Electrical Injuries in Emergency Medicine Clinical Presentation

  • Author: Tracy A Cushing, MD, MPH, FACEP, FAWM; Chief Editor: Rick Kulkarni, MD   more...
 
Updated: Apr 12, 2010
 

History

Electrical injuries can present with a variety of problems, including cardiac or respiratory arrest, coma, blunt trauma, and severe burns of several types. It is important to establish the type of exposure (high or low voltage), duration of contact, and falls or other trauma.

  • Low-voltage AC injury without loss of consciousness and/or arrest: These injuries are < 1000V exposures usually in the home or office setting. Typically, children with electrical injuries present after biting or chewing on an electrical cord and suffer oral burns. Adults working on home appliances or electrical circuits can also experience these electrical injuries. Low-voltage AC may result in significant injury if there is prolonged, tetanic muscle contraction.
  • Low-voltage AC injury with loss of consciousness and/or arrest: In respiratory arrest or ventricular fibrillation that is not witnessed, an electrical exposure may be difficult to diagnose. All unwitnessed arrests should include this possibility in the differential diagnosis. Query EMS personnel, family, and coworkers about this possibility. Inquire if a scream was heard before the patient’s collapse; this may be due to involuntary contraction of chest wall muscles from electrical current.
  • High-voltage AC injury without loss of consciousness and/or arrest: Usually high-voltage injuries do not cause loss of consciousness but instead cause devastating thermal burns. In occupational exposures, details of voltage can be obtained from the local power company.
  • High-voltage AC injury with loss of consciousness and/or arrest: This is an unusual presentation of high-voltage AC injuries, which do not often cause loss of consciousness. History may need to come from bystanders or EMS personnel.
  • Direct current (DC) injury: These injuries typically cause a single muscle contraction that throws the victim away from the source. They are rarely associated with loss of consciousness unless there is severe head trauma, and victims can often provide their own history.
  • Conducted electrical weapons (CEWs) such as tasers are weapons used by law enforcement that deliver high-voltage current that is neither true AC or DC but is most like a series of low-amplitude DC shocks.[15] They can deliver 50,000 V in a 5-second pulse, with an average current of 2.1 mA.[16] Though they have been temporally associated with deaths in the law enforcement setting, conducted electrical devices (CEDs) in healthy volunteers have been shown to be safe without evidence of delayed arrhythmia or cardiac damage as measured by troponin I.[17, 16] One study of their use in 1201 law enforcement incidents showed mostly superficial puncture wounds from the device probes, and significant injuries only from trauma subsequent to shock, not from the device itself. Of 2 deaths in custody, neither was related to CEW exposure.[18] Overall significant injuries from CEW exposure are rare, and usually occur due to trauma or in conjunction with intoxication.[19, 20]
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Physical

Electrical injuries can cause multiorgan dysfunction and a variety of burns and traumatic injuries. A thorough physical examination is required to assess the full extent of injuries. Occupational injuries have a high likelihood of future litigation, and physical examination findings should be documented with photographs if possible, with the proper releases, and filed in the patient's medical record.

Overall, low-voltage exposure tends to cause less overall morbidity than high-voltage, but it is important to ensure by accurate history that a seemingly low-voltage burn was not in fact from a high-voltage source (like a microwave, computer, or TV monitor—any device that "steps-up" voltage via a transformer). Low-voltage burns can still cause cardiac arrhythmia, seizure, and long-term complications if contact is near the chest or head.

