eMedicine Specialties > Sports Medicine > Wrist and Hand

Hand Dislocation: Treatment & Medication

Author: Jeff Chan, MD, MS, FACEP, Clinical Instructor, Stanford University; Staff Physician, Department of Emergency Medicine, Regional Medical Center of San Jose
Coauthor(s): Eleby R Washington III, MD, FACS, Associate Professor, Department of Surgery, Division of Orthopedics, Charles R Drew University of Medicine and Science; Igor Boyarsky, DO, Director of Triage, Assistant Professor, Department of Emergency Medicine, King-Drew Medical Center, University of California at Los Angeles
Contributor Information and Disclosures

Updated: Mar 27, 2009

Treatment

Although general guidelines have been developed for the treatment of hand injuries, a review of the literature suggests that return-to-play guidelines are dependent on factors such as the severity of the initial injury, age, hand dominance, and the chronicity of injury, which vary with each individual.10

Acute Phase

Rehabilitation Program

Physical Therapy

Patients with DIP dislocations that are stable may begin immediate AROM exercises. The less common unstable dorsal dislocation should be immobilized in 20° of flexion for 2-3 weeks before instituting AROM exercises. Complete collateral ligament injuries should be protected from lateral stress for at least 4 weeks. Palmar DIP dislocations may involve disruption of the insertion of the terminal extensor tendon as well as collateral ligaments and the palmar plate. Splinting the joint in extension for 8 weeks is required for the extensor tendon to heal.1,2,3,4,11,12,13,14,15
 
The duration and position of immobilization of PIP injuries depends on the structures involved and the severity of any disruptions. Dorsal PIP dislocations that are stable postreduction can generally be treated with buddy taping (see Image 8 or below). This limits hyperextension, preventing recurrent dislocation and allows a combination of AROM and gentle PROM exercises.


Buddy taping.

Buddy taping.

Buddy taping.

Buddy taping.


An alternative is the use of a dorsal aluminum foam splint (see Image 7 or below).

Hand dislocation. Dorsal aluminum foam splint.

Hand dislocation. Dorsal aluminum foam splint.

Hand dislocation. Dorsal aluminum foam splint.

Hand dislocation. Dorsal aluminum foam splint.


In the case of a significant collateral ligament injury, the finger used as a splint should be on the side of the injured collateral ligament whenever possible. Uncomplicated PIP dislocations are usually immobilized in extension for 7-10 days. More serious injuries may be followed by up to 6 weeks of buddy-taped AROM. The patient should follow up within 2 weeks to confirm proper splinting and the absence of skin problems.

Following closed reduction of dorsal PIP dislocations, immobilize the joint with a dorsally placed splint in 10-15° of flexion. Once comfortable, the patient can begin active flexion while in the splint. After 2 weeks, the splint can be discontinued and the finger buddy taped for 2 more weeks.

Dorsal PIP dislocations that are unstable in extension (including fracture-dislocations) should be initially treated with extension block splinting at the angle required to keep the joint and the fracture (if present) reduced. This splint is often placed dorsally as an outrigger splint anchored in a short-arm cast with the splint taped to the cast to maintain the flexion angle. The proximal phalanx must be immobilized (taped to the splint) to prevent the patient from achieving too much PIP extension by flexing the MCP joint while the middle and distal phalanges are left free.

Volar dislocations with an intact extensor mechanism that are stable postreduction can usually be treated with buddy taping. If open reduction is required without extensive surgical repair, the joint can usually be immobilized for a few days and then buddy taped. Volar dislocations accompanied by central slip injury require splinting of the PIP joint in extension (leaving the DIP joint free) usually for 4-6 weeks followed by daytime dynamic and nighttime static extension splinting for 2 weeks. The DIP joint should be actively flexed throughout the entire recovery phase.

All of these injuries should be aggressively treated to minimize swelling. Routine elevation consists of propping the forearm and hand up on a pillow at night, wearing a sling, or consciously holding the arm up during the day. Ice is used as needed, especially during the first few days. Wrapping the finger with Coban tape can also be used to treat edema.

