Updated: Jan 18, 2008
Plantar fasciitis is the pain caused by inflammation of the insertion of the plantar fascia on the medial process of the calcaneal tuberosity. Plantar fasciitis may cause significant heel pain, resulting in the alteration of a person's activities. This condition sometimes is called "heel spurs" by the general public. In actuality, many asymptomatic individuals have bony heel spurs, whereas many patients with plantar fasciitis have no bony heel spur.1
For excellent patient education resources, visit eMedicine's Sports Injury Center and Foot, Ankle, Knee, and Hip Center. Also, see eMedicine's patient education articles Running, Arch Pain, and Ankylosing Spondylitis, Neurologic Perspective.
Related eMedicine topics:
Plantar Fasciitis [in the Physical Medicine and Rehabilitation section]
Plantar Fasciitis [in the Emergency Medicine section]
Plantar Fasciitis [in the Orthopedic Surgery section]
A survey of professional football, baseball, and basketball team physicians and trainers found that plantar fasciitis was among the 5 most common foot and ankle injuries observed in professional athletes.2 It is estimated that approximately 1 million patient visits per year are due to plantar fasciitis.3
The plantar fascia is a thickened fibrous aponeurosis that originates from the medial tubercle of the calcaneus, runs forward to insert into the deep, short transverse ligaments of the metatarsal heads, and continues forward to form the fibrous flexor sheathes on the plantar aspect of the toes. The central plantar fascia is the thickest and strongest section, and this segment is also the most likely to be involved with plantar fasciitis. The function of the plantar fascia is to provide static support for the longitudinal arch of the foot and to assist with shock absorption during foot strike.
During running, the vertical forces in the foot at foot strike may reach 2-3 times an individual's body weight.4 The plantar fascia and longitudinal arch are also part of the foot's shock absorption mechanism.
The plantar fascia acts as a windlass mechanism during running (see Clinical, Physical, below). During the heel-off phase of gait, tension increases on the plantar fascia, which acts as a storage of potential energy. During toe-off, the plantar fascia passively contracts, converting the potential energy into kinetic energy and imparting greater foot acceleration.
Palpation over the medial tubercle of the calcaneus usually reproduces the pain of plantar fasciitis. In more severe cases, pain may also be reproduced by palpation over the proximal portion of the plantar fascia. Other maneuvers that may reproduce the pain of plantar fasciitis include passive dorsiflexion of the toes, which is sometimes called a "windlass" test, or having the athlete stand on the tiptoes and toe-walk. In a study by De Garceau et al, having the patient bear weight during the windlass test (see Image 1) increased the sensitivity of the test from 13.6% to 31.8%.6
Related eMedicine topics:
Calcaneus, Fractures
Fractures, Foot
Related eMedicine topics:
Pes Cavus
Pes Planus
Tibial Bowing
Contusions
Lumbosacral Radiculopathy
Abductor digiti quinti nerve entrapment, tarsal tunnel syndrome)
Calcaneal apophysitis (Sever disease)
Calcaneal stress fracture
Calcaneus bone injuries
Entrapment syndromes (eg, medial calcaneal branch of the posterior tibial nerve entrapment, abductor digiti quinti nerve entrapment, tarsal tunnel syndrome)
Fat pad syndrome (atrophy, heel bruise)
Infection
Osteomalacia
Plantar fascia rupture
Reiter syndrome
Tendinitis (eg, of the flexor hallucis longus, flexor hallucis brevis, peroneus longus, tibialis posterior)
Tumor
Related eMedicine topics:
Ankylosing Spondylitis [in the Neurology section]
Ankylosing Spondylitis and Undifferentiated Spondyloarthropathy [in the Rheumatology section]
Disorders of Bone Mineralization
Gout and Pseudogout [in the Emergency Medicine section]
Gout [in the Rheumatology section]
Nerve Entrapment Syndromes
Osteomalacia and Renal Osteodystrophy
Paget Disease
Rheumatoid Arthritis
Rickets
Tarsal Tunnel Syndrome
The initial physical therapy program for plantar fasciitis emphasizes stretching of the calf and foot. The stretching program should include wall stretches, with the knee in both the extended and flexed positions.
Resting and correcting training errors are critical to the treatment of plantar fasciitis. Athletes must modify activities that aggravate this condition; such modifications may be as simple as decreasing the amount, frequency, or intensity of the inciting activity(ies). Athletes are more compliant with a decreased level of activity if they are allowed to increase other nonaggravating activities.10
A strengthening program that emphasizes intrinsic foot muscle strengthening is added in the next phase of physical therapy. Exercises include towel curls, marble pick-ups, and toe taps.5
Anti-inflammatory medications are frequently used to treat plantar fasciitis. Although there is controversy as to whether or not nonsteroidal anti-inflammatory drugs (NSAIDs) actually assist in the physiologic healing process, these agents can be useful as an adjunct to control pain while the individual's plantar fasciitis is being treated with stretching, strengthening, and relative rest (see the Medication section, below).18,19
For cases that do not respond to conservative treatment, a surgical release of the plantar fascia may be considered. Overall, surgical release has a 70-90% success rate in treating patients with this condition; open, endoscopic, or radiofrequency lesioning techniques may be used.20,21,22,23,24,25,26
Potential complications of surgical intervention include flattening of the longitudinal arch and heel hypoesthesia, as well as those that are associated with plantar fascia rupture and corticosteroid injections. Longitudinal arch strain appears to account for over 50% of the chronic complications.27,28
Corticosteroid injections
Night splints
To minimize the chances of reoccurrence of plantar fasciitis, athletes should continue on a maintenance program of daily stretching and/or strengthening at least 2-3 times per week.