  • Cardiovascular
    • Patients may present in asystole or ventricular fibrillation (VF) in addition to other arrhythmias. Sudden death due to VF is more common with low-voltage AC, whereas asystole is more often associated with high-voltage AC or DC. Ventricular fibrillation can be caused at voltages as low as 50-120 mA, which is lower than the typical household current. One series showed cardiac arrhythmias following 41% of low-voltage injuries.[12]
    • Electricity can also cause conduction abnormalities and direct trauma to cardiac muscle fibers. Survivors of electrical shock can experience subsequent arrhythmia, usually sinus tachycardia and premature ventricular contractions (PVCs). One study identified 3 cases of delayed ventricular arrhythmias up to 12 hours after the incident.[21] Other studies have shown no risk of delayed arrhythmias in patients with initially normal ECGs, both in low-voltage household exposures and after CEW exposure.[22, 19, 23, 18] Long-term cardiac complications occurring from electrical injury are rare.
  • Respiratory: Chest wall muscle paralysis from tetanic contraction may cause respiratory arrest if the current pathway is over the thorax. Injury to the respiratory control center of the brain can also cause respiratory arrest. The lungs are a poor conductor of electricity and generally are not as susceptible to direct injury from current as tissues with lower resistance.
  • Skin: A variety of burns and thermal injuries occurs from electricity that affect the skin and soft tissues. These are often the most severe sequelae of electrical burns after cardiac arrhythmias, which may initially appear minor despite significant deep tissue injury subsequently requiring fasciotomy or amputation. Burns are often most severe at the source and ground contact points; the source is usually the hands or the head while the ground is often in the feet. The strength and duration of contact with the source largely influence severity and extent of tissue damage. All burns should be carefully documented and, if possible, photographed.
    • High-voltage electrothermal burns: Typically, these show a contact point and ground point: where the person touched the circuit and where he or she was grounded. These may produce significant damage to underlying tissue while largely sparing the surface of the skin. These burns may appear as painless, depressed areas with central necrosis and minimal bleeding. The presence of surface burns does not accurately predict the extent of possible internal injuries, as skin with high resistance will transmit energy to deeper tissues with lower resistance. A high-voltage burn is shown below. High-voltage electrical burns to the chest. High-voltage electrical burns to the chest.
    • Arc burns: When an arc of current passes from an object of high to low resistance, it creates a high temperature pathway that causes skin lesions at the site of contact with the source and at the ground contact point (not always the feet). These areas typically have a dry parchment center and a rim of congestion around them. There will be clues to the internal pathway taken by the arc based on the location of these surface wounds. Arcs can also cause electrothermal, flash, and flame burns, so multiple burns of varying appearance may be observed. Arcs do not occur in low-voltage injuries. An arc burn is shown below. Arcing electrical burns through the shoe around thArcing electrical burns through the shoe around the rubber sole. High-voltage (7600 V) alternating current nominal. Note cratering.
    • Flash burns: Flash burns are caused by heat from a nearby electrical arc that can reach upwards of 5000o C. These can pass over the surface of the body or through, depending on the path of the arc causing the flash. They may "splash" over the surface of the body, resulting in diffuse but relatively superficial partial-thickness burns. There is no internal electrical component. A flash burn is shown below. Superficial electrical burns to the knees (flash/fSuperficial electrical burns to the knees (flash/ferning).
    • Flame burns: Flame burns are caused by ignition of clothing or nearby objects. These cause thermal burns similar to other flame burns.
    • Low-voltage burns: These behave like ordinary thermal burns and range from local erythema to full-thickness burns. These require several seconds of contact to cause skin burns, sometimes reaching current levels high enough to cause VF before causing any significant skin damage.[4] Direct contact burns may occur only if the circuit through the person was prolonged for more than a few seconds. Low-voltage burns are shown below. Energized site of low-voltage electrical burn in aEnergized site of low-voltage electrical burn in a 50-year-old electrician. Grounded sites of a low-voltage injury in a 33-yeaGrounded sites of a low-voltage injury in a 33-year-old male suicide patient.
    • Contact burns: Contact burns usually have a pattern from the contacted item (branding) and may appear similar to flash burns. To differentiate them, full-thickness contact burns have unburned surface hair, whereas flash burns singe the hairs, which are largely gone by the time the patient presents to the ED. A contact burn is shown below. Contact electrical burn. This was the ground of a Contact electrical burn. This was the ground of a 120-V alternating current nominal circuit. Note vesicle with surrounding erythema. Note thermal and contact electrical burns cannot be distinguished easily.
    • Pediatric oral burns: These are most commonly encountered in children younger than 6 years who bite or suck on a household electrical cord. A local arc of current crosses from one side of the mouth to the other. The orbicularis oris muscle may be involved, and cosmetic deformity of the lips may occur if the burn crosses the commissure. Significant edema may be noted and within 2-3 days eschar formation. Life-threatening bleeding can occur at 2-3 weeks post injury if the labial artery is exposed when the eschar falls off. Initial presentations may underestimate the extent of the ultimate injury; patients require aggressive airway management.[9] These patients should be referred for early follow-up to a burn specialist, plastic surgeon, and an oral surgeon.
  • Neurologic
    • Most acute CNS or spinal deficits resulting from electrical injuries are due to secondary blunt trauma or burns. Often, the patient has transient confusion, amnesia, and impaired recall of events if not frank loss of consciousness. Direct effects of electrical current are most severe if the respiratory control center of the brainstem is affected resulting in respiratory arrest. Current may also cause seizure or direct spinal cord injury if there is hand-to-hand flow. Spinal cord injury can result from direct current effects or blunt trauma. Unless a patient is completely lucid with full recollection of the events, initial C-spine immobilization is indicated.
    • Currents cause acute muscle tetany at relatively low currents and frequencies, like those found in most households. Muscle tetany causes victims to grasp the source, prolonging contact time, and can also paralyze respiratory muscles resulting in asphyxiation.
    • Long-term neurologic complications include seizures, peripheral nerve damage, delayed spinal cord syndromes, and psychiatric problems from depression to aggressive behavior.
  • Musculoskeletal: Acute injuries include fracture from blunt trauma and compartment syndrome from burns. The chest and any extremity should be examined for circumferential burn. Palpate the extremity and perform distal neurologic, vascular, and motor examination to determine if there is suspicion of a compartment syndrome. If this is the case, compartment pressure can be measured and early fasciotomy may help prevent subsequent amputation.[11] If available, surgical consultation should be obtained early for a patient with these concerns or for appropriate trauma consultation. Massive muscle damage can cause severe rhabdomyolysis and subsequent renal failure.
  • ENT/head: The head is a common point of entry for high-voltage injuries. Patients may have perforated tympanic membranes, facial burn, and cervical spine injury. Approximately 6% of victims develop cataracts, usually months after the initial injury, with increasing frequency the closer contact is to the head.[24, 10]
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Causes