For patients seen early after dorsal MCP dislocations that are stable after reduction, rubber band extension block splinting (about 20°) for 2-3 weeks should be tried to prevent recurrence through hyperextension. Collateral ligament injuries should be immobilized in incomplete flexion (50°) for 3 weeks, followed by AROM with the digit buddy taped to protect against lateral deviation stress. Additional physical therapy should be completed with modalities to prevent hand edema and maintain mobilization of all of the joints of the hand.

In CMC dislocations, the hand and wrist are frequently placed in an extension-type splint or cobra-type (extension) cast. Initial therapy should be directed at elevating the hand, decreasing the swelling, and mobilizing all of the stable joints of the hand.

Occupational Therapy

Early AROM exercises should be encouraged with almost all of these injuries. This is relatively easy to achieve with injuries that have been treated with buddy taping or extension block splinting. PROM exercises have a very limited role in the early stages of treatment, with the exception of volar dislocations that are associated with central slip injuries. These need aggressive AROM and PROM exercises of the DIP joint while maintaining the PIP joint in full extension. This helps to minimize both stiffness at the DIP joint and adhesions between the 2 flexor tendons.

In the early stages of MCP and CMC dislocations, the role of occupational therapy is usually limited. However, aggressive mobilization of the fingers of the hand and the wrist joint are carried out after the dislocation is stable and as early in the course of treatment as possible.

Medical Issues/Complications

Appropriate expectations regarding the outcome of any hand injury should be discussed with the patient.

All patients with these injuries should follow up within a week to be checked for some of the more common complications.16 Dorsal PIP dislocations that initially appear stable may develop subluxation with extension, requiring more aggressive treatment. Volar PIP dislocations may also lose reduction, or they may have an occult central slip injury at initial presentation (usually reflected by tenderness over the insertion) that progresses to a complete rupture over the next few days.

Stiffness is a common complication at all stages of treatment, especially with PIP dislocations. The patient should perform gentle AROM exercises of any joint that does not require rigid immobilization. Resolution of stiffness and soreness may take as long as 12-18 months, and permanent residual enlargement of the joint is a possibility.

Collateral ligament injuries (usually radial) commonly accompany finger dislocations and usually respond well to closed treatment with no significant long-term disability. However, the patient should be made aware that the pain from these injuries may take months, up to a year, to completely resolve and that permanent enlargement of the joint is a common sequela. Complications associated with MCP and CMC dislocations usually fit into the following 3 categories:

  • Skin problems
  • Recurrent subluxations or dislocations
  • Stiffness or lack of motion of the joints
The patient should be monitored in the week following the dislocation to evaluate whether any posttraumatic skin ulceration, slough, or breakdown is present that may need to be treated with wound care or antibiotics. Early on, patients should undergo intermittent repeat radiography to verify that the reduction is maintained.

After appropriate immobilization, patients can have evidence of dislocation or subluxation of the extensor tendon to the radial or ulnar side of the MCP joints occupying a place between the 2 metacarpal heads. This is best treated by surgical intervention and proper repair of the torn extensor expansion with a row of interrupted sutures.

In long-term hand dislocation or recurrences, surgery is suggested to expose the joint dorsally and to perform a capsulectomy on either side of the MCP joint to achieve stable reduction. In some cases of acute MCP joint dislocation, the collateral ligament may rupture where it attaches to the tubercle of the metacarpal. This may lead to recurrent instability, but it can usually be treated nonoperatively, except when the collateral ligament rupture occurs on the radial side of the MCP joint of the little finger. In this case, the short abductor muscle of the little finger may pull it into ulnar deviation, requiring operative repair.

Surgical Intervention

Surgery is indicated for any open dislocation (irrigation and debridement [I & D]) and any dislocation not reducible by closed means (open reduction). Any joint that is grossly unstable or that cannot maintain reduction with buddy taping or extension block splinting may require repair of supporting structures (volar plate or collateral ligaments) to maintain the reduction. Volar dislocations accompanied by complete loss of extension at the PIP joint may require open reattachment of the central slip.

