Updated: Aug 12, 2008
Elbow dislocation is the most common dislocation in children; in adults, it is the second most common dislocation after that of the shoulder.1,2,3,4 The elbow is amazingly stable, relying more on bony anatomy configuration for stability rather than ligaments. Considerable force is necessary to dislocate the elbow; sports activities account for up to 50% of elbow dislocations, and this type of injury is more commonly seen in adolescent and young adult populations.
Posterior elbow dislocations comprise over 90% of elbow injuries. Early recognition of this injury is required due to the need for early reduction, given a higher likelihood for poor function and possible neurovascular compromise with delays in reduction.1,2,3,4,5 Associated fractures are not infrequent with elbow dislocations, given the force that is required to dislocate the elbow.
Anterior dislocations are seen much less commonly than posterior dislocations. Divergent dislocations, which result in the ulna and radius dislocating in opposite directions, are even more rare. In the pediatric population, radial head subluxation is the main cause of elbow dislocations.
For excellent patient education resources, visit eMedicine's Breaks, Fractures, and Dislocations Center. Also, see eMedicine's patient education articles Elbow Dislocation and Broken Elbow.
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Joint Reduction, Elbow Dislocation, Posterior
Joint Reduction, Radial Head Dislocation
Posttraumatic Heterotopic Ossification
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The rate of elbow dislocation is 6-13 cases per 100,000 people, and this injury occurs more frequently in males than in females. Of all elbow dislocations, 10-50% are sports related. More than 90% of elbow dislocations are posterior dislocations.
The elbow is primarily a flexion-extension hinge joint, which also allows for pronation and supination. Normal range of motion (ROM) at the elbow should be extension to 0° and flexion to 150°.1,2
The humerus and ulna form a very stable unit, which is generally resistant to disruption unless considerable force is applied. This inherent stability also reduces the likelihood of redislocation. The primary bony stabilizers are the coronoid and radial head.
The medial collateral ligament (MCL) and lateral collateral ligament (LCL) comprise the ligamentous stability of the elbow and act as a back-up system to the elbow's natural bony stability. The MCL consists of 3 bands, the anterior oblique, posterior oblique, and the transverse. The anterior band provides most of the resistance to valgus stress. The LCL has 2 bands, the ulnar collateral and radial collateral.
The 2 main compartments of the elbow are the anterior and posterior compartments. The anterior compartment contains the brachial artery and the ulnar and median nerves. This compartment is more commonly affected by dislocations and is the reason for clinical concern regarding brachial artery disruption and median or ulnar nerve entrapment.1,2,4,6
The ulnar nerve passes posteriorly to the medial epicondyle of the humerus, and then it travels deep in the forearm before becoming more superficial again at the wrist. The close proximity of the ulnar nerve to the medial epicondyle allows for the increased likelihood of entrapment when a dislocation occurs. The median nerve is also frequently affected and travels intimately with the brachial artery, which predisposes to simultaneous injury for both the artery and nerve. The posterior compartment contains the radial nerve and triceps brachii muscle.
Anatomically, the mechanism for elbow dislocations is believed to occur as a continuum of damaged/torn structures, beginning laterally with the ulnar portion of the LCL, followed by complete LCL disruption, then damage to the anterior and posterior compartments. The posterior MCL can then become damaged, leaving the anterior portion intact. Further force can allow the elbow to pivot about the anterior bundle of the MCL, potentially damaging it. The LCL, therefore, is considered to be the initial weak link in elbow dislocations.
In the pediatric population, the clinician should be aware of the 6 ossification centers of the elbow joint as well as the annular ligament. The capitellum, radial head, internal (medial) epicondyle, trochlea, olecranon, and external (lateral) epicondyle (CRITOE) is the order in which the ossification centers appear. These centers may often be mistaken for fractures on x-rays. NOTE: A general rule of thumb for the time of appearance of the ossification centers is "1-3-5-7-9-11," which are the ages in years, corresponding to the CRITOE pneumonic.
In cases in which there is radial head subluxation, the radial head slips under the annular ligament and becomes trapped.
Biomechanically, no single sport definitively increases the risk of elbow dislocations; however, sports that increase the likelihood of a person falling onto an outstretched hand (ie, FOOSH injury) (eg, gymnastics, rollerblading, cycling) may theoretically increase the risk of elbow dislocation.
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Neurovascular assessment and documentation of the clinical evaluation are essential in any elbow dislocation because associated brachial artery and ulnar nerve injuries are frequent. Median nerve injuries are also common.
