Updated: Aug 22, 2008
Hand injuries are commonly seen in the emergency department. Emergency physicians should be able to identify and manage digital dislocations. Complications can occur if the diagnosis is missed or delayed or if the joint is incompletely reduced or splinted improperly.
Patients should be referred to a hand specialist following treatment of hand dislocations.
Traumatic force to the hand can be transmitted to bone, soft tissue, nerves, and vascular structures. Because the structures of the hand are close to the surface and near each other, injury often results in a combination of fractures, dislocations, and soft tissue injury.
The distal interphalangeal (DIP) and proximal interphalangeal (PIP) joints both have lateral ligaments and a fibrous volar plate. Common dislocations are posterior or lateral. Typical forces resulting in DIP dislocations include a “jamming” blow to the end of the finger. The forces that commonly lead to dislocation of the PIP joint include an axial load or hyperextension. Lateral dislocations can result from radial- or ulnar-directed force on the joint.
Finger metacarpophalangeal (MCP) dislocations are rare and frequently are trapped by the surrounding ligaments, necessitating surgical relocation. MCP or palmar dislocations occur when a hyperextension movement occurs with rotation. The finger is bent back toward the top of the hand and is twisted during the injury. The finger may have been pushed, or compressed, during the injury. MCP dislocations are typically associated with fractures.
In thumb metacarpophalangeal (MCP) joint dislocations, the mechanism encountered most often is hyperextension that leads to volar dislocations. A significant lateral force can disrupt the collateral ligaments, resulting in instability. The gamekeeper's (skier's) thumb often results from a fall onto the hand with the thumb in abduction, such as when gripping a ski pole.Carpometacarpal dislocations are not always high-energy injuries. Identification involves careful analysis of subtle findings on radiographs and may require additional radiographic views. Missed diagnosis of carpometacarpal dislocation can result in significant morbidity.
The annual incidence of all types of dislocations in the hand is approximately 67,000 in the United States.
Anatomical restoration of dislocated joints is imperative to achieve good long-term outcomes. Accurate and stable reduction, early fixation, and initiating range of motion exercise are very important. Dislocations can lead to osteoarthritis, compression neuropathies, and carpal tunnel syndrome. Additional disability from chondrolysis, carpal instability, and traumatic arthritis can also occur.
Pediatric transcarpal fractures in children are rare, but the emergency physician must be cognizant that they do occur.
Historical data about the mechanism of trauma can help lead the emergency physician’s assessment of hand injuries.
Arthritis, Rheumatoid
Fractures, Hand
Gamekeeper Thumb
Hand Injury, Soft Tissue
Rapid pain relief, securing imaging studies, and expeditious reduction (closed or open) must be the triad that drives the emergency department protocol for treating isolated digit or hand dislocations.
Triage/initial evaluation and treatment
Once a hand dislocation is identified and pain control in initiated, then relocation is the next step in patient care. Most hand dislocations are easily reduced by the emergency physician. Some dislocations may not be reducible by closed means because of the interposition of the volar plate or associated ligaments or tendons in the joint. If several attempts at reduction are not successful, consultation and open reduction and internal fixation (ORIF) often is indicated. A thorough assessment of stability should be performed following a successful reduction.
Specific reductions
Complex and open dislocations should be evaluated by a hand surgeon for open reduction. In addition, those individuals with fracture-related dislocation require further evaluation by a hand surgeon.
Near immediate pain relief can be provided when the patient receives an injection of a local anesthetic along the path of the digital nerve, also known as a digital block, web-space, or palmar block. Of course, the digital nerve block must follow a very thorough neurosensory examination and (when indicated) discussion with the hand specialist.
Oral medications should be prescribed for the patient who is being discharged from the emergency department. Medications types may include nonsteroidal anti-inflammatory drugs with or without opiates.
Pain control is essential to quality patient care. It ensures patient comfort, promotes pulmonary toilet, and aids physical therapy regimens. Many analgesics have sedating properties that benefit patients who have sustained injuries.
Amide local anesthetic used in 1-2% concentration. Inhibits depolarization of type C sensory neurons by blocking sodium channels.
Must be used without epinephrine for digital block.
Local anesthetic injection can be improved:
Use smaller gauge needles, such as 27 gauge or 30 gauge. Make sure the solution is at body temperature. Infiltrate very slowly to minimize the pain.
Use buffered lidocaine. Buffering solution is effective in reducing pain of local lidocaine injection.
Sodium bicarbonate can be added to injectable lidocaine vial to produce "buffered" lidocaine.
Shelf-life of buffered lidocaine is approximately 1 wk.
All vials should be marked "buffered" and labeled with the time and date and signed by individual who created the buffered mixture.
Add ratio of 1 part bicarbonate to 9 parts lidocaine.
Stable at room temperature for 1 wk.
Cool skin before injection with ethyl chloride.
Use "imaging" discussion during the injection.
Time from administration to onset of action is 2-5 min with a duration of 1.5-2 h.
1% Xylocaine contains 10 mg of lidocaine for each 1 mL of solution.
2% Xylocaine contains 20 mg of lidocaine for each 1 mL of solution.