DIP injuries are considered chronic after 3 weeks. Chronically subluxed joints may require opening to resect scar tissue to allow tension-free reduction.

Complex fracture-dislocations may require surgery for some combination of open reduction, internal fixation, and repair of supporting structures. External fixation (compass hinge) has been used on some complex PIP fracture-dislocations to provide distraction across the joint, maintain reduction, and allow controlled ROM. Details of surgical therapy for MCP and CMC dislocations were addressed under Procedures.

Consultations

A hand surgeon should see any patient with hand dislocations which require or may require surgery. In addition to the above surgical indications, referral to a hand surgeon is usually indicated for fracture-dislocations, most volar dislocations, and any dislocation with associated tendon injury.

Other Treatment

Other forms of treatment for hand dislocations are rarely indicated.

Recovery Phase

Rehabilitation Program

Occupational Therapy

Stable hand injuries should continue to be treated with gentle AROM exercises and should not require extensive formal occupational therapy unless complications develop. Boutonniere injuries should be treated by early occupational therapy to ensure that ROM exercises are done properly and often, as well as for splint fabrication and more aggressive ROM when full-time immobilization is discontinued. Occupational therapy is also important in most postoperative patients, in fracture-dislocations, and in any patient whose injury begins to develop significant stiffness at any point in the course of recovery.

Medical Issues/Complications

Several complications can arise during this phase. At the first follow-up, the examiner should specifically look for recurrent instability/subluxation in the joint. Early boutonniere deformity (ie, hyperextension at the DIP joint with fixed flexion at the PIP joint) is indicative of a central slip injury and often progresses to a disabling deformity without appropriate treatment. Stiffness can occur in any of these injuries; this can be minimized in most cases by avoidance of excess immobilization and appropriate involvement of occupational therapy and hand surgery at an early stage in high-risk injuries.

In MCP and CMC dislocations, the same problems that are observed in the acute phase can be observed in the recovery phase (see Acute Phase, Physical Therapy). However, stiffness becomes a more predominant complication, and it can occur in any of the joints of the fingers, the MCP joints, and the CMC joints. Treatment involves aggressive occupational therapy to prevent joint ankylosis.

Surgical Intervention

Most patients needing surgery for hand dislocation injuries are best served by treatment in the acute phase. If surgery is needed, it is most likely for the reasons below (see Consultations).

Consultations

A hand surgeon should be involved early in the treatment of any complicated PIP or DIP injury. The need for such consultation would be indicated if the patient presents late after the injury, was inadequately assessed at initial presentation, or develops an unforeseen complication such as recurrent subluxation in a joint that appeared to be stable after reduction.

Other Treatment (Injection, manipulation, etc.)

Other forms of treatment are rarely indicated.

Maintenance Phase

Rehabilitation Program

Physical Therapy

Most patients with uncomplicated hand dislocations should recover good function and do not require ongoing treatment in the maintenance phase.

Occupational Therapy

Any patient with significant chronic stiffness, especially in the PIP joint, can benefit from occupational therapy. Even if surgery is planned, the patient benefits the most if ROM is maximized by dynamic splinting and exercises preoperatively.

Medical Issues/Complications

Most long-term complications of finger dislocations are related to either stiffness or instability. Stiffness is initially best treated with intensive occupational therapy, but a hand surgeon should assess all significant contractures. Lateral instability is usually due to collateral ligament injury, instability in extension to volar plate injury, or a missed fracture.17 Most volar plate injuries heal with immobilization that prevents hyperextension (usually buddy taping), so this complication is often preventable. Depending on the demands of the sport and position played, the finger and specific structures involved, and the degree of instability, some of these problems may benefit from surgery.

