Updated: Apr 18, 2008
Trigger finger results from thickening of the flexor tendon within the distal aspect of the palm.1,2 This thickening causes abnormal gliding of the tendon within the tendon sheath. Specifically, the affected tendon is caught at the edge of the first annular (A1) pulley. Patients can have difficulty flexing the affected digit if the tendon is caught distal to the A1 pulley, or extending the digit, if the tendon is caught proximal to the pulley. The condition is very painful, especially when the locked digit snaps (releases) beyond the restriction by the use of increased force. Images 1-2 illustrate normal and thickened tendons, respectively.
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Normally, the tendons of the finger flexors glide back and forth under a restraining pulley.3,4,5 Thickening of the flexor tendon sheath restricts the normal gliding mechanism. A nodule may develop on the tendon, causing the tendon to get stuck at the proximal edge of the A1 pulley when the patient is attempting to extend the digit, thereby causing difficulty. When more forceful attempts are made to extend the digit, by using increased force from the finger extensors or by applying an external force (for example, by exerting force on the finger with the other hand), the digit classically snaps open with significant pain at the distal palm and into the proximal aspect of the affected digit. Less commonly, the nodule is restricted distal to the A1 pulley, resulting in difficulty flexing the digit.
Trigger finger is a relatively common condition.
No racial predisposition is known to be associated with trigger finger.
This condition has a higher incidence in women (75%) than in men.
Trigger digits are most commonly seen in adults, with the average age range for its occurrence being 52-62 years.
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Carpal Tunnel Syndrome
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Diabetes mellitus, particularly in patients with multiple trigger fingers
Ganglion involving the tendon sheath
Infection within the tendon sheaths
Ganglion cyst of the wrist
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Physical therapy is generally not required for patients with trigger finger. For chronic cases, however, treatment may include a trial of heating modalities followed by sustained nonballistic stretching of the flexor tendon, as well as soft-tissue mobilization of the A1 pulley. Following injection or surgery, a home exercise (stretching) program may be one component of treatment for patients. No therapy programs have been documented to improve trigger finger.
If a trial of therapy is recommended for patients with chronic trigger finger or for individuals who require postsurgical hand therapy, the physician may refer them to a physical or occupational therapist, depending on his/her preference and the therapists' availability. The treatment provided by an occupational therapist is very similar to the above-discussed physical therapy treatment. In addition, the occupational therapist may provide a patient with strategies for completing activities of daily living with limited or no use of the affected hand while it is splinted or is recovering from surgery.
Surgical consultation for operative treatment may be required. Typically, such procedures are performed by an orthopedic hand surgeon or a plastic surgeon.
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For this musculoskeletal condition, medications are used primarily to decrease pain and inflammation in conjunction with the rehabilitation plan. Thus, the most common medication treatments are focal corticosteroid injection and the administration of NSAIDs.
Oral NSAIDs can help to decrease pain and inflammation. Various oral NSAIDs can be used, although none of these agents holds a clear distinction as the drug of choice. The choice of NSAID is largely a matter of convenience (how frequently doses must be taken to achieve adequate analgesic and anti-inflammatory effects) and cost.
DOC for patients with mild to moderate pain. NSAIDs inhibit inflammatory reactions and pain by decreasing prostaglandin synthesis.
200-800 mg PO tid/qid
<6 months: Not established
6 months to 12 years: 4-10 mg/kg/dose PO tid/qid
>12 years: Administer as in adults
Coadministration with aspirin increases risk of inducing serious NSAID-related adverse effects; probenecid may increase concentrations and, possibly, toxicity of NSAIDs; may decrease effect of hydralazine, captopril, and beta blockers; may decrease diuretic effects of furosemide and thiazides; monitor PT closely (instruct patients to watch for signs of bleeding); may increase risk of methotrexate toxicity; phenytoin levels may be increased when administered concurrently
Documented hypersensitivity, peptic ulcer disease, recent GI bleeding or perforation, renal insufficiency, or high risk of bleeding
C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus
D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus
Category D in third trimester of pregnancy; caution in congestive heart failure, hypertension, and decreased renal and hepatic function; to minimize risks of adverse effects, avoid taking multiple NSAIDs concurrently; caution in anticoagulation abnormalities or during anticoagulant therapy
In contrast to the widespread systemic distribution that occurs when an oral anti-inflammatory drug is administered, a local corticosteroid injection can achieve the focal placement of a potent anti-inflammatory agent at the site of maximal tenderness or inflammation. A variety of corticosteroid preparations are available. Commonly, the corticosteroid is mixed with a local anesthetic agent prior to injection. The clinician has numerous local anesthetic agents from which to choose.
Corticosteroids are commonly used in local injections administered to bursae or joints. The drugs provide a local anti-inflammatory effect while minimizing some of the GI and other risks of systemic medications.
