Peripheral Vascular Injuries Clinical Presentation

  • Author: Niels Rathlev, MD, FACEP; Chief Editor: Rick Kulkarni, MD   more...
 
Updated: Jun 17, 2011
 

History

  • In peripheral vascular injury, the mechanism of injury is an important prognostic factor. Shotgun and military rifle injuries as well as knee dislocations are particularly high risk for vascular injury.
  • The time interval between injury and evaluation must be considered. "Warm" ischemia at body temperature for more than 6 hours results in irreversible nerve and muscle damage in 10% of patients. Cooling the extremity may avoid this complication.
  • Previous history of vascular injury or disease
  • Extensive or pulsatile external hemorrhage
  • Anticoagulation therapy or impaired hemostatic function
  • Prior venous thrombosis or embolism in the patient or a family member
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Physical

Deciding whether the injury requires surgical intervention is a major priority of initial management.

The presence of "hard" signs of vascular injury has a 92-95% sensitivity for injuries requiring intervention. The vast majority of patients exhibiting the following "hard" signs require intervention with a positive predictive value of 95%.

  • Bruit or thrill: These are present in only 45% of patients with an arteriovenous fistula
  • Active or pulsatile hemorrhage
  • Pulsatile or expanding hematoma
  • Signs of limb ischemia and elevated compartment pressure including the 5 "P's:" Pallor, paresthesias, pulse deficit, paralysis, and pain on passive extension of the compartment. Pain on passive extension is the earliest and most sensitive physical finding.
  • Diminished or absent pulses: This is not a sensitive prognostic finding, as up to 25% of patients with major vascular injuries requiring repair have normal pulses distal to the injury.

The following "soft" signs are much less useful in predicting or excluding major vascular injuries that require intervention. The positive predictive value of "soft" signs indicating abnormal findings on an arteriogram is only 35%. The vast majority of these lesions do not require emergent repair.

  • Hypotension or shock
  • Neurologic deficit due to primary nerve injury occurs immediately after injury. In contrast, ischemic neuropathy is delayed in onset, developing within minutes to hours after injury.
  • Stable, nonpulsatile or small hematoma
  • Proximity of the wound to major vascular structures
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Causes

Causes of peripheral vascular injuries include the following:

  • Gunshot wounds, particularly high-energy rifle and close-range shotgun wounds, cause 70-80% of all vascular injuries that require intervention.
  • Stab wounds - Only 5-10% of cases require intervention.
  • Blunt trauma accounts for 5-10% of cases. The presence of a fracture or dislocation increases the risk. Blunt injuries are often more severe than penetrating injuries due to trauma to adjacent structures. The risk of eventual limb amputation is higher with blunt mechanisms of injury.
  • Iatrogenic injury now accounts for more than 10% of cases. Endovascular procedures such as cardiac catheterization and central line placement are the two most common iatrogenic causes of vascular injury that require intervention. The incidence of iatrogenic injuries is growing in concert with the increased utilization of endovascular procedures.
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Contributor Information and Disclosures
Author

Niels Rathlev, MD, FACEP  Chair, Department of Emergency Medicine, Tufts University School of Medicine and Baystate Medical Center

Niels Rathlev, MD, FACEP is a member of the following medical societies: American College of Emergency Physicians and Society for Academic Emergency Medicine

Disclosure: Nothing to disclose.

Specialty Editor Board

David A Peak, MD  Assistant Residency Director of Harvard Affiliated Emergency Medicine Residency, Attending Physician, Massachusetts General Hospital; Consulting Staff, Department of Hyperbaric Medicine, Massachusetts Eye and Ear Infirmary

David A Peak, MD is a member of the following medical societies: American College of Emergency Physicians, American Medical Association, Society for Academic Emergency Medicine, and Undersea and Hyperbaric Medical Society

Disclosure: Nothing to disclose.

Francisco Talavera, PharmD, PhD  Adjunct Assistant Professor, University of Nebraska Medical Center College of Pharmacy; Editor-in-Chief, Medscape Drug Reference

Disclosure: Medscape Salary Employment

David B Levy, DO, FACEP, FAAEM  Chairman, Department of Emergency Medicine, St Elizabeth Health Center; Associate Professor of Emergency Medicine, Northeastern Ohio Universities College of Medicine

David B Levy, DO, FACEP, FAAEM is a member of the following medical societies: American Academy of Emergency Medicine, American Medical Informatics Association, and Society for Academic Emergency Medicine

Disclosure: Nothing to disclose.

John D Halamka, MD, MS  Associate Professor of Medicine, Harvard Medical School, Beth Israel Deaconess Medical Center; Chief Information Officer, CareGroup Healthcare System and Harvard Medical School; Attending Physician, Division of Emergency Medicine, Beth Israel Deaconess Medical Center

John D Halamka, MD, MS is a member of the following medical societies: American College of Emergency Physicians, American Medical Informatics Association, Phi Beta Kappa, and Society for Academic Emergency Medicine

Disclosure: Nothing to disclose.

Chief Editor

Rick Kulkarni, MD  Attending Physician, Department of Emergency Medicine, Cambridge Health Alliance, Division of Emergency Medicine, Harvard Medical School

Rick Kulkarni, MD is a member of the following medical societies: Alpha Omega Alpha, American Academy of Emergency Medicine, American College of Emergency Physicians, American Medical Association, American Medical Informatics Association, Phi Beta Kappa, and Society for Academic Emergency Medicine

Disclosure: WebMD Salary Employment

References
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