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Unlike the shoulder, a previous elbow dislocation does not predispose a patient to future dislocations. Elbow dislocations are commonly caused by a fall on an outstretched hand or by a traumatic event. Radial head subluxations in children are usually caused by pulling or yanking on the child's arm when the child's elbow is extended.
Compartment Syndrome, Upper Extremity
Elbow fractures
Monteggia fracture
Pediatric radial head subluxation (nursemaid's elbow)
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Early ROM exercises in stable, reduced elbow dislocations has been shown to be associated with an improved outcome. However, immobilization of the affected elbow for longer than 3 weeks in patients following an elbow dislocation has been associated with loss of ROM compared with patients who start early ROM exercises.10
Seek surgical intervention by an orthopedist if any signs of neurovascular compromise, associated fractures, or nonreducible dislocations are present.
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Obtain orthopedic consultation if any signs of neurovascular compromise, associated fractures, or nonreducible dislocations are present.
Patients with limitations in ROM on follow-up evaluation may benefit from more aggressive physical therapy to regain loss of mobility.
Depending on the severity of the elbow dislocation, it may take several months for the elbow to fully heal. Muscle-strengthening activities, in addition to the ROM program, are important to improve endurance of the elbow. Incorporate sport-specific training as the athlete progresses through rehabilitation to ensure a safe return to his/her sport.13
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Surgical intervention may be needed for a functional flexion contracture or for chronic residual instability.14,15,16
Use of medication for elbow dislocations is beneficial in the acute setting when reduction of the dislocation is to take place. Choosing both an anxiolytic and a pain medication is ideal for a conscious sedation to facilitate reduction. Once reduction has occurred, pain may still be an issue, and it would be reasonable to provide the patient with oral pain medication to use in the outpatient setting.
Anxiolytics allow for relaxation and mild sedation when reduction of a dislocated elbow is attempted. These agents also allow for a lower dose of analgesics to be used.
DOC for anxiolytics. Shorter-acting benzodiazepine sedative-hypnotic that is useful in patients requiring acute and/or short-term sedation. Midazolam is also useful for its amnestic effects.
2.5-5 mg IV; not to exceed 2.5 mg IV over 2 min; not to exceed a total of 10 mg
Allow 2-3 min between doses to assess effect.
May start with 1 mg IV and slowly titrate to effect.
0.05-0.1 mg/kg IV; not to exceed the total dose of 10 mg
0.05-0.15 mg/kg IM 30-60 min before the procedure; not to exceed the total dose of 10 mg
0.25-0.5 mg/kg PO; not to exceed the total dose of 10 mg
Intranasal: 0.2 mg/kg; may repeat in 5-15 min
CNS depressants; alcohol may increase sedation and respiratory depression; narcotic agents may increase hypotension; may increase midazolam serum concentrations with cimetidine, ranitidine, erythromycin, diltiazem, verapamil, fluconazole, ketoconazole, itraconazole; protease inhibitors may decrease the midazolam metabolism
Documented hypersensitivity to midazolam, any components, and cherries (syrup only); acute narrow-angle glaucoma; existing CNS depression
D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus
Caution in patients with renal and hepatic impairment, CHF, and pulmonary disease; respiratory/cardiac monitoring is essential during sedation in children
Sedative hypnotic with short onset of effects and a relatively long half-life. By increasing the action of gamma-aminobutyric acid (GABA), which is a major inhibitory neurotransmitter in the brain, this agent may depress all levels of CNS, including the limbic and reticular formation. When the patient needs to be sedated for longer than a 24-hour period, this medication is excellent.
1-4 mg/dose IV given slowly over 2-5 min; may repeat in 10-15 min; not to exceed 8 mg/12 h
0.05 mg/kg PO/IM (range 0.02-0.09 mg/kg)
0.05 mg/kg IV (range 0.02-0.09 mg/kg; may consider smaller doses 0.01-0.03 mg/kg) and repeat q20 min to achieve effect; give slow IV
Other CNS/respiratory depressants may increase effects
Documented hypersensitivity; narrow-angle glaucoma; severe hypotension
D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus
Use with caution in patients with impaired renal or hepatic function or who have compromised pulmonary function.
Depresses all levels of the CNS (eg, limbic and reticular formation), possibly by increasing the activity of GABA. Individualize the dosage and increase cautiously to avoid adverse effects.