40 mg (1 mL), intralesionally, is common for injection at many sites; often mixed with a few mL of a local anesthetic, such as 1% lidocaine
Not established
Local corticosteroid injection is not known to give rise to the same degree of medication interaction that oral or other systemic administration of corticosteroids produces; co-administration with anticoagulants may increase risk of hemorrhage or local bruising
Documented hypersensitivity; skin infection at the site of injection
C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus
Never inject corticosteroids through an infected area of skin; diabetic patients may sometimes experience transient elevation of blood glucose level after a local corticosteroid injection
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[Best Evidence] Peters-Veluthamaningal C, Winters JC, Groenier KH, et al. Corticosteroid injections effective for trigger finger in adults in general practice: a double-blinded randomized placebo controlled trial. Ann Rheum Dis. Jan 7 2008;[Medline].
[Best Evidence] Baumgarten KM, Gerlach D, Boyer MI. Corticosteroid injection in diabetic patients with trigger finger. A prospective, randomized, controlled double-blinded study. J Bone Joint Surg Am. Dec 2007;89(12):2604-11. [Medline].
Nonsteroidal anti-inflammatory drugs (NSAIDs). In: Green SM, ed. Tarascon Pocket Pharmacopoeia 2000. Loma Linda, Calif: Tarascon Pub; 2000:11-2.
Akhtar S, Bradley MJ, Quinton DN, et al. Management and referral for trigger finger/thumb. BMJ. Jul 2 2005;331(7507):30-3. [Medline].
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trigger finger, digital flexor tenosynovitis, digital tenovaginitis stenosans, flexor tendon stenosing tenosynovitis, locked finger, stick palsy, trigger digit, trigger thumb, volar flexor tenosynovitis, flexor pollicis longus tendon nodule
Patrick M Foye, MD, FAAPMR, FAAEM, Associate Professor of Physical Medicine and Rehabilitation, Co-Director of Musculoskeletal Fellowship, Co-Director of Back Pain Clinic, Director of Coccyx Pain (Tailbone Pain, Coccydynia) Service, University of Medicine and Dentistry of New Jersey, New Jersey Medical School
Patrick M Foye, MD, FAAPMR, FAAEM is a member of the following medical societies: American Academy of Physical Medicine and Rehabilitation, American Association of Neuromuscular and Electrodiagnostic Medicine, Association of Academic Physiatrists, and International Spine Intervention Society
Disclosure: Nothing to disclose.
Todd P Stitik, MD, Professor, Department of Physical Medicine and Rehabilitation, Acting Director of Sports Medicine, UMDNJ-New Jersey School of Medicine; Lead Physician, Practice Medical Director of University Hospital PM & R Clinic
Todd P Stitik, MD is a member of the following medical societies: American Academy of Physical Medicine and Rehabilitation, Association of Academic Physiatrists, Phi Beta Kappa, and Physiatric Association of Spine, Sports and Occupational Rehabilitation
Disclosure: Nothing to disclose.
Robert E Windsor, MD, FAAPMR, FAAEM, FAAPM, President and Director, Georgia Pain Physicians, PC; Clinical Associate Professor, Department of Physical Medicine and Rehabilitation, Emory University School of Medicine
Robert E Windsor, MD, FAAPMR, FAAEM, FAAPM is a member of the following medical societies: American Academy of Pain Medicine, American Academy of Physical Medicine and Rehabilitation, American College of Sports Medicine, American Medical Association, International Association for the Study of Pain, Physiatric Association of Spine, Sports and Occupational Rehabilitation, and Texas Medical Association
Disclosure: Nothing to disclose.
Francisco Talavera, PharmD, PhD, Senior Pharmacy Editor, eMedicine
Disclosure: Nothing to disclose.
Michael T Andary, MD, MS, Residency Program Director, Professor, Department of Physical Medicine and Rehabilitation, Michigan State University College of Osteopathic Medicine
Michael T Andary, MD, MS is a member of the following medical societies: American Academy of Physical Medicine and Rehabilitation, American Association of Neuromuscular and Electrodiagnostic Medicine, American Medical Association, and Association of Academic Physiatrists
Disclosure: Nothing to disclose.
Kelly L Allen, MD, Consulting Staff, Department of Physical Medicine and Rehabilitation, Lourdes Regional Rehabilitation Center, Our Lady of Lourdes Medical Center
Disclosure: Nothing to disclose.
Rene Cailliet, MD, Professor-Chairman Emeritus, Department of Rehabilitation Medicine, University of Southern California School of Medicine; Former Director, Department of Rehabilitation Medicine, Santa Monica Hospital Medical Center
Rene Cailliet, MD is a member of the following medical societies: American Academy of Pain Medicine, American Academy of Physical Medicine and Rehabilitation, American Pain Society, Association of American Medical Colleges, International Association for the Study of Pain, and Pan American Medical Association
Disclosure: Nothing to disclose.