Electrical injury occurs when a person becomes part of an electrical circuit or is affected by the thermal effects of a nearby electrical arc. Injuries are caused by high-voltage AC, low-voltage AC, or DC.

  • High-voltage AC: High-voltage injuries most commonly occur from a conductive object touching an overhead high-voltage power line. In the United States, most electric power is distributed and transmitted by bare aluminum or copper conductors, which are insulated by air. If the air is breached by a conductor, (eg, an aluminum pole, antenna, sailboat mast, crane), any person touching the conductor can be injured. Occupational injuries may include direct contact with electrical switching equipment and energized components.
  • Low-voltage AC: Generally, 2 types of low-voltage injury occur: children biting into electrical cords producing severe lip, face, and tongue injuries, or the adult who becomes grounded while touching an appliance or other object that is energized. The latter type of injury is decreasing with the increasing use of ground fault circuit interrupters (GFCIs) in circuits where people might easily become grounded. GFCIs stop current flow in the event of a leakage current (ground fault) if the ground fault is greater than 0.005 amps (0.6 W at 120 V).
  • Direct current (DC): DC injuries are generally encountered when the third energized rail of an electrical train system is contacted while the person is grounded. This sets up a circuit of electric current through the victim, causing severe electrothermal burns and myonecrosis.[2]
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Contributor Information and Disclosures
Author

Tracy A Cushing, MD, MPH, FACEP, FAWM  Assistant Professor, Department of Emergency Medicine, University of Colorado School of Medicine; Attending Physician, Denver Health Medical Center

Tracy A Cushing, MD, MPH, FACEP, FAWM is a member of the following medical societies: American College of Emergency Physicians, Society for Academic Emergency Medicine, and Wilderness Medical Society

Disclosure: Nothing to disclose.

Coauthor(s)

Ronald K Wright, MD, JD  Associate Professor (Retired), Department of Pathology, University of Miami School of Medicine; Private Practice, Forensic Pathology

Ronald K Wright, MD, JD is a member of the following medical societies: American Academy of Forensic Sciences, American Medical Association, American Society for Clinical Pathology, College of American Pathologists, and National Association of Medical Examiners

Disclosure: Nothing to disclose.

