Updated: Mar 27, 2009
Olecranon bursitis is a condition in which there is inflammation of the bursa that overlies the olecranon process at the proximal aspect of the ulna (see Images 1-3 or below).
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Olecranon bursitis is a relatively common condition.
Based on its location between the ulna and the skin at the posterior tip of the elbow, the olecranon bursa functions to provide a mechanism for the skin to glide freely over the olecranon process, thereby preventing tissue tears.
Bursal inflammation may be caused by a variety of mechanisms. Due to the superficial location of the olecranon bursa, it is susceptible to inflammation that is caused by acute or repetitive trauma (see Images 1-3). Acute injuries during sports activities can include any action that involves direct trauma to the posterior elbow (eg, falls). Common causes of olecranon bursal inflammation that are unrelated to sports activities include repetitive microtrauma (eg, the elbow constantly rubbing against a table during writing). Less commonly, the inflammation may be due to infection (septic bursitis).
Olecranon bursitis is shown in a patient with the elbow extended. Image © 2007 by Patrick Foye, MD, UMDNJ New Jersey Medical School.
Arthritis, Rheumatoid. Rheumatoid nodules at the elbow. Photograph by David Effron MD, FACEP
Olecranon fracture.
Bursal inflammation may be caused by a variety of mechanisms. Due to the superficial location of the olecranon bursa, it is susceptible to inflammation caused by acute or repetitive trauma, and less commonly, infection.
Elbow and Forearm Overuse Injuries
Gout
Gout and Pseudogout
Olecranon Fractures
Triceps Tendon Avulsion
Fracture of the olecranon process of the ulna
Olecranon traction osteophyte (with or without avulsion)
Presence of infection (the most important consideration)
Rheumatoid arthritis
Synovial cyst of the elbow joint
Triceps tendinitis/tear
In general, physical and occupational therapy are not needed for olecranon bursitis. In some cases of nonseptic bursitis, however, the physician may recommend a course of physical or occupational therapy to speed the patient's recovery time. Individuals who exhibit olecranon bursitis are often advised to apply the RICE (ie, rest, ice, compression, elevation) method of treatment.
Physical therapy modalities (eg, phonophoresis, electrical stimulation) may also be helpful to further reduce pain and inflammation, although these modalities are not necessary for most patients with this condition.10 The therapist can also educate the patient and present compensatory strategies for resting the involved upper extremity while healing is occurring. If the patient's condition becomes severe and does not respond to conservative treatment, surgery may be indicated. For patients who undergo bursectomy (bursal excision), postoperative physical therapy may be recommended for regaining or maintaining ROM and strength of the elbow.
Usually, no surgical intervention is required in cases of olecranon bursitis; however, very severe cases may require bursectomy. A study by Ogilvie-Harris and Gilbart demonstrated endoscopic bursal resection relieves pain symptoms in patients with olecranon bursitis.11 Fortunately, most cases of olecranon bursitis respond to nonsurgical treatment.
Medical issues and complications of olecranon bursitis during the recovery phase are the same as those listed for the acute phase (see Acute Phase, Medical Issues/Complications).
For cases of olecranon bursitis in which there is repeated recurrence, consider use of a posterior plaster splint to limit elbow motion for 1-2 weeks following aspiration. For severely recalcitrant cases, consider referral to an orthopedic surgeon for possible bursal excision.
Oral NSAIDs can be helpful to reduce the pain and inflammation of olecranon bursitis, but these products probably should be avoided if joint aspiration reveals a hemorrhagic bursitis. Injectable corticosteroid can be beneficial in cases in which the history, physical examination, and joint aspiration do not raise a significant suspicion for infection. Various oral NSAIDs can be used, but none holds a clear distinction as the drug of choice (DOC).
Empiric antibiotic selection is based upon the suspected source of the microorganisms (skin flora with local invasion via puncture or abrasion vs hematogenous spread from a primary infection at another body site). Antibiotic selection is further modified by the results of the culture and sensitivity. Initial antibiotic selection would also be directed by the result of the Gram stain of the aspirate. Antibiotic treatment may start with a broad-spectrum antibiotic; then, when the culture results are available, the antibiotic regimen may be modified as appropriate.
NSAIDs can help decrease pain and inflammation. Various oral NSAIDs can be used, but none holds a clear distinction as the DOC. Choice of NSAID is largely a matter of convenience (eg, how frequently doses must be taken to achieve adequate analgesic and anti-inflammatory effects) and cost.
Commonly used NSAID. Many doses are available, either with or without a prescription.