Other treatment may include orthotic devices and arch supports.
The goals of pharmacotherapy are to reduce morbidity and prevent complications.
The use of NSAIDs in chronic inflammatory diseases such as plantar fasciitis is somewhat controversial.18 NSAIDs may or may not be beneficial to the physiologic processes of soft-tissue healing. However, NSAIDs have been found to be useful in controlling pain (a useful adjunct in allowing more rapid progress with physical therapy) and in controlling acute inflammation. The disadvantages of these medications are many, including the risk of gastrointestinal (GI) bleeding, gastric pain, and renal damage.41
Member of the propionic acid group of NSAIDs. Available in low-dose form as an OTC medication. Highly protein bound, metabolized in the liver, and eliminated primarily in the urine. Ibuprofen may reversibly inhibit platelet function.
600-800 mg PO tid/qid
Maximum 40 mg/kg PO divided tid/qid
Coadministration with aspirin increases the risk of inducing serious NSAID-related side effects; probenecid may increase the concentrations and, possibly, the toxicity of NSAIDs; may decrease the effect of hydralazine, captopril, and beta-blockers; may decrease the diuretic effects of furosemide and thiazides; may increase PT duration when taking anticoagulants (instruct patients to watch for signs of bleeding); may increase the risk of methotrexate toxicity; phenytoin levels may be increased when administered concurrently
Documented hypersensitivity; patients with peptic ulcer disease, recent GI bleeding or perforation, renal insufficiency, or high risk of bleeding
B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals
D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus
Caution in patients with congestive heart failure, hypertension, and decreased renal and hepatic function; caution in the presence of anticoagulation abnormalities or during anticoagulant therapy
Member of the propionic acid group of NSAIDs. Available in low-dose form as an over-the-counter medication. Highly protein bound, metabolized in the liver, and eliminated primarily in the urine. Naproxen may reversibly inhibit platelet function.
250-550 mg PO bid/tid; maximum 1100 mg when used for pain control and acute musculoskeletal injury; maximum daily dose is 1650 mg for all conditions
Maximum 10 mg/kg PO divided bid
Coadministration with aspirin increases the risk of inducing serious NSAID-related side effects; probenecid may increase the concentrations and, possibly, the toxicity of NSAIDs; may decrease the effect of hydralazine, captopril, and beta-blockers; may decrease the diuretic effects of furosemide and thiazides; may increase PT duration when taking anticoagulants (instruct patients to watch for signs of bleeding); may increase the risk of methotrexate toxicity; phenytoin levels may be increased when administered concurrently
Documented hypersensitivity; peptic ulcer disease; recent GI bleeding or perforation; renal insufficiency
B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals
D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus
Acute renal insufficiency, interstitial nephritis, hyperkalemia, hyponatremia, and renal papillary necrosis may occur; patients with preexisting renal disease or compromised renal perfusion risk acute renal failure; leukopenia occurs rarely, is transient, and usually returns to normal during therapy; persistent leukopenia, granulocytopenia, or thrombocytopenia warrants further evaluation and may require discontinuation of the drug.
Corticosteroids are strong anti-inflammatory agents. The general risks involved with the use of these agents include skin atrophy, skin hypopigmentation, soft-tissue atrophy, infection, bleeding, and failure to work. A steroid flare-up, which consists of increased pain for up to several days, may occur in up to 2% of individuals who use corticosteroids.42
Injectable corticosteroid, used to treat localized areas of inflammation. Good evidence exists to suggest that injected corticosteroids alter the long-term pathology of chronic inflammation18,42 ; however, many patients receive acute symptomatic improvement.42 Triamcinolone acetonide is an injectable intermediate-acting, steroid anti-inflammatory agent.
1 mL of 40 mg/mL solution injected into plantar fascia origin via central or lateral approach
Administer as in adults
Local anesthetics containing the preservatives methylparaben, propylparaben, and phenol may cause flocculation of the steroid; corticosteroids may blunt antibody response in patients receiving immunizations concomitantly
Documented hypersensitivity; fungal, viral, and bacterial skin infections
C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus
Injectable corticosteroids in pregnancy have not been studied; carefully monitor infants for hypoadrenalism when born to mothers who have received substantial exposure to corticosteroids; caution in patients with exposure to chicken pox, strongyloides infestation, active tuberculosis, ocular herpes simplex, psychiatric conditions, ulcerative colitis, diverticulitis, recent intestinal anastomoses, history of peptic ulcer disease, renal insufficiency, hypertension, osteoporosis, diabetes mellitus, thromboembolic disorders, seizures, hypoalbuminemia, hypothyroidism, cirrhosis, hyperlipidemias, glaucoma, cataracts and myasthenia gravis; caution in children, as growth and development may be affected by prolonged courses of corticosteroids, especially if given systemically
Injectable corticosteroid, used to treat localized areas of inflammation. No good evidence exists to suggest that injected corticosteroids alter the long-term pathology of chronic inflammation; however, many patients receive acute symptomatic improvement.42 Betamethasone sodium is an injectable intermediate-acting, steroid anti-inflammatory agent.
