eMedicine Specialties > Emergency Medicine > Trauma & Orthopedics

Cervical Strain: Treatment & Medication

Author: Warren Magnus, DO, Staff Physician, Fremont Medical Center, Las Vegas, NV
Coauthor(s): Paul D Moczarski, DO, Attending Physician, Emergency Resources Group and EmCare, Jacksonville, Florida
Contributor Information and Disclosures

Updated: Apr 7, 2009

Treatment

Prehospital Care

All persons involved in MVAs who sustain neck injuries should, at a minimum, receive cervical collars prior to transport. Many emergency medical service (EMS) protocols require these patients to be placed on a backboard in full spinal precautions. Because MVAs often involve enough force to seriously injure the cervical spine, such precautions are essential to prevent further injury.

Emergency Department Care

  • Apply ice to acute strain injuries.
  • Administer analgesia and pain control.
  • Administer muscle relaxants.
  • Soft collars are commonly used but have not been proven effective. A single-blind study with 6-month follow-up conducted by Borchgrevink et al found that patients who received "usual care," with early mobilization and pain control, fared better than similarly treated patients placed in soft collars.2 This was reinforced by Kongsted et al in 2007.3

Consultations

At various times over the last 2 decades, there have been various calls for early multidisciplinary management including aggressive early physical therapy and other modalities. Current consensus, however, shows that consultations are rarely required for strain injuries; however, follow-up with a physician familiar with rehabilitation therapies is essential for longer-term management, particularly for patients who have experienced an occupational injury.

Medication

The pharmacology of cervical strain involves pain control and palliation. Nonsteroidal anti-inflammatory drugs (NSAIDs) and acetaminophen (Tylenol) are mainstays of therapy. Muscle relaxants may prove valuable when treating severe strain injuries to reduce pain and muscle contracture.

Analgesics

For minor strain injuries, oral outpatient analgesics provide adequate pain control. OTC medications also may suffice.


Acetaminophen (Tylenol, Panadol, Aspirin Free Anacin)

Rapidly absorbed from GI tract and distributed widely to all body tissues. Serum half-life is 1-3 h but may be altered in impaired liver function. Posthepatic metabolites excreted in urine.

Adult

375-650 mg PO q4-6h prn or 1000 mg PO q6-8h prn

Pediatric

15 mg/kg/dose PO q4-6h prn

Rifampin can reduce analgesic effects; possible increase in hepatotoxicity with use of barbiturates, carbamazepine, hydantoins, or isoniazid

Documented hypersensitivity; known G-6-PD deficiency

Pregnancy

B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals

Precautions

Hepatotoxicity possible in chronic alcoholics following various dose levels of acetaminophen; severe or recurrent pain or high or continued fever possible indication of serious illness

Nonsteroidal anti-inflammatory agents

NSAIDs control mild to moderate pain and decrease inflammatory reactions. This entire family of medications may ease pain in strain injuries. Tailor dosage on an individual basis.


Ibuprofen (Ibuprin, Advil, Motrin)

Rapidly absorbed orally and distributed widely through body tissues. Serum half-life is 1.8-2 h. Rapidly metabolized and excreted in urine. Complete clearance of single dose occurs in approximately 24 h.

Adult

400-600 mg PO q4-6h prn or 800 mg PO q8h prn

Pediatric

Not recommended for pain control; used as antipyretic

Coadministration with aspirin increases risk of inducing serious NSAID-related adverse effects; probenecid may increase concentrations and, possibly, toxicity; may decrease effect of hydralazine, captopril, and beta-blockers; may decrease diuretic effects of furosemide and thiazides; may increase PT in patients on anticoagulants (instruct patients to watch for signs of bleeding); may increase risk of methotrexate toxicity; may increase phenytoin levels

Documented hypersensitivity; hypersensitivity to aspirin, iodides, or other NSAIDs; peptic ulcer disease; recent GI bleeding or perforation; renal insufficiency; high risk of bleeding

Pregnancy

C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus

D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus

Precautions

Caution in congestive heart failure, hypertension, and decreased renal or hepatic function; caution in anticoagulation abnormalities or during anticoagulant therapy (monitor PT closely); monitor for signs of bleeding


Ketorolac tromethamine (Toradol)

Provides effective control of moderate to severe pain, with higher potency than other NSAIDs, which results in more marked GI upset, platelet inhibition, and renal effects.

