Updated: Jan 4, 2008
Bicipital tendinitis, or biceps tendinitis, is an inflammatory process of the long head of the biceps tendon and is a common cause of shoulder pain due to its position and function.1,2,3,4 The tendon is exposed on the anterior shoulder as it passes through the humeral bicipital groove and inserts onto the superior aspect of the labrum of the glenohumeral joint. Disorders of the biceps tendon can result from impingement or as an isolated inflammatory injury. Other causes are secondary to compensation for rotator cuff disorders, labral tears, and intra-articular pathology.
For excellent patient education resources, visit eMedicine's Arthritis Center and Sports Injury Center. Also, see eMedicine's patient education articles Tendinitis, Rotator Cuff Injury, and Repetitive Motion Injuries.
Related eMedicine topics:Bicipital tendinitis is frequently diagnosed in association with rotator cuff disease as a component of the impingement syndrome or secondary to intra-articular pathology, such as labral tears.
Related eMedicine topic:
Superior Labral Lesions
Related Medscape topic:
American Orthopaedic Society for Sports Medicine 31st Annual Meeting-NSAIDs and Physical Therapy Effective for Superior Labral Tears
As its name implies, the biceps has 2 proximal heads with a common distal insertion onto the radius. The long head of the biceps merges with the short head of the biceps to form the body of the biceps brachii muscle. This muscle is a powerful supinator and flexor of the forearm.
The long head biceps tendon lies in the bicipital groove of the humerus between the greater and lesser tuberosities and angles 90° inward at the upper end of the groove, crossing the humeral head to insert at the upper edge of the glenoid labrum and supraglenoid tubercle. The long head of the biceps tendon helps to stabilize the humeral head, especially during abduction and external rotation.
Bicipital tendinitis frequently occurs from overuse syndromes of the shoulder, which are fairly common in overhead athletes such as baseball pitchers, swimmers, gymnasts, racquet sport enthusiasts (eg, tennis players), and rowing/kayak athletes.5,6,7 Trauma may occur because of direct injury to the biceps tendon when the arm is passed into excessive abduction and external rotation. This pattern of shoulder injury can also occur in the left shoulder of right-handed golfers. Many overuse injuries coexist with some degree of bicipital tendinitis and rotator cuff tendinitis.
The athletic shoulder differs qualitatively from the biomechanics of the shoulder in daily life because of the higher energies and repetitive motions that are involved in athletic activities. Sports activities that require repetitive overhead motion with inadequate reparative time may cause the biceps tendon to break down.
Related eMedicine topic:
Osteochondroma and Osteochondromatosis
Superior Labrum Lesions
Fracture of the greater or lesser tuberosity
Glenohumeral instability (humeral subluxation)
Glenoid labrum tear
Inflammatory arthropathy
Neoplasm
Peripheral nerve entrapment
Strain and tear of the subscapularis
Synovitis
See also the following on eMedicine:
Adhesive Capsulitis [in the Orthopedic Surgery section]
Adhesive Capsulitis [in the Physical Medicine and Rehabilitation section]
Bursitis [in the Orthopedic Surgery section]
Bursitis [in the Emergency Medicine section]
Multidirectional Glenohumeral Instability
Nerve Entrapment Syndromes [in the Neurology section]
Nerve Entrapment Syndromes of the Lower Extremity [in the Orthopedic Surgery section]
Peripheral Nerve Injuries
Posterior Glenohumeral Instability
Rotator Cuff Disease
Superior Labral Lesions [in the Orthopedic Surgery section]
Related Medscape topic:
Resource Center Arthritis
The initial goals of the acute phase of treatment for bicipital tendinitis are to reduce inflammation and swelling. Patients should restrict over-the-shoulder movements, reaching, and lifting.
Patients should apply ice to the affected area for 10-15 minutes, 2-3 times per day for the first 48 hours. Nonsteroidal anti-inflammatory drugs (NSAIDs), such as ibuprofen, are used for 3-4 weeks to treat inflammation and pain. The degree of immobilization depends upon the degree of the injury and the patient's discomfort. Most authors agree that prolonged immobilization tends to result in a stiff shoulder.