Specialty Editor Board

Jerry Balentine, DO  Professor of Emergency Medicine, New York College of Osteopathic Medicine; Executive Vice President, Chief Medical Officer, Attending Physician in Department of Emergency Medicine, St Barnabas Hospital

Jerry Balentine, DO is a member of the following medical societies: American College of Emergency Physicians, American College of Osteopathic Emergency Physicians, American College of Physician Executives, American Osteopathic Association, and New York Academy of Medicine

Disclosure: Nothing to disclose.

Francisco Talavera, PharmD, PhD  Adjunct Assistant Professor, University of Nebraska Medical Center College of Pharmacy; Editor-in-Chief, Medscape Drug Reference

Disclosure: Medscape Salary Employment

Eric L Legome, MD  Chief, Department of Emergency Medicine, Kings County Hospital Center; Associate Professor, Department of Emergency Medicine, New York Medical College

Eric L Legome, MD is a member of the following medical societies: Alpha Omega Alpha, American Academy of Emergency Medicine, American College of Emergency Physicians, Council of Emergency Medicine Residency Directors, and Society for Academic Emergency Medicine

Disclosure: Nothing to disclose.

John D Halamka, MD, MS  Associate Professor of Medicine, Harvard Medical School, Beth Israel Deaconess Medical Center; Chief Information Officer, CareGroup Healthcare System and Harvard Medical School; Attending Physician, Division of Emergency Medicine, Beth Israel Deaconess Medical Center

John D Halamka, MD, MS is a member of the following medical societies: American College of Emergency Physicians, American Medical Informatics Association, Phi Beta Kappa, and Society for Academic Emergency Medicine

Disclosure: Nothing to disclose.

Chief Editor

Rick Kulkarni, MD  Attending Physician, Department of Emergency Medicine, Cambridge Health Alliance, Division of Emergency Medicine, Harvard Medical School

Rick Kulkarni, MD is a member of the following medical societies: Alpha Omega Alpha, American Academy of Emergency Medicine, American College of Emergency Physicians, American Medical Association, American Medical Informatics Association, Phi Beta Kappa, and Society for Academic Emergency Medicine

Disclosure: WebMD Salary Employment

References
  1. Lee RC. Injury by electrical forces: pathophysiology, manifestations, and therapy. Curr Probl Surg. Sep 1997;34(9):677-764. [Medline].

  2. Rabban J, Adler J, Rosen C, Blair J, Sheridan R. Electrical injury from subway third rails: serious injury associated with intermediate voltage contact. Burns. Sep 1997;23(6):515-8. [Medline].

  3. Spies C, Trohman RG. Narrative review: Electrocution and life-threatening electrical injuries. Ann Intern Med. Oct 3 2006;145(7):531-7. [Medline].

  4. Koumbourlis AC. Electrical injuries. Crit Care Med. Nov 2002;30(11 Suppl):S424-30. [Medline].

  5. Price T, Cooper MA. Electrical and Lighting Injuries. In: Marx J, Hockberger R, Walls R. Rosen's Emergency Medicine. Vol 3. 5th ed. Mosby; 2002:2010-2020.

  6. Rai J, Jeschke MG, Barrow RE, Herndon DN. Electrical injuries: a 30-year review. J Trauma. May 1999;46(5):933-6. [Medline].

  7. Casini V. Worker Deaths by Electrocution: A summary of NIOSH Surveillance and Investigative Findings. Washington DC: Department of Health and Human Services (NIOSH); May 1998. 5-8.

  8. Luz DP, Millan LS, Alessi MS, et al. Electrical burns: a retrospective analysis across a 5-year period. Burns. Nov 2009;35(7):1015-9. [Medline].

  9. Thomas SS. Electrical burns of the mouth: still searching for an answer. Burns. Mar 1996;22(2):137-40. [Medline].

  10. Ferreiro I, Melendez J, Regalado J, Bejar FJ, Gabilondo FJ. Factors influencing the sequelae of high tension electrical injuries. Burns. Nov 1998;24(7):649-53. [Medline].

  11. Kopp J, Loos B, Spilker G, Horch RE. Correlation between serum creatinine kinase levels and extent of muscle damage in electrical burns. Burns. Nov 2004;30(7):680-3. [Medline].