Adult

10 mg PO q4-6h prn; not to exceed 40 mg/d
30 mg IV q6h prn
30-60 mg IM q6h prn; repeat doses should be at the 30 mg IM level

Pediatric

Not established

Coadministration with aspirin increases risk of inducing serious NSAID-related adverse effects; probenecid may increase concentrations and, possibly, toxicity; may decrease effect of hydralazine, captopril, and beta-blockers; may decrease diuretic effects of furosemide and thiazides; may increase PT in patients on anticoagulants (instruct patients to watch for signs of bleeding); may increase risk of methotrexate toxicity; may increase phenytoin levels

Documented hypersensitivity; not to be administered into CNS; peptic ulcer disease; recent GI bleeding or perforation; renal insufficiency; high risk of bleeding

Pregnancy

C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus

D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus

Precautions

Acute renal insufficiency, hyperkalemia, hyponatremia, interstitial nephritis, and renal papillary necrosis possible; increases risk of acute renal failure in patients with preexisting renal disease or compromised renal perfusion; may increase risk of bleeding (monitor patient for signs of bleeding); low WBC counts (rare) usually return to normal during ongoing therapy; discontinue therapy if leukopenia, granulocytopenia, or thrombocytopenia persists

Muscle relaxants

These agents provide adjunctive therapy to allow rest, control pain, and aid physical therapy for musculoskeletal injury.


Orphenadrine citrate (Norflex)

Action not well understood, but its analgesic properties make it clinically effective for muscular injury.

Adult

100 mg PO bid prn
60 mg IM q12h prn

Pediatric

Not established

Documented hypersensitivity; GI obstruction; glaucoma; myasthenia gravis; cardiospasm

Pregnancy

C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus

Precautions

Caution in cardiac arrhythmias, anxiety, hemodynamic instability, tremors, confusion, and congestive heart failure


Cyclobenzaprine hydrochloride (Flexeril)

Centrally acting skeletal muscle relaxant structurally related to TCAs with similar liabilities. Can be useful adjunct to other therapies for acute musculoskeletal pain.

Adult

10 mg PO tid prn

Pediatric

Not established

Coadministration with MAOIs or TCAs may increase toxicity; may have additive effect when used concurrently with anticholinergics; may enhance effects of alcohol, CNS depressants, and barbiturates

Documented hypersensitivity; MAOIs within last 14 d; hyperthyroidism

Pregnancy

B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals

Precautions

Caution in angle-closure glaucoma or urinary hesitancy; may impair consciousness and ability to operate machinery

More on Cervical Strain

Overview: Cervical Strain
Differential Diagnoses & Workup: Cervical Strain
Treatment & Medication: Cervical Strain
Follow-up: Cervical Strain
Multimedia: Cervical Strain
References

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Further Reading

Keywords

cervical strain, neck strain, neck pain, whiplash, whiplash neck sprain, hyperextension strain to the cervical spine, cervical spine injuries, cervical spine strain injuries, edema of cervical tissue, radicular pain in cervical spine injuries, rapid cervical strain injuries, low-velocity cervical strain injuries

Contributor Information and Disclosures

Author

Warren Magnus, DO, Staff Physician, Fremont Medical Center, Las Vegas, NV
Warren Magnus, DO is a member of the following medical societies: American Osteopathic Association
Disclosure: Nothing to disclose.

Coauthor(s)

Paul D Moczarski, DO, Attending Physician, Emergency Resources Group and EmCare, Jacksonville, Florida
Paul D Moczarski, DO is a member of the following medical societies: American Osteopathic Association
Disclosure: Nothing to disclose.

Medical Editor

David FM Brown, MD, Assistant Professor, Department of Medicine, Division of Emergency Medicine, Harvard Medical School; Associate-Chief, Attending Physician, Department of Emergency Medicine, Massachusetts General Hospital
David FM Brown, MD is a member of the following medical societies: American College of Emergency Physicians and Society for Academic Emergency Medicine
Disclosure: Schering  Honoraria Speaking and teaching

Pharmacy Editor

Francisco Talavera, PharmD, PhD, Senior Pharmacy Editor, eMedicine
Disclosure: Nothing to disclose.

Managing Editor

Eric Legome, MD, Chair, Department of Emergency Medicine, St Vincent's Hospital, Manhattan
Eric Legome, MD is a member of the following medical societies: Alpha Omega Alpha, American Academy of Emergency Medicine, American College of Emergency Physicians, Council of Emergency Medicine Residency Directors, and Society for Academic Emergency Medicine
Disclosure: Nothing to disclose.

CME Editor

John D Halamka, MD, MS, Associate Professor of Medicine, Harvard Medical School, Beth Israel Deaconess Medical Center; Chief Information Officer, CareGroup Healthcare System and Harvard Medical School; Attending Physician, Division of Emergency Medicine, Beth Israel Deaconess Medical Center
John D Halamka, MD, MS is a member of the following medical societies: American College of Emergency Physicians, American Medical Informatics Association, Phi Beta Kappa, and Society for Academic Emergency Medicine
Disclosure: Nothing to disclose.

Chief Editor

Rick Kulkarni, MD, Medical Director, Assistant Professor of Surgery, Section of Emergency Medicine, Yale-New Haven Hospital
Rick Kulkarni, MD is a member of the following medical societies: Alpha Omega Alpha, American Academy of Emergency Medicine, American College of Emergency Physicians, American Medical Association, American Medical Informatics Association, Phi Beta Kappa, and Society for Academic Emergency Medicine
Disclosure: WebMD Salary Employment

 
 
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