Physical therapy plays a minor role in the treatment of acute bicipital tendinitis; however, some authors recommend daily weighted, pendulum stretch exercises for uncomplicated and mild cases of acute bicipital tendinitis. Use of transcutaneous electrical nerve stimulation (TENS) has been reported with some success.
Phonophoresis and iontophoresis are examples of methods that are used to deliver steroids into inflamed tissue without an injection. Phonophoresis uses ultrasound, whereas iontophoresis uses electrical repulsion to transport medicines through the skin. In order to deliver an effective steroid concentration, the target area should be superficial, and serial application is necessary.
It is important to realize that performing analgesic and steroid injections into the bicipital groove is not without risk. Use care to avoid direct injection into the long head of the biceps tendon itself because this can result in direct trauma to — and may lead to atrophy and/or rupture of — the tendon. Other complications from injections include postinjection infection and inflammatory reaction. In order to exclude a possible missed differential diagnosis, radiographic imaging should be considered if no improvement occurs after treatment.
Consider orthopedic consultation if the patient's symptoms persist longer than 2 months or if biceps tendon rupture occurs.
A local injection of an anesthetic and steroid can be given in the bicipital groove. A combination of 2-3 mL of anesthetic with 1 mL of methylprednisolone (Depo-Medrol; Pfizer Inc, New York, NY) is typically recommended 3-6 weeks after the acute injury. A repeat injection can be performed 4 weeks later if the symptoms have not decreased by 50%. Caution is indicated with additional injections or with patients older than 40 years because there is an increased risk of biceps tendon rupture from repetitive injections. Restrict lifting and overhead activities by the patient for 30 days after the injection.
Physical therapy and rehabilitation are directed toward restoring the integrity and strength of the dynamic and static stabilizers of the shoulder joint while restoring the affected shoulder's ROM, which is critical for most athletes.
The goal of the recovery phase is to achieve and maintain full and painless ROM. Weighted, pendulum stretch exercises are combined with isometric toning. These exercises are recommended 3 times per week throughout the recovery phase. Passive stretching with ROM exercises removes residual shoulder stiffness. The uninvolved shoulder can be used as a standard comparison to achieve symmetric ROM.
Although a rehabilitation program should improve strength and flexibility, adding an interval program can help restore normal joint arthrokinematics. Interval tennis and baseball programs have been developed for highly competitive athletes as these individuals recover from bicipital tendinitis. The patient progresses in a series of steps and stages, with the goal of returning safely to competition without reinjury. The progression of therapy is dependent upon a gradual, painless increase in activity without excessive fatigue.
Failure to recognize concomitant injuries with bicipital tendinitis could result in delayed healing and damage from inappropriate treatment. Physical therapy for shoulder injuries or a misdiagnosed injury may aggravate other conditions in the elbow and neck.
Consider orthopedic consultation if the patient's symptoms persist longer than 2 months or if biceps tendon rupture occurs.
Weighted, pendulum swings should begin with moist heat application to the shoulder on the affected side, followed by therapy with 5- to 10-lb weights, which are held lightly in the hand. The shoulder muscle should be relaxed and the arm kept vertical and close to the body. The arm is allowed to swing back and forth, no greater than 1 inch in any direction. Note: This exercise is not appropriate for patients who have shoulder separation or strain, upper back strain, or neck strain.
The maintenance phase concentrates on the patient developing increased strength and endurance on the affected side. This phase can begin as soon as patient discomfort is effectively controlled and should continue for at least 3 weeks after the pain has completely resolved. When performing strengthening exercises, it is safer for the individual to start out with low tension, followed by a gradual increase in force, because flare-ups can occur.
The patient continues isotonic and isokinetic stretching and is allowed limited participation in sports activities. Monitor the patient and adjust his/her activities as progress allows. Note: Conditioning and proper throwing techniques are important for certain athletes because improper mechanics may result in tissue fatigue and damage.