  12. Hussmann J, Kucan JO, Russell RC, Bradley T, Zamboni WA. Electrical injuries--morbidity, outcome and treatment rationale. Burns. Nov 1995;21(7):530-5. [Medline].

  13. Baker MD, Chiaviello C. Household electrical injuries in children. Epidemiology and identification of avoidable hazards. Am J Dis Child. Jan 1989;143(1):59-62. [Medline].

  14. Rabban JT, Blair JA, Rosen CL, Adler JN, Sheridan RL. Mechanisms of pediatric electrical injury. New implications for product safety and injury prevention. Arch Pediatr Adolesc Med. Jul 1997;151(7):696-700. [Medline].

  15. Ordog GJ, Wasserberger J, Schlater T, Balasubramanium S. Electronic gun (Taser) injuries. Ann Emerg Med. Jan 1987;16(1):73-8. [Medline].

  16. Sloane CM, Chan TC, Levine SD, Dunford JV, Neuman T, Vilke GM. Serum troponin I measurement of subjects exposed to the Taser X-26. J Emerg Med. Jul 2008;35(1):29-32. [Medline].

  17. Cevik C, Otahbachi M, Miller E, Bagdure S, Nugent KM. Acute stress cardiomyopathy and deaths associated with electronic weapons. Int J Cardiol. Mar 6 2009;132(3):312-7. [Medline].

  18. Bozeman WP, Hauda WE 2nd, Heck JJ, Graham DD Jr, Martin BP, Winslow JE. Safety and injury profile of conducted electrical weapons used by law enforcement officers against criminal suspects. Ann Emerg Med. Apr 2009;53(4):480-9. [Medline].

  19. Strote J, Walsh M, Angelidis M, Basta A, Hutson HR. Conducted Electrical Weapon Use by Law Enforcement: An Evaluation of Safety and Injury. J Trauma. Dec 22 2009;[Medline].

  20. Robb M, Close B, Furyk J, Aitken P. Review article: Emergency Department implications of the TASER. Emerg Med Australas. Aug 2009;21(4):250-8. [Medline].

  21. Jensen PJ, Thomsen PE, Bagger JP, Norgaard A, Baandrup U. Electrical injury causing ventricular arrhythmias. Br Heart J. Mar 1987;57(3):279-83. [Medline].

  22. Dawes DM, Ho JD, Reardon RF, Miner JR. Echocardiographic evaluation of TASER X26 probe deployment into the chests of human volunteers. Am J Emerg Med. Jan 2010;28(1):49-55. [Medline].

  23. Claudet I, Marechal C, Debuisson C, Salanne S. [Risk of arrhythmia and domestic low-voltage electrical injury.]. Arch Pediatr. Feb 26 2010;[Medline].

  24. Saffle JR, Crandall A, Warden GD. Cataracts: a long-term complication of electrical injury. J Trauma. Jan 1985;25(1):17-21. [Medline].

  25. Haberal MA. An eleven-year survey of electrical burn injuries. J Burn Care Rehabil. Jan-Feb 1995;16(1):43-8. [Medline].

  26. Rosen CL, Adler JN, Rabban JT, et al. Early predictors of myoglobinuria and acute renal failure following electrical injury. J Emerg Med. Sep-Oct 1999;17(5):783-9. [Medline].

  27. Bailey B, Gaudreault P, Thivierge RL. Cardiac monitoring of high-risk patients after an electrical injury: a prospective multicentre study. Emerg Med J. May 2007;24(5):348-52. [Medline].

  28. Dollery W. Towards evidence based emergency medicine: best BETs from the Manchester Royal infirmary. Management of household electrical injury. J Accid Emerg Med. Jul 1998;15(4):228. [Medline].

  29. Chen EH, Sareen A. Do children require ECG evaluation and inpatient telemetry after household electrical exposures?. Ann Emerg Med. Jan 2007;49(1):64-7. [Medline].

  30. Bracken TD, Kavet R, Patterson RM, Fordyce TA. An integrated job exposure matrix for electrical exposures of utility workers. J Occup Environ Hyg. Aug 2009;6(8):499-509. [Medline].

  31. Achauer BM, Martinez SE. Burn wound pathophysiology and care. Crit Care Clin. Mar 1985;1(1):47-58. [Medline].

  32. Bailey B, Gaudreault P, Thivierge RL. Cardiac monitoring of high-risk patients after an electrical injury: a prospective multicentre study. Emerg Med J. May 2007;24(5):348-52. [Medline].