Inject 0.5 mL of 6 mg/mL solution into plantar fascia origin via central or lateral approach
Administer as in adults
Local anesthetics containing the preservatives methylparaben, propylparaben, and phenol may cause flocculation of the steroid; corticosteroids may blunt antibody response in patients concomitantly receiving immunizations
Documented hypersensitivity; patients with fungal, viral, and bacterial skin infections
C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus
Injectable corticosteroids in pregnancy have not been studied; carefully monitor infants for hypoadrenalism when born to mothers who have received substantial exposure to corticosteroids; caution in patients with exposure to chicken pox, strongyloides infestation, active tuberculosis, ocular herpes simplex, psychiatric conditions, ulcerative colitis, diverticulitis, recent intestinal anastomoses, history of peptic ulcer disease, renal insufficiency, hypertension, osteoporosis, diabetes mellitus, thromboembolic disorders, seizures, hypoalbuminemia, hypothyroidism, cirrhosis, hyperlipidemias, glaucoma, cataracts, and myasthenia gravis; caution in children because growth and development may be affected by prolonged courses of corticosteroids, especially if given systemically
Athletes with plantar fasciitis may return to activities as limited by their symptoms. The physician might need to plan a strict activities regimen because many athletes tend to ignore pain during activity. Generally, athletes should start at 50% of their usual distance or time with a gradual increase of activity by approximately 10% per week.
In rare cases, the plantar fascia may rupture spontaneously. The risk of such a rupture is greatly increased by a history of treatment with a corticosteroid injection.27
Long-term sequelae of rupture occur in approximately 50% of the patients who have a plantar fascia rupture.27,28 Moreover, longitudinal arch strain accounts for over 50% of the chronic complications of plantar fascia rupture.27,28
Instruct athletes with plantar fasciitis to warm up sufficiently before initiating activity, continue stretching programs, and ice down after activity. Make sure that they wear appropriate shoes and change to a new pair every 250-500 miles (400-800 km).7 Alternating between 2 pairs of shoes seems to help some athletes by allowing the cushioning in the shoes to recover more completely between runs.
Plantar fasciitis can be a frustrating problem for many athletes because of its slow resolution; however, this condition is often resolved in most patients with conservative treatment.19,43 Athletes should be cautioned not to expect overnight resolution, especially if they have more chronic pain or if they continue their activities.19
Educate athletes about the importance of foot and calf strengthening and stretching, appropriate training volume and intensity, and appropriate shoe selection and rotation to decrease the risk of future injury.
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heel spurs, heel pain, inflammation of the plantar fascia, calcaneal pain
Craig C Young, MD, Professor, Departments of Orthopedic Surgery and Community and Family Medicine, Medical Director of Sports Medicine, Sports Medicine Fellowship Director, Medical College of Wisconsin
Craig C Young, MD is a member of the following medical societies: American Academy of Family Physicians, American College of Sports Medicine, American Medical Society for Sports Medicine, Phi Beta Kappa, and Wilderness Medical Society
Disclosure: Nothing to disclose.
Joseph P Garry, MD, Director of Sports Medicine and Sports Medicine Fellowship, Associate Professor of Family Medicine and Exercise and Sport Science, Department of Family Medicine, East Carolina University Brody School of Medicine
Joseph P Garry, MD is a member of the following medical societies: American Academy of Family Physicians, American College of Sports Medicine, American Heart Association, American Medical Society for Sports Medicine, North American Primary Care Research Group, and North Carolina Medical Society
Disclosure: Nothing to disclose.
Francisco Talavera, PharmD, PhD, Senior Pharmacy Editor, eMedicine
Disclosure: Nothing to disclose.
Russell D White, MD, Professor of Medicine, Department of Community and Family Medicine, University of Missouri-Kansas City School of Medicine, Truman Medical Center Lakewood
Disclosure: Nothing to disclose.
Jon B Whitehurst, MD, Clinical Instructor of Surgery, University of Illinois College of Medicine; Partner and Executive Board Member, Rockford Orthopedic Associates; Orthopedic Chairman, Rockford Memorial Hospital
Jon B Whitehurst, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, American Orthopaedic Society for Sports Medicine, and Arthroscopy Association of North America
Disclosure: Nothing to disclose.
Craig C Young, MD, Professor, Departments of Orthopedic Surgery and Community and Family Medicine, Medical Director of Sports Medicine, Sports Medicine Fellowship Director, Medical College of Wisconsin
Craig C Young, MD is a member of the following medical societies: American Academy of Family Physicians, American College of Sports Medicine, American Medical Society for Sports Medicine, Phi Beta Kappa, and Wilderness Medical Society
Disclosure: Nothing to disclose.
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