Surgical intervention is not recommended for bicipital tendinitis if the patient is making a slow and gradual improvement. Surgical treatment is only indicated after a 6-month trial of conservative care is unsuccessful. Although good results have been reported with arthroscopic decompression, acromioplasty with anterior acromionectomy is the standard surgical treatment for bicipital tendinitis. The biceps tendon does not generally undergo tenodesis unless severe attritional wear or eminent rupture is found.7,16 No attempt is made to repair biceps tendon ruptures older than 6 weeks.
Tenodesis is not recommended when it is believed that the tendinitis is reversible. Specific indications for tenodesis of the biceps long head include the following7,16 :
Growing evidence has shown a shift from routine tenodesis to a more individual approach, with considerations such as physiologic age, activity level, expectations, and specific combinations of shoulder pathology as important factors.7 Although new repair techniques continue to be developed, the preference is for preservation of the biceps-labral complex rather than routine surgery. When surgery is performed for bicipital tendinitis, the procedure is typically performed through an open anterior incision. Variations of this surgery include arthroscopic techniques and open exposures with suture anchors through the subclavian portal.
Consider orthopedic consultation if the patient's symptoms persist longer than 2 months or if biceps tendon rupture occurs.
The goals of pharmacotherapy are to reduce patient morbidity and prevent complications.
NSAIDs are anti-inflammatory and non-narcotic medications that have analgesic and antipyretic activities. The mechanism of action of these agents is not known, but NSAIDs may inhibit cyclooxygenase activity and prostaglandin synthesis. Other mechanisms may exist as well, such as inhibition of leukotriene synthesis, lysosomal enzyme release, lipoxygenase activity, neutrophil aggregation, and various cell membrane functions. The treatment of pain tends to be patient specific.
DOC for mild to moderate pain. Inhibits inflammatory reactions and pain by decreasing prostaglandin synthesis.
400-800 mg PO tid/qid
<12 years: Not indicated
>12 years: 20-40 mg/kg/d PO divided tid/qid
Concomitant use with anticoagulants may potentiate anticoagulant effects; effects of oral diabetic hypoglycemic agents may be potentiated with combination use with ibuprofen, leading to hypoglycemia; may decrease the clearance and absorption of methotrexate, lithium, diuretics, and antihypotensives
Documented hypersensitivity to ibuprofen, other NSAIDs, or aspirin; avoid in patients with peptic ulcer disease, recent GI bleeding or perforation, renal insufficiency, and high risk of bleeding
B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals
D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus
Caution in patients with congestive heart failure, hypertension, and decreased renal and hepatic function; caution in the presence of anticoagulation abnormalities or during anticoagulant therapy
Local anesthetics block the generation of conduction impulses in a nerve, thereby preventing the transmission of pain.
An amide-type local anesthetic that shares similar properties with other drugs in this classification, including lidocaine (Xylocaine; AstraZeneca, Mississauga, Ontario, Canada). Has the advantage of a longer duration of anesthesia.
Administer the smallest dose and concentration that is required to produce the desired results. Dose varies with the anesthetic procedure, the area to be anesthetized, the vascularity of the tissues, and individual tolerance.
Local anesthesia: 5-10 mL (0.25% sol) 12.5-25 mg; not to exceed 2.5 g/kg
<12 years: Not recommended
>12 years: Administer as adults
May enhance the effects of CNS depressants; coadministration may increase the toxicity of MAOIs, TCAs, beta-blockers, vasopressors, and phenothiazines; have anti-arrhythmic effects, which may cause additive toxicity interactions with phenytoin, procainamide, propranolol, and quinidine
Documented hypersensitivity
B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals
Systemic absorption produces cardiovascular and CNS effects; rate of absorption is dependent upon the dose, rate of administration, and vascularity of the injection site; aspirate for blood before injection to avoid accidental intravenous administration; adverse reactions include restlessness, anxiety, dizziness, blurred vision, tremors, confusion, seizure, hypotension, palpitations, and syncope; consider reduced dose in patients who have lowered hepatic clearance from disease or age
Glucocorticoids stimulate synthesis of enzymes that decrease inflammatory responses and suppress the immune system.