Surgical Intervention

A number of procedures are possible to treat instability, stiffness, and fracture nonunions in and around the interphalangeal joints. Severe DIP problems may be treated with arthrodesis, depending on the demands placed on the hand. However, this is not a good option for the PIP joint. Any procedure on the PIP joint is likely to result in some loss of ROM. Therefore, compliance with occupational therapy is critical.

Consultations

Most patients with chronic instability or stiffness in a finger (especially the PIP joint) should be referred for assessment by a hand surgeon.

Other Treatment

Steroid injection may rarely be indicated in a chronically swollen, painful joint in those with a hand dislocation.

Medication

Drugs used for injuries involving the hand include various analgesics. Local anesthetics are also used for digital blocks when necessary.

Nonsteroidal Anti-inflammatory Drugs (NSAIDs)

NSAIDs are most commonly used for the relief of mild to moderate pain. Anti-inflammatory effects may be useful as well. Although the effects of NSAIDs in the treatment of pain tend to be patient specific, ibuprofen is usually the drug of choice (DOC) for initial therapy. Other options include ketoprofen and naproxen.


Ibuprofen (Ibuprin, Advil, Motrin)

DOC for the treatment of mild to moderate pain, if no contraindications exist. This drug inhibits inflammatory reactions and pain probably by decreasing the activity of the enzyme cyclooxygenase, inhibiting prostaglandin synthesis.

Adult

600-800 mg PO q8h prn; not to exceed 3.2 g/d

Pediatric

<6 months: Not established
6 months to 12 years: 20-40 mg/kg/d PO divided tid/qid
>12 years: Administer as in adults

Probenecid may increase the concentrations and possibly the toxicity of NSAIDs; ibuprofen may decrease the effect of angiotensin-converting enzyme (ACE) inhibitors and diuretics when coadministered; PT duration may increase when coadministered with anticoagulants (monitor PT duration and bleeding); ibuprofen and other NSAIDs may increase serum lithium levels and the risk of methotrexate toxicity.

Documented hypersensitivity; hypersensitivity to other NSAIDs; peptic ulcer disease; recent GI bleeding or perforation; renal insufficiency; high risk of bleeding

Pregnancy

B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals

D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus

Precautions

Should be avoided in late pregnancy (premature closure of ductus arteriosus); use with caution in patients with congestive heart failure, hypertension, and decreased renal and hepatic function


Naproxen (Anaprox, Naprelan, Naprosyn, Aleve)

For the relief of mild to moderate pain; inhibits inflammatory reactions and pain by decreasing the activity of cyclooxygenase, which results in a decrease of prostaglandin synthesis.

Adult

500 mg PO, followed by 250 mg q6-8h or 500 mg bid; not to exceed 1.25 g/d

Pediatric

<2 years: Not established
>2 years: 2.5 mg/kg/dose PO; not to exceed 10 mg/kg/d

Probenecid and lithium may increase the concentrations and possibly the toxicity of NSAIDs; conversely, naproxen may decrease the effect of diuretics and ACE inhibitors; PT duration may increase when coadministered with anticoagulants (monitor PT duration and bleeding); coadministration with phenytoin may increase serum phenytoin levels, increasing the pharmacologic and toxic effects of phenytoin.

Documented hypersensitivity; peptic ulcer disease; recent GI bleeding or perforation; renal insufficiency; high risk of bleeding

Pregnancy

B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals

D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus

Precautions

Should be avoided in late pregnancy because it can cause premature closure of the ductus arteriosus; acute renal insufficiency, hyperkalemia, hyponatremia, interstitial nephritis, and renal papillary necrosis may occur; it increases the risk of acute renal failure in patients with preexisting renal disease or compromised renal perfusion; low WBC counts occur rarely and usually return to the reference range in ongoing therapy; discontinue therapy if persistent leukopenia, granulocytopenia, or thrombocytopenia occurs

Analgesics

Analgesic agents are used for pain relief. Acetaminophen is used in patients with mild pain, especially those with a contraindication to NSAID use. Narcotics are used in those with moderate to severe pain.


