eMedicine Specialties > Emergency Medicine > Trauma & Orthopedics

Lumbar (Intervertebral) Disk Disorders

Author: Jere F Baldwin, MD, Medical Director, Department of Emergency Medicine, Mercy Hospital Port Huron
Coauthor(s): Jeffrey Horwitz, DO, Director, Assistant Clinical Professor, Department of Emergency Medicine, North Shore University Hospital at Forest Hills
Contributor Information and Disclosures

Updated: Jan 28, 2009

Introduction

Background

Lumbar disk disease is a frequent source of low back pain. Sciatica is defined as neuralgia along the course of the sciatic nerve.

Pathophysiology

The intervertebral disks act as shock absorbers and are found between the bodies of the vertebrae. They have a central area composed of a colloidal gel, called the nucleus pulposus, which is surrounded by a fibrous capsule, the annulus fibrosis. This structure is held together by the anterior longitudinal ligament, which is anterior to the vertebral bodies, and the posterior longitudinal ligament, which is posterior to the vertebral bodies and anterior to the spinal cord. The muscles of the trunk provide additional support.

The most common site of disk herniation is at the L5-S1 interspace in the lumbosacral region. This is believed to be due to the thinning of the posterior longitudinal ligament as it extends caudally.

Nomenclature specific to lumbar disk disease is as follows:

  • Disk bulge - Annular fibers intact
  • Disk protrusion - Localized bulging with damage of some annular fibers
  • Disk extrusion - Extended bulge with loss of annular fibers, but disk remains intact
  • Disk sequestration - Fragment of disk broken off from the nucleus pulposus

Frequency

United States

Sciatica has been reported by various authors to occur in 1-10% of the population.

Mortality/Morbidity

Low back pain usually is self-limited and of short duration.

Sex

The male-to-female ratio is approximately 1:1.

Age

The group most commonly affected is adults aged 25-45 years.

Clinical

History

The history may be sufficient to make presumptive diagnosis of a disk disorder, or it may guide the physician's usage of ancillary testing and consultations to further differentiate both the specific type of disk disease and potential other etiologies of the patient's back pain.

  • Patients with disk disease usually are not able to give a precise time that the problem began because it usually is preceded by multiple episodes of less severe low back pain.
  • Asking the patient the location of the pain is important.
    • Pain that is localized to the lower back and gluteal area often is associated with disk disease.
    • Pain associated with nerve root involvement commonly radiates down the leg, particularly below the level of the knee.
    • Ask the patient about any unusual recent activity, especially if it involved the patient remaining in a flexed or rotated position. Find out if the patient experienced any recent trauma.
    • Pain with flexion, rotation, or prolonged sitting or standing, and sharp (rather than dull) pain are suggestive of disk disease.
    • The onset of pain may begin suddenly1 or gradually after injury.
    • Typically, the pain is located bilaterally at the posterior belt line.
    • The pain pattern usually is referred rather than radicular.
    • Back motion, which includes sitting, standing, lifting, bending, and twisting, usually aggravates the pain; it often is relieved with rest and a recumbent position.

Physical

Nerve roots exit the spine below the intervertebral disks; thus, herniation of a disk involves the nerve root below it.

  • Observe the patient for abnormal gait, which is suggestive of a loss of the normal rhythm. Have ambulatory patients walk on their toes to test the function of S1.
  • Observe the patient for abnormal posture, which is suggestive of splinting or guarding from pain.
  • Test the patient's ability to dorsiflex the foot while sitting to test the L5 nerve root. Test for sensory loss that corresponds to a dermatomal area.
  • Palpation of the lumbar spine and lower back is not helpful in the diagnosis of disk disease, but it should be done to rule out other causes of low back pain.
  • A positive straight leg raising test is indicative of nerve root involvement.
    • This test is performed while the patient is lying supine with one leg either straight or flexed at the knee, with the sole of the foot flat on the stretcher. The other leg is kept straight and lifted by the examiner.
    • If pain occurs when the leg is lifted between 30-70 degrees from horizontal and travels down the leg until below the knee, the test is positive.
  • Nerve root stretch test results are often negative.
  • Patients may exhibit decreased lumbar range of motion (ROM).
  • The usual motor, sensory, and reflex examinations (including perianal sensation and anal sphincter tone when appropriate) should be performed.
  • A careful abdominal and vascular examination is mandatory in evaluation of these patients.

Causes

  • The normal aging process of the musculoskeletal system aggravates acute events.
  • Risk factors
    • Age
    • Activity
    • Smoking
    • Obesity
    • Vibration (eg, driving a car)
    • Sedentary lifestyle
    • Psychosocial factors

More on Lumbar (Intervertebral) Disk Disorders

Overview: Lumbar (Intervertebral) Disk Disorders
Differential Diagnoses & Workup: Lumbar (Intervertebral) Disk Disorders
Treatment & Medication: Lumbar (Intervertebral) Disk Disorders
Follow-up: Lumbar (Intervertebral) Disk Disorders
References

References

  1. Gregory DS, Seto CK, Wortley GC, Shugart CM. Acute lumbar disk pain: navigating evaluation and treatment choices. Am Fam Physician. Oct 1 2008;78(7):835-42. [Medline].

  2. Deen HG Jr. Diagnosis and management of lumbar disk disease. Mayo Clin Proc. Mar 1996;71(3):283-7. [Medline].

  3. Deyo RA. Diagnostic evaluation of LBP: reaching a specific diagnosis is often impossible. Arch Intern Med. Jul 8 2002;162(13):1444-7; discussion 1447-8. [Medline].