200-800 mg PO tid/qid
<6 months: Not established
6 months to 12 years: 4-10 mg/kg/dose PO tid/qid
>12 years: Administer as in adults.
May increase the retention of sodium and fluid and may raise the blood pressure in patients on ACE inhibitors and diuretics; may increase the risk of bleeding (eg, GI) among individuals who are already taking alcohol, aspirin, corticosteroids, heparin, or warfarin
Documented hypersensitivity; aspirin/NSAID-induced asthma; relative contraindications: use caution in elderly patients or patients with a history of GI bleed, hypertension, or CHF
C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus
D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus
To minimize side-effect risk, avoid administering multiple NSAIDs concurrently; caution needed in patients who are on anticoagulants or systemic corticosteroids and in patients with a bleeding disorder or significant alcohol use
For the relief of mild to moderate pain and inflammation. Small dosages are initially indicated in small and elderly patients and in those with renal or liver disease. Doses over 75 mg do not increase therapeutic effects. Administer high doses with caution, and closely observe the patient for response.
25-50 mg PO q6-8h prn; not to exceed 300 mg/d
<3 months: Not established
3 months to 12 years: 0.1-1 mg/kg PO q6-8h
>12 years: Administer as in adults.
Coadministration with aspirin increases the risk of inducing serious NSAID-related side effects; probenecid may increase the concentrations and, possibly, the toxicity of NSAIDs; may decrease the effect of hydralazine, captopril, and β -blockers; may decrease the diuretic effects of furosemide and thiazides; monitor PT duration closely (instruct patients to watch for signs of bleeding); may increase the risk of methotrexate toxicity; phenytoin levels may be increased when administered concurrently.
Documented hypersensitivity; aspirin/NSAID-induced asthma; caution in elderly patients or patients with a history of GI bleed, hypertension, or CHF
B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals
D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus
Caution in patients with CHF, hypertension, and decreased renal and hepatic function; caution in the presence of anticoagulation abnormalities or during anticoagulant therapy
In contrast to the widespread systemic distribution of an oral anti-inflammatory drug, a local corticosteroid injection can achieve focal placement of a potent anti-inflammatory agent at the site of maximal tenderness or inflammation.1,3,9,10,13
When corticosteroid injections are used, a variety of corticosteroid preparations are available to choose from. Commonly, the corticosteroid is mixed with a local anesthetic agent before administering the injection. Again, there are various local anesthetic agents to choose from.
Decreases inflammation by suppressing migration of polymorphonuclear leukocytes and reversing increased capillary permeability. Commonly used for injections into bursae or joints.
10 mg (1 mL) intralesionally; often mixed with a few mL lidocaine
Not established
Local corticosteroid injections are not known to have the same degree of medication interactions as those seen with oral or other systemic administration of corticosteroids.
Documented hypersensitivity; skin infection at the site of injection; use caution when performing injections in any patient who is on anticoagulants or who has a history of bleeding disorders, due to the risk of hemorrhage or local bruising
C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus
Never inject corticosteroids through an infected area of skin; diabetic patients may sometimes experience a transient elevation of blood glucose levels after a local corticosteroid injection.
The athlete with olecranon bursitis may be expected to return to play without restrictions when he/she has demonstrated resolution of symptoms and any positive physical examination findings (eg, swelling, tenderness to palpation), as well as shown adequate performance in sports-specific practice drills without recurrence of symptoms or physical examination findings.
Complications of olecranon bursitis include progressive or persistent pain with associated difficulty in using the affected upper extremity. Potential complications due to focal corticosteroid injection include bleeding, bruising, infection, and allergic reactions. Transient elevation of blood glucose levels may occur after corticosteroid injection in a diabetic patient. Intravascular injection could potentially cause cardiac arrhythmia due to the local anesthetic component. Peripheral nerve dysfunction is possible if the injection is administered near or within a major nerve.
Avoid excessive pressure over the elbow. Avoid further trauma, if preventable. If an athlete plays contact sports, there may be no way to guarantee avoidance of further trauma to the site. Consider the use of elbow pads to cushion the region from further trauma.
In the absence of infection, most cases of olecranon bursitis respond very well to a series of 1-2 joint aspirations (with or without corticosteroid injection) combined with the nonsurgical treatments outlined above (see Treatment). Some patients may experience recurrence of olecranon bursitis, in which even a relatively minor bump causes a significant effusion to return at this site.