3 mg/kg injection locally
Administer as in adults
Coadministration with cimetidine or beta-blockers, increases toxicity of lidocaine; coadministration with procainamide and tocainide may result in additive cardiodepressant action; may increase effects of succinylcholine
Documented hypersensitivity to amide-type local anesthetics; avoid in Adams-Stokes syndrome and Wolff-Parkinson-White syndrome; avoid in severe sinoatrial, atrioventricular (AV), or intraventricular block, if artificial pacemaker not in place
B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals
Use a solution without preservatives; caution in heart failure, hepatic disease, hypoxia, hypovolemia or shock, respiratory-depression and bradycardia; may increase risk of CNS and cardiac side effects in elderly persons; high plasma concentrations can cause seizures, heart block, and AV conduction abnormalities; must be used without epinephrine for digital blocks
Drug combination indicated for relief of moderately severe to severe pain.
1-2 tab or cap PO q4-6h prn
<12 years: 10-15 mg/kg/dose acetaminophen PO q4-6h prn; not to exceed 2.6 g/d of acetaminophen or 5 mg hydrocodone bitartrate/dose
>12 years: 750 mg acetaminophen PO q4h; not to exceed 5 doses/d acetaminophen or 10 mg hydrocodone bitartrate/dose
Phenothiazines may decrease analgesic effects; CNS depressants or tricyclic antidepressants may increase toxicity
Documented hypersensitivity; high-altitude cerebral edema; elevated intracranial pressure
C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus
Tablets contain metabisulfite, which may cause hypersensitivity; caution in patients dependent on opiates because this substitution may result in acute opiate-withdrawal symptoms; caution in severe renal or hepatic dysfunction
Drug combination indicated for relief of moderately severe to severe pain. DOC for aspirin-hypersensitive patients. Different strengths available.
1-2 tab or cap PO q4-6h prn
0.05-0.15 mg/kg/dose oxycodone PO q4-6h prn; not to exceed 5 mg/dose of oxycodone
Phenothiazines may decrease analgesic effects; CNS depressants or tricyclic antidepressants may increase toxicity
Documented hypersensitivity
C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus
Duration of action may increase in elderly persons; be aware of total daily dose of acetaminophen patient is receiving; do not exceed 4000 mg/24 h of acetaminophen; higher doses may cause liver toxicity
Drug combination indicated for relief of moderately severe to severe pain.
1-2 tab or cap PO q4-6h prn
0.05-0.15 mg/kg/dose oxycodone PO q4-6h prn; not to exceed 5 mg/dose of oxycodone
Phenothiazines may decrease analgesic effects; CNS depressants or tricyclic antidepressants may increase toxicity; may potentiate anticoagulant effects of warfarin
Documented hypersensitivity; liver damage; hypoprothrombinemia; vitamin K deficiency; bleeding disorders; asthma
Because of association of aspirin with Reye syndrome, do not use in children (<16 y) who have the flu
D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus
Duration of action may increase in elderly persons; caution in renal or liver impairment, peptic ulcer disease, and erosive gastritis
Patients with painful injuries usually experience significant anxiety. Anxiolytics allow the clinician to administer a smaller analgesic dose to achieve the same effect.
Sedative hypnotic in benzodiazepine class that has short onset of effect and relatively long half-life. By increasing action of GABA, a major inhibitory neurotransmitter, may depress all levels of CNS, including limbic and reticular formation. Excellent for patients who need to be sedated for >24 h.
1-10 mg/d PO divided bid/qid; not to exceed 4 mg/dose
0.05-0.1 mg/kg IV slowly over 2-5 min; may repeat a dose of 0.05 mg/kg IV slowly
Alcohol, phenothiazines, barbiturates, or MAOIs increase CNS toxicity
Documented hypersensitivity; preexisting CNS depression; hypotension; narrow-angle glaucoma
D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus
Caution in renal or hepatic impairment, myasthenia gravis, organic brain syndrome, or Parkinson disease
These agents are most commonly used for the relief of mild to moderately severe pain. Although the effects of NSAIDs in the treatment of pain tend to be patient specific, ibuprofen is usually the DOC for initial therapy. Other options include ketoprofen and naproxen.
DOC for treatment of mild to moderately severe pain, if no contraindications. Inhibits inflammatory reactions and pain, probably by decreasing activity of enzyme cyclooxygenase, inhibiting prostaglandin synthesis.
200-400 mg PO q4-6h prn; not to exceed 3.2 g/d
<6 months: Not established
6 months to 12 years: 20-40 mg/kg/d PO divided tid/qid
>12 years: Administer as in adults
Aspirin increases risk of inducing serious NSAID-related adverse effects; probenecid may increase concentrations and, possibly, toxicity; may decrease effects of hydralazine, captopril, and beta-blockers; may decrease diuretic effects of furosemide and thiazides; may increase PT in patients taking anticoagulants—monitor PT regularly and instruct patients to watch for signs of bleeding; may increase risk of methotrexate toxicity; may increase phenytoin levels
Documented hypersensitivity; peptic ulcer disease; recent GI bleeding or perforation; renal insufficiency; high risk of bleeding
B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals
D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus
Caution in CHF, hypertension, and decreased renal or hepatic function; caution in coagulation abnormalities or during anticoagulant therapy
Used for relief of mild to moderately severe pain and inflammation.