Methylprednisolone is a potent, intermediate-acting glucocorticoid, which has no mineralocorticoid activity. A useful anti-inflammatory and immunosuppressant agent.
4-80 mg/d intra-articular, intrasynovial, intrabursal, intralesional, or soft-tissue injection
140-835 mcg/kg/d intra-articular, intrasynovial, intrabursal, intralesional, or soft-tissue injection
Glucocorticoids may decrease the effects of PO anticoagulants, isoniazid, insulin, PO hypoglycemic agents, and salicylates
Documented hypersensitivity to ingredients of adrenocorticoid preparations; systemic fungal infections
C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus
Caution in patients with hypertension, diabetes mellitus, tuberculosis, psychiatric disorders, glaucoma, and gastric ulcers; glucocorticoids suppress the immune system, which may result in complications in patients receiving live vaccines and in patients with concomitant infectious disease
Return to activity should not occur until the patient's discomfort and pain is controlled effectively and a monitoring program has been initiated. Some authors recommend waiting 3 weeks after pain has completely resolved before allowing participation in competitive activities.
Individuals who return to a high level of athletic activity too soon may find themselves with symptom flare-ups. Continued straining of an injury that has not healed completely can put the individual at risk for chronic degenerative tissue damage and biceps tendon rupture.
Prevention of bicipital tendinitis is similar to prevention of rotator cuff injuries, including warm-ups before exercise, the use of passive stretching and strengthening exercises, avoidance of painful activities, and the use of proper biomechanics. Increased attention should be made for those athletes at high risk of bicipital tendinitis, such as baseball pitchers. High angular velocity and torques combined with the repetitious nature of pitching results in vulnerability of the shoulder to injury.
Although the prognosis of bicipital tendinitis is dependent upon the degree of injury, most patients do well with treatment (see Treatment: Acute Phase, Recovery Phase, Maintenance Phase, and Surgical Intervention). However, a significant number of patients develop degenerative changes, and spontaneous rupture of the biceps tendon occurs in 10% of patients.
Inform the patient that an increased risk of biceps tendon rupture or chronic inflammatory changes exists if the directed restrictions are not followed.
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bicipital tendinitis, biceps tendinitis/tendonitis, attrition tendinitis/tendonitis of the biceps
Britt A Durham, MD, Director of Risk Management, Assistant Professor, Department of Emergency Medicine, King-Drew Medical Center and University of California at Los Angeles; CFO of Durcress Medical Group
Britt A Durham, MD is a member of the following medical societies: American Academy of Emergency Medicine
Disclosure: Nothing to disclose.
Richard Chambers, MD, Chief of Orthopedic, Diabetes and Amputee Service, Clinical Associate Professor, Department of Orthopedic Surgery, Rancho Los Amigos Medical Center, University of Southern California
Richard Chambers, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, American Diabetes Association, American Orthopaedic Foot and Ankle Society, and Phi Beta Kappa
Disclosure: Nothing to disclose.
David T Bernhardt, MD, Director of Adolescent and Sports Medicine Fellowship, Associate Professor, Department of Pediatrics, University of Wisconsin
David T Bernhardt, MD is a member of the following medical societies: American Academy of Pediatrics, American College of Sports Medicine, and American Medical Society for Sports Medicine
Disclosure: Nothing to disclose.
Francisco Talavera, PharmD, PhD, Senior Pharmacy Editor, eMedicine
Disclosure: Nothing to disclose.
Jon B Whitehurst, MD, Clinical Instructor of Surgery, University of Illinois College of Medicine; Partner and Executive Board Member, Rockford Orthopedic Associates; Orthopedic Chairman, Rockford Memorial Hospital
Jon B Whitehurst, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, American Orthopaedic Society for Sports Medicine, and Arthroscopy Association of North America
Disclosure: Nothing to disclose.
Sherwin SW Ho, MD, Associate Professor, Department of Surgery, Section of Orthopedic Surgery and Rehabilitation Medicine, University of Chicago
Sherwin SW Ho, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, American Orthopaedic Society for Sports Medicine, and Arthroscopy Association of North America
Disclosure: Nothing to disclose.
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