  33. Bailey B, Gaudreault P, Thivierge RL. Neurologic and neuropsychological symptoms during the first year after an electric shock: results of a prospective multicenter study. Am J Emerg Med. May 2008;26(4):413-8. [Medline].

  34. Baxter CR, Waeckerle JF. Emergency treatment of burn injury. Ann Emerg Med. Dec 1988;17(12):1305-15. [Medline].

  35. Canady JW, Thompson SA, Bardach J. Oral commissure burns in children. Plast Reconstr Surg. Apr 1996;97(4):738-44; discussion 745; 746-55. [Medline].

  36. Colic M, Ristic L, Jovanovic M. Emergency treatment and early fluid resuscitation following electrical injuries. Acta Chir Plast. 1996;38(4):137-41. [Medline].

  37. Cooper MA. Electrical and lightning injuries. Emerg Med Clin North Am. Aug 1984;2(3):489-501. [Medline].

  38. El-Badawy A, Mabrouk AR. Epidemiology of childhood burns in the burn unit of Ain Shams University in Cairo, Egypt. Burns. Dec 1998;24(8):728-32. [Medline].

  39. Fish RM. Electric injury, part I: treatment priorities, subtle diagnostic factors, and burns. J Emerg Med. Nov-Dec 1999;17(6):977-83. [Medline].

  40. Fish RM. Electric injury, part III: cardiac monitoring indications, the pregnant patient, and lightning. J Emerg Med. Feb 2000;18(2):181-7. [Medline].

  41. Gomez R, Cancio LC. Management of burn wounds in the emergency department. Emerg Med Clin North Am. Feb 2007;25(1):135-46. [Medline].

  42. Hammond JS, Ward CG. High-voltage electrical injuries: management and outcome of 60 cases. South Med J. Nov 1988;81(11):1351-2. [Medline].

  43. Leibovici D, Shemer J, Shapira SC. Electrical injuries: current concepts. Injury. Nov 1995;26(9):623-7. [Medline].

  44. Milano M. Oral electrical and thermal burns in children: review and report of case. ASDC J Dent Child. Mar-Apr 1999;66(2):116-9, 85. [Medline].

  45. Singerman J, Gomez M, Fish JS. Long-term sequelae of low-voltage electrical injury. J Burn Care Res. Sep-Oct 2008;29(5):773-7. [Medline].

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Arcing electrical burns through the shoe around the rubber sole. High-voltage (7600 V) alternating current nominal. Note cratering.
Contact electrical burn. This was the ground of a 120-V alternating current nominal circuit. Note vesicle with surrounding erythema. Note thermal and contact electrical burns cannot be distinguished easily.
Contact electrical burns, 120-V alternating current nominal. The right knee was the energized side, and the left was ground. These are contact burns and are difficult to distinguish from thermal burns. Note entrance and exit are not viable concepts in alternating current.
Electrical burns to the hand.
Electrical burns to the foot.
High-voltage electrical burns to the chest.
Superficial electrical burns to the knees (flash/ferning).
Energized site of low-voltage electrical burn in a 50-year-old electrician.
Grounded sites of high-voltage injury on the chest of a 16-year-old boy who climbed up an electric pole.
Energized site of the high-voltage injury depicted in Media File 9 (16-year-old boy who climbed up an electric pole).
Entrance site of a low-voltage injury.
Grounded sites of a low-voltage injury in a 33-year-old male suicide patient.
Grounded site of a low-voltage injury in the same 33-year-old male patient depicted in Media File 12.
Grounded sites of low-voltage injury on the feet.
A histologic picture of an electrical burn showing elongated pyknotic keratinocyte nuclei with vertical streaming and homogenization of the dermal collagen (40X). Courtesy of Elizabeth Satter, MD.
Table 1. Physiologic Effects of Different Electrical Currents
Effect Current (milliamps)
Tingling sensation/perception1-4
Let-go current – Children3-4
Let-go current - Women6-8
Let-go current – Men7-9
Skeletal muscle tetany16-20
Respiratory muscle paralysis20-50
Ventricular fibrillation50-120
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