10 mg PO
5 mg IV; may repeat with 2.5 mg if needed
0.2-0.3 mg/kg PO; not to exceed 10 mg; 45-60 min before the procedure
0.04-0.3 mg/kg/dose IV/IM; not to exceed 0.6 mg/kg in 8h period
CNS depressants, cimetidine, and erythromycin may decrease metabolism; valproic acid may displace diazepam from the binding sites, resulting in increased sedation; use with ritonavir is not recommended
Documented hypersensitivity to diazepam or any component (emulsified diazepam injection contains soybean oil, egg yolk, phospholipids); comatose patients; narrow-angle glaucoma
D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus
Caution in patients with other CNS depressants; hypoalbuminemia, and liver or hepatic dysfunction
Pain control is essential to quality patient care. Analgesics ensure patient comfort, promote pulmonary toilet, and have sedating properties, which are beneficial for patients who have sustained trauma or who have sustained injuries.
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Indicated for moderate to severe acute and chronic pain.
2.5-20 mg/dose IV/IM/SC; usual dose of 10 mg; dosing q2-6h
<12 years: 0.05-0.1 mg/kg IV 5 min before the procedure
>12 years: 3-4 mg IV; may repeat in 5 min prn
Phenothiazines may antagonize the analgesic effects of opiate agonists; tricyclic antidepressants, MAO inhibitors, and other CNS depressants may potentiate the adverse effects of morphine.
Documented hypersensitivity to morphine sulfate; increased intracranial pressure; severe respiratory depression; severe liver or renal insufficiency
B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals
Avoid in the presence of hypotension, respiratory depression, nausea, emesis, constipation, and urinary retention; caution in patients with atrial flutter and other supraventricular tachycardias; has vagolytic action and may increase the ventricular response rate
Potent narcotic analgesic with a much shorter half-life than morphine sulfate. DOC for conscious sedation analgesia. Ideal for analgesic action of short duration during anesthesia and immediate postoperative period.
Excellent choice for pain management and sedation with short duration (30-60 min) and easy to titrate. Easily and quickly reversed by naloxone. After initial dose, the subsequent doses should not be titrated more frequently than q3h or q6h thereafter.
When using the transdermal dosage form, most patients are controlled with 72 h dosing intervals. However, some patients require dosing intervals of 48 h.
25-50 mcg IV; repeat doses of 25 mcg up to 4-5 times q5 min if needed
1-2 mcg/kg/dose IV/IM with repeated doses at 30-min intervals prn
Phenothiazines may antagonize the analgesic effects of opiate agonists; tricyclic antidepressants may potentiate the adverse effects of fentanyl when both drugs are used concurrently.
Documented hypersensitivity; increased intracranial pressure; severe hepatic or renal insufficiency; severe respiratory depression
C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus
Caution in the presence of hypotension, respiratory depression, constipation, nausea, emesis, and urinary retention; idiosyncratic reaction, known as chest wall rigidity syndrome, may require neuromuscular blockade in order to increase ventilation.
Drug combination indicated for the relief of moderate to severe pain.
1-2 tab PO q4-6h prn
0.05-0.15 mg/kg/dose up to 5 mg/dose PO q4-6h based on oxycodone component
Phenothiazines may decrease the analgesic effects of this medication; the toxicity increases with the coadministration of either CNS depressants or tricyclic antidepressants.
Documented hypersensitivity to oxycodone, acetaminophen; severe liver or renal insufficiency
C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus
The duration of action may increase in the elderly; be aware of the total daily dose of acetaminophen the patient is receiving; do not exceed 4,000 mg/24h of acetaminophen; higher doses may cause liver toxicity.
Indicated for the treatment of mild to moderate pain.
1-2 tab PO q4h; not to exceed 12 tab/24 h
<3 years: 0.5-1 mg codeine/kg/dose PO q4-6h
3-6 years: 5 mL PO tid/qid prn
7-12 years: 10 mL PO tid/qid prn
>12 years: 15 mL PO q4h prn
CNS depressants, alcohol, phenothiazines, and tricyclic antidepressants can increase the adverse effects of codeine or increase the hepatotoxicity of acetaminophen
Documented hypersensitivity to acetaminophen, codeine, or any component
C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus
Caution in patients with hypersensitivity to morphine, hydromorphone, levorphanol, oxycodone, oxymorphone, and hydrocodone
Drug combination indicated for moderate to severe pain.