Administer small dosages initially to patients with small body size, elderly persons, and those with renal or liver disease. Doses higher than 75 mg do not increase therapeutic effects. Administer high doses with caution, and closely observe patient for response.
25-50 mg PO q6-8h prn; not to exceed 300 mg/d
<3 months: Not established
3 months to 12 years: 0.1–1 mg/kg PO q6-8h
> 12 years: Administer as in adults
Aspirin increases risk of inducing serious NSAID-related adverse effects; probenecid may increase concentrations and, possibly, toxicity; may decrease effects of hydralazine, captopril, and beta-blockers; may decrease diuretic effects of furosemide and thiazides; may increase PT in patients taking anticoagulants—monitor PT regularly and instruct patients to watch for signs of bleeding; may increase risk of methotrexate toxicity; may increase phenytoin levels
Documented hypersensitivity
B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals
D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus
Caution in CHF, hypertension, and decreased renal or hepatic function; caution in coagulation abnormalities or during anticoagulant therapy
Used for relief of mild to moderately severe pain. Inhibits inflammatory reactions and pain by decreasing activity of enzyme cyclooxygenase, which decreases prostaglandin synthesis.
500 mg PO initial dose, followed by 250 mg PO q6-8h; not to exceed 1.25 g/d
<2 years: Not established
>2 years: 2.5 mg/kg/dose PO; not to exceed 10 mg/kg/d
Aspirin increases risk of inducing serious NSAID-related adverse effects; probenecid may increase concentrations and, possibly, toxicity; may decrease effects of hydralazine, captopril, and beta-blockers; may decrease diuretic effects of furosemide and thiazides; may increase PT in patients taking anticoagulants—monitor PT regularly and instruct patients to watch for signs of bleeding; may increase risk of methotrexate toxicity; may increase phenytoin levels
Documented hypersensitivity; peptic ulcer disease; recent GI bleeding or perforation; renal insufficiency
B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals
D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus
Acute renal insufficiency, interstitial nephritis, hyperkalemia, hyponatremia, and renal papillary necrosis may occur; patients with preexisting renal disease or compromised renal perfusion risk acute renal failure; leukopenia occurs rarely, is transient, and usually returns to normal during therapy; persistent leukopenia, granulocytopenia, or thrombocytopenia warrants further evaluation and may require discontinuation of drug
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hand dislocation, dislocated hand, digital dislocations, finger dislocation, wrist dislocation, metacarpophalangeal dislocation, palmar dislocation, thumb metacarpophalangeal joint dislocation, MCP joint dislocation, proximal interphalangeal joint dislocation, PIP joint dislocation, distal interphalangeal joint dislocation, DIP joint dislocation
Trevor John Mills, MD, MPH, Chief of Emergency Medicine, Veterans Affairs Northern California Health Care System; Associate Professor of Emergency Medicine, Louisiana State University Health Sciences Center
Trevor John Mills, MD, MPH is a member of the following medical societies: American Academy of Emergency Medicine, American College of Emergency Physicians, American Public Health Association, Society for Academic Emergency Medicine, Southern Medical Association, and Wilderness Medical Society
Disclosure: Nothing to disclose.
James E Keany, MD, FACEP, Medical Director, JetWest International Air Ambulance; Consulting Staff, Department of Emergency Services, Mission Hospital Regional Medical Center; Host of The Bodcast at Jim.MD
James E Keany, MD, FACEP is a member of the following medical societies: American College of Emergency Physicians, American College of Sports Medicine, and California Medical Association
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Francisco Talavera, PharmD, PhD, Senior Pharmacy Editor, eMedicine
Disclosure: Nothing to disclose.
Eric Legome, MD, Chair, Department of Emergency Medicine, St Vincent's Hospital, Manhattan
Eric Legome, MD is a member of the following medical societies: Alpha Omega Alpha, American Academy of Emergency Medicine, American College of Emergency Physicians, Council of Emergency Medicine Residency Directors, and Society for Academic Emergency Medicine
Disclosure: Nothing to disclose.
John D Halamka, MD, MS, Associate Professor of Medicine, Harvard Medical School, Beth Israel Deaconess Medical Center; Chief Information Officer, CareGroup Healthcare System and Harvard Medical School; Attending Physician, Division of Emergency Medicine, Beth Israel Deaconess Medical Center
John D Halamka, MD, MS is a member of the following medical societies: American College of Emergency Physicians, American Medical Informatics Association, Phi Beta Kappa, and Society for Academic Emergency Medicine
Disclosure: Nothing to disclose.
Rick Kulkarni, MD, Medical Director, Assistant Professor of Surgery, Section of Emergency Medicine, Yale-New Haven Hospital
Rick Kulkarni, MD is a member of the following medical societies: Alpha Omega Alpha, American Academy of Emergency Medicine, American College of Emergency Physicians, American Medical Association, American Medical Informatics Association, Phi Beta Kappa, and Society for Academic Emergency Medicine
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