Acetaminophen (Tylenol, Panadol, Aspirin Free Anacin)

DOC for mild pain in patients with documented hypersensitivity to aspirin or NSAIDs, those with upper GI disease, or those who are taking PO anticoagulants.

Adult

325-650 mg PO q4-6h or 1000 mg tid/qid; not to exceed 4 g/d

Pediatric

<12 years: 10-15 mg/kg/dose PO q4-6h prn; not to exceed 2.6 g/d
>12 years: 325-650 mg PO q4h; not to exceed 5 doses/d

Rifampin can reduce the analgesic effects of acetaminophen; coadministration with barbiturates, carbamazepine, hydantoins, and isoniazid may increase hepatotoxicity

Documented hypersensitivity; known G6PD

Pregnancy

B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals

Precautions

Hepatotoxicity is possible in people with long-term alcoholism following various dose levels; severe or recurrent pain or high or continued fever may indicate a serious illness; APAP is contained in many OTC products, and combined use with these products may result in cumulative APAP doses exceeding the recommended maximum dose


Acetaminophen and codeine (Tylenol With Codeine #3)

A drug combination indicated for the treatment of mild to moderate pain.

Adult

30-60 mg/dose PO based on codeine content q4-6h or 1-2 tab q4h; not to exceed 12 tab/d

Pediatric

0.5-1 mg/kg/dose PO based on codeine content q4-6h; 10-15 mg/kg/dose based on acetaminophen content; not to exceed 2.6 g/d of acetaminophen

Toxicity increases with CNS depressants or tricyclic antidepressants

Pregnancy

C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus

Precautions

Caution in patients dependent on opiates, because this substitution may result in acute opiate withdrawal symptoms; caution in the presence of severe renal or hepatic dysfunction


Hydrocodone bitartrate and acetaminophen (Vicodin ES)

A drug combination indicated for the relief of moderate to severe pain.

Adult

1-2 tab or cap PO q4-6h prn

Pediatric

<12 years: 10-15 mg/kg/dose PO acetaminophen q4-6h prn; not to exceed 2.6 g/d acetaminophen
>12 years: 750 mg PO acetaminophen q4h; not to exceed 10 mg hydrocodone bitartrate in single dose; not to exceed 5 doses/d

Coadministration with phenothiazines may decrease analgesic effects; toxicity increases with CNS depressants or tricyclic antidepressants

Documented hypersensitivity; elevated intracranial pressure

Pregnancy

C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus

Precautions

Tablets contain metabisulfite, which may cause hypersensitivity; caution in patients dependent on opiates, because this substitution may result in acute opiate withdrawal symptoms; caution in the presence of severe renal or hepatic dysfunction

Local Anesthetics

Local anesthetic agents are used for digital block to facilitate reduction or examination of fingers.


Lidocaine (Anestacon, Xylocaine, Dilocaine, Zilactin-L)

Amide local anesthetic used in 1-2% concentration. Inhibits depolarization of type C sensory neurons by blocking sodium channels. For procedures such as these, 1% lidocaine without epinephrine is DOC.

Adult

Use amount needed to achieve adequate local anesthesia administered SC; not to exceed 4 mg/kg

Pediatric

Administer as in adults

Coadministration with cimetidine or beta-blockers increases the toxicity of lidocaine; coadministration with procainamide and tocainide may result in additive cardiodepressant action; may increase the effects of succinylcholine

Documented hypersensitivity; Adams-Stokes syndrome (avoid); Wolff-Parkinson-White syndrome (avoid); severe sinoatrial (SA), atrioventricular (AV), or intraventricular block (avoid if artificial pacemaker not in place)

Pregnancy

B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals

Precautions

Use a solution without preservatives; caution in patients with heart failure, hepatic disease, hypoxia, hypovolemia or shock, respiratory depression, and bradycardia; may increase the risk of CNS and cardiac side effects in elderly patients; high plasma concentrations can cause seizures, heart block, and AV conduction abnormalities

More on Hand Dislocation

Overview: Hand Dislocation
Differential Diagnoses & Workup: Hand Dislocation
Treatment & Medication: Hand Dislocation
Follow-up: Hand Dislocation
Multimedia: Hand Dislocation
References
Further Reading

References

  1. Isani A, Melone CP Jr. Ligamentous injuries of the hand in athletes. Clin Sports Med. Oct 1986;5(4):757-72. [Medline].