The patient should be educated regarding the nature of the diagnosis, causative factors, and treatment plan for olecranon bursitis. The most important aspect of patient education is ensuring that the patient knows to immediately report any signs or symptoms of persistent drainage or infection, particularly if a corticosteroid injection has been given. Diabetic patients should be instructed that they may experience a transient increase in blood glucose levels.
Patients should be informed that symptomatic improvement from the corticosteroid usually does not begin to take effect until a few days after the injection. Patients should also understand that they may experience a transient mild increase in symptoms during the window of time when the local anesthetic has worn off but when the steroids have not begun to have a therapeutic effect.
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olecranon bursitis, bursitis, tendinitis, elbow pain, olecranon, student's elbow, draftsman's elbow, swollen/inflamed elbow, painful elbow
Patrick M Foye, MD, FAAPMR, FAAEM, Associate Professor of Physical Medicine and Rehabilitation, Co-Director of Musculoskeletal Fellowship, Co-Director of Back Pain Clinic, Director of Coccyx Pain Service (Tailbone Pain Service: www.TailboneDoctor.com), University of Medicine and Dentistry of New Jersey, New Jersey Medical School
Patrick M Foye, MD, FAAPMR, FAAEM is a member of the following medical societies: American Academy of Physical Medicine and Rehabilitation, American Association of Neuromuscular and Electrodiagnostic Medicine, Association of Academic Physiatrists, and International Spine Intervention Society
Disclosure: Nothing to disclose.
Todd P Stitik, MD, Professor, Department of Physical Medicine and Rehabilitation; Director, Outpatient Occupational/Musculoskeletal Medicine, UMDNJ-New Jersey School of Medicine
Todd P Stitik, MD is a member of the following medical societies: American Academy of Physical Medicine and Rehabilitation, Association of Academic Physiatrists, Phi Beta Kappa, and Physiatric Association of Spine, Sports and Occupational Rehabilitation
Disclosure: Nothing to disclose.
Scott F Nadler, DO, Assistant Director of Occupational/Musculoskeletal Medicine, Assistant Professor of Physical Medicine and Rehabilitation, University of Medicine and Dentistry of New Jersey, Director of Sports Medicine, University Hospital
Scott F Nadler, DO is a member of the following medical societies: American College of Occupational and Environmental Medicine, American College of Sports Medicine, American Medical Association, Association of Academic Physiatrists, North American Spine Society, and Physiatric Association of Spine, Sports and Occupational Rehabilitation
Disclosure: Nothing to disclose.
Andrew L Sherman, MD, MS, Associate Professor of Clinical Rehabilitation Medicine, Vice Chairman, Chief of Spine and Musculoskeletal Services, Program Director, SCI Fellowship and PMR Residency Programs, Department of Rehabilitation Medicine, Leonard A Miller School of Medicine, University of Miami
Andrew L Sherman, MD, MS is a member of the following medical societies: American Academy of Physical Medicine and Rehabilitation, American Association of Neuromuscular and Electrodiagnostic Medicine, American College of Sports Medicine, American Medical Association, American Paraplegia Society, American Spinal Injury Association, and Association of Academic Physiatrists
Disclosure: Nothing to disclose.
Francisco Talavera, PharmD, PhD, Senior Pharmacy Editor, eMedicine
Disclosure: Nothing to disclose.
Henry T Goitz, MD, Fellowship Director, Sports Medicine, Department of Orthopedic Surgery, Henry Ford Hospital
Henry T Goitz, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons and American Orthopaedic Society for Sports Medicine
Disclosure: Nothing to disclose.
Jon B Whitehurst, MD, Clinical Instructor of Surgery, University of Illinois College of Medicine; Partner and Executive Board Member, Rockford Orthopedic Associates; Orthopedic Chairman, Rockford Memorial Hospital
Jon B Whitehurst, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, American Orthopaedic Society for Sports Medicine, and Arthroscopy Association of North America
Disclosure: Nothing to disclose.
Sherwin SW Ho, MD, Associate Professor, Department of Surgery, Section of Orthopedic Surgery and Rehabilitation Medicine, University of Chicago
Sherwin SW Ho, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, American Orthopaedic Society for Sports Medicine, and Arthroscopy Association of North America
Disclosure: Nothing to disclose.
Greg Gazzillo, 4th year medical student, New Jersey Medical School–UMDNJ, Class of 2007, assisted with the 2006 revision of this manuscript.
Debra Ibrahim, 4th year medical student, New York College of Osteopathic Medicine, Class of 2008, assisted with the 2007 revision of this manuscript.
Evish Kamrava, 4th year medical student, St. George's University School of Medicine, Class of 2009, assisted with the 2008 revision of this manuscript.
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