pustaver MR. Mechanical low back pain: etiology and conservative management. J Manipulative Physiol Ther. Jul-Aug 1994;17(6):376-84. [Medline].
Rungee JL. Low back pain during pregnancy. Orthopedics. Dec 1993;16(12):1339-44. [Medline].
Sizer PS Jr, Matthijs O, Phelps V. Influence of age on the development of pathology. Curr Rev Pain. 2000;4(5):362-73. [Medline].
Solomonow M, He Zhou B, Baratta RV, et al. Biexponential recovery model of lumbar viscoelastic laxity and reflexive muscular activity after prolonged cyclic loading. Clin Biomech (Bristol, Avon). Mar 2000;15(3):167-75. [Medline].
Spengler D. Herniated nucleus pulposus surgery: effect of compensation. The Back Letter. 1997;vol 12.
Spitzer WO, Skovron ML, Salmi LR, et al. Scientific monograph of the Quebec Task Force on Whiplash-Associated Disorders: redefining "whiplash" and its management. Spine. Apr 15 1995;20(8 Suppl):1S-73S. [Medline].
Von Feldt JM, Ehrlich GE. Pharmacologic therapies. Phys Med Rehabil Clin N Am. May 1998;9(2):473-87, ix. [Medline].
Waddell G, McCulloch JA, Kummel E, Venner RM. Nonorganic physical signs in low-back pain. Spine. Mar-Apr 1980;5(2):117-25. [Medline].
Wilson L, Hall H, McIntosh G, Melles T. Intertester reliability of a low back pain classification system. Spine. Feb 1 1999;24(3):248-54. [Medline].
| Nerve Root | Motor Examination | Functional Test |
| L3 | Extend quadriceps | Squat down and rise |
| L4 | Dorsiflex ankle | Walk on heels |
| L5 | Dorsiflex great toe | Walk on heels |
| S1 | Stand on toes* | Walk on toes (plantarflex ankle) |
| *When testing the S1 innervated gastrocnemius muscle, the ability to stand on the toes once represents fair (3/5) strength. The patient must stand on his or her toes 5 times in a row to be rated normal (5/5) strength. Note that this approach should allow the physician to detect weakness at a much milder stage than if gastrocnemius strength were assessed only by using the examiner's hand to apply resistance to ankle plantar flexion. | ||
| Nerve Root | Pin-Prick Sensation | Reflex |
| L3 | Lateral thigh and medial femoral condyle | Patellar tendon reflex |
| L4 | Medial leg and medial ankle | Patellar tendon reflex |
| L5 | Lateral leg and dorsum of foot | Medial hamstring |
| S1 | Sole of foot and lateral ankle | Achilles tendon reflex |