  2. Kahler DM, McCue FC 3rd. Metacarpophalangeal and proximal interphalangeal joint injuries of the hand, including the thumb. Clin Sports Med. Jan 1992;11(1):57-76. [Medline].

  3. Lairmore JR, Engber WD. Serious, often subtle, finger injuries. Avoiding diagnosis and treatment pitfalls. Phys Sportsmed. 1998;26(6):57-69.

  4. Bach AW. Finger joint injuries in active patients: pointers for acute and late-phase management. Phys Sportsmed. 1999;27(3).

  5. Mall NA, Carlisle JC, Matava MJ, Powell JW, Goldfarb CA. Upper extremity injuries in the National Football League: part I: hand and digital injuries. Am J Sports Med. Oct 2008;36(10):1938-44. [Medline].

  6. Lubahn JD. Dorsal fracture dislocations of the proximal interphalangeal joint. Hand Clin. Feb 1988;4(1):15-24. [Medline].

  7. Hubbard LF. Metacarpophalangeal dislocations. Hand Clin. Feb 1988;4(1):39-44. [Medline].

  8. Gurland M. Carpometacarpal joint injuries of the fingers. Hand Clin. Nov 1992;8(4):733-44. [Medline].

  9. Inoue G, Maeda N. Irreducible palmar dislocation of the proximal interphalangeal joint of the finger. J Hand Surg [Am]. Mar 1990;15(2):301-4. [Medline].

  10. Kovacic J, Bergfeld J. Return to play issues in upper extremity injuries. Clin J Sport Med. Nov 2005;15(6):448-52. [Medline].

  11. Freiberg A, Pollard BA, Macdonald MR, Duncan MJ. Management of proximal interphalangeal joint injuries. J Trauma. Mar 1999;46(3):523-8. [Medline].

  12. McDevitt ER. Treatment of PIP joint dislocations. Phys Sportsmed. 1998;26(8):85-6.

  13. Melone CP Jr. Joint injuries of the fingers and thumb. Emerg Med Clin North Am. May 1985;3(2):319-31. [Medline].

  14. Thayer DT. Distal interphalangeal joint injuries. Hand Clin. Feb 1988;4(1):1-4. [Medline].

  15. Vicar AJ. Proximal interphalangeal joint dislocations without fractures. Hand Clin. Feb 1988;4(1):5-13. [Medline].

  16. Wilson RL, Liechty BW. Complications following small joint injuries. Hand Clin. May 1986;2(2):329-45. [Medline].

  17. Kiefhaber TR, Stern PJ, Grood ES. Lateral stability of the proximal interphalangeal joint. J Hand Surg [Am]. Sep 1986;11(5):661-9. [Medline].

  18. Best Evidence: Talsma E, de Haart M, Beelen A, Nollet F. The effect of mobilization on repaired extensor tendon injuries of the hand: a systematic review. Arch Phys Med Rehabil. Dec 2008;89(12):2366-72. [Medline].

  19. Durakbasa O, Guneri B. The volar surgical approach in complex dorsal metacarpophalangeal dislocations. Injury. Feb 18 2009;epub ahead of print. [Medline].

  20. Kneser U, Goldberg E, Polykandriotis E, et al. Biomechanical and functional analysis of the pins and rubbers tractions system for treatment of proximal interphalangeal joint fracture dislocations. Arch Orthop Trauma Surg. Jan 2009;129(1):29-37. [Medline].

  21. Liss FE, Green SM. Capsular injuries of the proximal interphalangeal joint. Hand Clin. Nov 1992;8(4):755-68. [Medline].