1-2 tab or cap PO q4-6h prn
Not established
Coadministration with phenothiazines may decrease analgesic effects; the toxicity increases with CNS depressants or tricyclic antidepressants
Documented hypersensitivity to hydrocodone, acetaminophen; CNS depression; severe respiratory depression
C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus
The tablets contain metabisulfite, which may cause hypersensitivity; caution in patients who are dependent on opiates because this substitution may result in acute opiate-withdrawal symptoms; caution in patients with severe renal or hepatic dysfunction
A follow-up examination before the patient's return to play is necessary to reassess motion of the formerly dislocated elbow following the immobilization and early ROM period. Most individuals can return to play 3-6 weeks following an elbow dislocation.
In sports in which an elbow dislocation occurs in a player's nondominant arm, return to play may occur at the earlier end of the rehabilitation spectrum — as long as motion is back to a level that is suitable to the physician and athlete. For elbow dislocations that occur in a player's dominant arm, return to play may take a longer time period. Throwing sports, such as baseball, may require the patient to undergo periods of rest up to 3 months following a dislocation, followed by a strengthening rehabilitative program once full motion is achieved.
Complications of elbow dislocation primarily include neurovascular compromise, compartment syndrome, and loss of ROM. Chronic regional pain syndrome may occur. Close attention to the neurologic examination pre- and postreduction as well as at the follow-up visit may alert the physician to potential neurologic problems.
Elbow dislocations in children due to radial head subluxation (nursemaid's elbow) are often preventable. A child should not be forcibly pulled, lifted, or swung in the air by the hand or wrist. Always lift a small child from under the arms, rather than by the hand or wrist. Athletes who participate in high-risk sports, where falling on an outstretched arm is common, may be advised to wear protective gear to prevent elbow injuries.
Approximately 50% of patients with dislocated elbows achieve a full recovery, including full ROM. One third of patients experience some limitation of motion at the elbow, usually less than 10° of compromised motion. The remaining 10-15% of patients have more significant losses in function, primarily related to limited ROM. Some correlation exists between the severity of the initial injury and the likelihood of having significant motion limitations further in time from the injury occurrence.
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elbow dislocation, dislocation of elbow, dislocated elbow, radial head dislocation, ulnar dislocation, radial head subluxation, FOOSH injury, falling on an outstretched hand, nursemaid's elbow, elbow injury, elbow trauma
Mark E Halstead, MD, Clinical Instructor, Departments of Pediatrics and Orthopedics, Washington University School of Medicine; Team Physician, St Louis Rams, Washington University Athletics
Mark E Halstead, MD is a member of the following medical societies: American Academy of Pediatrics, American College of Sports Medicine, and American Medical Society for Sports Medicine
Disclosure: Nothing to disclose.
David T Bernhardt, MD, Director of Adolescent and Sports Medicine Fellowship, Associate Professor, Department of Pediatrics, University of Wisconsin
David T Bernhardt, MD is a member of the following medical societies: American Academy of Pediatrics, American College of Sports Medicine, and American Medical Society for Sports Medicine
Disclosure: Nothing to disclose.
Joseph P Garry, MD, Director of Sports Medicine and Sports Medicine Fellowship, Associate Professor of Family Medicine and Exercise and Sport Science, Department of Family Medicine, East Carolina University Brody School of Medicine
Joseph P Garry, MD is a member of the following medical societies: American Academy of Family Physicians, American College of Sports Medicine, American Heart Association, American Medical Society for Sports Medicine, North American Primary Care Research Group, and North Carolina Medical Society
Disclosure: Nothing to disclose.
Francisco Talavera, PharmD, PhD, Senior Pharmacy Editor, eMedicine
Disclosure: Nothing to disclose.
Henry T Goitz, MD, Chief, Sports Medicine, Associate Professor, Department of Orthopaedic Surgery, Medical College of Ohio
Henry T Goitz, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons and American Orthopaedic Society for Sports Medicine
Disclosure: Nothing to disclose.
Jon B Whitehurst, MD, Clinical Instructor of Surgery, University of Illinois College of Medicine; Partner and Executive Board Member, Rockford Orthopedic Associates; Orthopedic Chairman, Rockford Memorial Hospital
Jon B Whitehurst, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, American Orthopaedic Society for Sports Medicine, and Arthroscopy Association of North America
Disclosure: Nothing to disclose.
Sherwin SW Ho, MD, Associate Professor, Department of Surgery, Section of Orthopedic Surgery and Rehabilitation Medicine, University of Chicago
Sherwin SW Ho, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, American Orthopaedic Society for Sports Medicine, and Arthroscopy Association of North America
Disclosure: Nothing to disclose.
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