  4. Jarvik JG, Hollingworth W, Martin B, et al. Rapid magnetic resonance imaging vs radiographs for patients with low back pain: a randomized controlled trial. JAMA. Jun 4 2003;289(21):2810-8. [Medline].

  5. Jensen MC, Brant-Zawadzki MN, Obuchowski N, et al. Magnetic resonance imaging of the lumbar spine in people without back pain. N Engl J Med. Jul 14 1994;331(2):69-73. [Medline].

  6. Carragee E. Surgical treatment of lumbar disk disorders. JAMA. Nov 22 2006;296(20):2485-7. [Medline].

  7. Deyo RA, Gray DT, Kreuter W, et al. United States trends in lumbar fusion surgery for degenerative conditions. Spine. Jun 15 2005;30(12):1441-5; discussion 1446-7. [Medline].

  8. Dullerud R, Nakstad PH. CT changes after conservative treatment for lumbar disk herniation. Acta Radiol. Sep 1994;35(5):415-9. [Medline].

  9. Weinstein JN, Tosteson TD, Lurie JD, et al. Surgical vs nonoperative treatment for lumbar disk herniation: the Spine Patient Outcomes Research Trial (SPORT): a randomized trial. JAMA. Nov 22 2006;296(20):2441-50. [Medline].

  10. Weinstein JN, Lurie JD, Tosteson TD, et al. Surgical vs nonoperative treatment for lumbar disk herniation: the Spine Patient Outcomes Research Trial (SPORT) observational cohort. JAMA. Nov 22 2006;296(20):2451-9. [Medline].

  11. Frost H, Lamb SE, Doll HA, et al. Randomised controlled trial of physiotherapy compared with advice for low back pain. BMJ. Sep 25 2004;329(7468):708. [Medline].

  12. Gilbert FJ, Grant AM, Gillan MG, et al. Low back pain: influence of early MR imaging or CT on treatment and outcome--multicenter randomized trial. Radiology. May 2004;231(2):343-51. [Medline].

Further Reading

Keywords

lumbar disk disorders, lumbar disk disease, low back pain, sciatica, intervertebral disk disorders, back pain, back pain diagnosis, back pain treatment, back pain pictures, back pain x-rays, sciatic nerve, disk herniation, disk bulge, disk protrusion, disk extrusion, disk sequestration, herniated disk

Contributor Information and Disclosures

Author

Jere F Baldwin, MD, Medical Director, Department of Emergency Medicine, Mercy Hospital Port Huron
Jere F Baldwin, MD is a member of the following medical societies: American Academy of Family Physicians, American College of Emergency Physicians, American Medical Association, Michigan State Medical Society, and Society for Academic Emergency Medicine
Disclosure: Nothing to disclose.

Coauthor(s)

Jeffrey Horwitz, DO, Director, Assistant Clinical Professor, Department of Emergency Medicine, North Shore University Hospital at Forest Hills
Jeffrey Horwitz, DO is a member of the following medical societies: Society for Academic Emergency Medicine
Disclosure: Nothing to disclose.

Medical Editor

Mark S Slabinski, MD, FACEP, FAAEM, Vice President, EMP Medical Group
Mark S Slabinski, MD, FACEP, FAAEM is a member of the following medical societies: Alpha Omega Alpha, American Academy of Emergency Medicine, American College of Emergency Physicians, American Medical Association, and Ohio State Medical Association
Disclosure: Nothing to disclose.

Pharmacy Editor

Francisco Talavera, PharmD, PhD, Senior Pharmacy Editor, eMedicine
Disclosure: Nothing to disclose.

Managing Editor

David B Levy, DO, FACEP, FAAEM, Chairman, Department of Emergency Medicine, St Elizabeth Health Center; Associate Professor of Emergency Medicine, Northeastern Ohio Universities College of Medicine
David B Levy, DO, FACEP, FAAEM is a member of the following medical societies: American Academy of Emergency Medicine, American Medical Informatics Association, and Society for Academic Emergency Medicine
Disclosure: Nothing to disclose.

CME Editor

John D Halamka, MD, MS, Associate Professor of Medicine, Harvard Medical School, Beth Israel Deaconess Medical Center; Chief Information Officer, CareGroup Healthcare System and Harvard Medical School; Attending Physician, Division of Emergency Medicine, Beth Israel Deaconess Medical Center
John D Halamka, MD, MS is a member of the following medical societies: American College of Emergency Physicians, American Medical Informatics Association, Phi Beta Kappa, and Society for Academic Emergency Medicine
Disclosure: Nothing to disclose.

Chief Editor

Rick Kulkarni, MD, Medical Director, Assistant Professor of Surgery, Section of Emergency Medicine, Yale-New Haven Hospital
Rick Kulkarni, MD is a member of the following medical societies: Alpha Omega Alpha, American Academy of Emergency Medicine, American College of Emergency Physicians, American Medical Association, American Medical Informatics Association, Phi Beta Kappa, and Society for Academic Emergency Medicine
Disclosure: WebMD Salary Employment

 
 
HONcode

We subscribe to the
HONcode principles of the
Health On the Net Foundation

All material on this website is protected by copyright, Copyright© 1994- by Medscape.
This website also contains material copyrighted by 3rd parties.

DISCLAIMER: The content of this Website is not influenced by sponsors. The site is designed primarily for use by qualified physicians and other medical professionals. The information contained herein should NOT be used as a substitute for the advice of an appropriately qualified and licensed physician or other health care provider. The information provided here is for educational and informational purposes only. In no way should it be considered as offering medical advice. Please check with a physician if you suspect you are ill.