  22. Stern PJ, Lee AF. Open dorsal dislocations of the proximal interphalangeal joint. J Hand Surg [Am]. May 1985;10(3):364-70. [Medline].

Further Reading

Related eMedicine Topics

Best Evidence

Clinical Trials

National Guidelines Clearinghouse

Keywords

hand dislocation, broken finger, dislocated finger, injured finger, proximal interphalangeal joint dislocation PIP joint dislocation, distal interphalangeal joint dislocation, DIP joint dislocation, carpometacarpal joint dislocation, CMC joint dislocation, metacarpophalangeal joint dislocation, MCP joint dislocation

Contributor Information and Disclosures

Author

Jeff Chan, MD, MS, FACEP, Clinical Instructor, Stanford University; Staff Physician, Department of Emergency Medicine, Regional Medical Center of San Jose
Jeff Chan, MD, MS, FACEP is a member of the following medical societies: American College of Emergency Physicians
Disclosure: Nothing to disclose.

Coauthor(s)

Eleby R Washington III, MD, FACS, Associate Professor, Department of Surgery, Division of Orthopedics, Charles R Drew University of Medicine and Science
Eleby R Washington III, MD, FACS is a member of the following medical societies: American Academy of Orthopaedic Surgeons, American College of Surgeons, American Medical Association, International College of Surgeons, and National Medical Association
Disclosure: Nothing to disclose.

Igor Boyarsky, DO, Director of Triage, Assistant Professor, Department of Emergency Medicine, King-Drew Medical Center, University of California at Los Angeles
Igor Boyarsky, DO is a member of the following medical societies: American Medical Association, American Medical Student Association/Foundation, American Osteopathic Association, American Society of Addiction Medicine, and Society for Academic Emergency Medicine
Disclosure: Nothing to disclose.

Medical Editor

Gerard A Malanga, MD, Director of Pain Management, Overlook Hospital; Director of PM&R Sports Medicine Fellowship, Atlantic Health; Clinical Professor, Department of Physical Medicine and Rehabilitation, UMDNJ-New Jersey Medical School; Clinical Chief, Rehabilitation Medicine and Electrodiagnosis, St Michael's Medical Center; Fellow, American College of Sports Medicine
Gerard A Malanga, MD is a member of the following medical societies: Alpha Omega Alpha, American Academy of Physical Medicine and Rehabilitation, American College of Sports Medicine, North American Spine Society, and Physiatric Association of Spine, Sports and Occupational Rehabilitation
Disclosure: Cephalon Honoraria Speaking and teaching; Endo Honoraria Speaking and teaching; Forest Labs Honoraria Speaking and teaching

Pharmacy Editor

Francisco Talavera, PharmD, PhD, Senior Pharmacy Editor, eMedicine
Disclosure: eMedicine Salary Employment

Managing Editor

Henry T Goitz, MD, Academic Chair and Associate Director, Detroit Medical Center Sports Medicine Institute; Director, Education, Research, and Injury Prevention Center; Co-Director, Orthopaedic Sports Medicine Fellowship
Henry T Goitz, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons and American Orthopaedic Society for Sports Medicine
Disclosure: Nothing to disclose.

CME Editor

Jon B Whitehurst, MD, Clinical Instructor of Surgery, University of Illinois College of Medicine; Partner and Executive Board Member, Rockford Orthopedic Associates; Orthopedic Chairman, Rockford Memorial Hospital
Jon B Whitehurst, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, American Orthopaedic Society for Sports Medicine, and Arthroscopy Association of North America
Disclosure: Nothing to disclose.

Chief Editor

Sherwin SW Ho, MD, Associate Professor, Department of Surgery, Section of Orthopedic Surgery and Rehabilitation Medicine, University of Chicago
Sherwin SW Ho, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, American Orthopaedic Society for Sports Medicine, and Arthroscopy Association of North America
Disclosure: Nothing to disclose.

 
 
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