eMedicine Specialties > Sports Medicine > Wrist and Hand

Wrist Dislocation: Treatment & Medication

Author: Kadeer M Halimi, DO, Department of Emergency Medicine, Texas A&M University Health Sciences Center
Coauthor(s): Derek K Lichota, MD, Assistant Professor, Department of Surgery, Texas A&M University College of Medicine; Senior Staff, Department of Orthopedics, Division of Sports Medicine, Scott and White Memorial Hospital
Contributor Information and Disclosures

Updated: Aug 21, 2008

Treatment

Acute Phase

Rehabilitation Program

Physical Therapy

The rehabilitation of wrist injuries can be divided into 5 phases, as follows5 :

  • Phase I: Control inflammation and edema by using rest, ice, compression, and elevation (RICE).
  • Phase II: Restore the normal soft-tissue environment by using modalities such as scar massage, whirlpool therapy, and elastomer application.
  • Phase III: Increase range of motion (ROM).
  • Phase IV: Increase strength.
  • Phase V: Work on hardening and/or conditioning.

Proper pain control and anti-inflammatory medication are the cornerstone of all phases of rehabilitation.

Related eMedicine topics:
Skin, Tissue Expansion
Toxicity, Nonsteroidal Anti-inflammatory Agents

Related Medscape topics:
Resource Center Pain Management: Advanced Approaches to Chronic Pain Management
Resource Center Pain Management: Pharmacologic Approaches

Occupational Therapy

Occupational therapy can be an essential part of rehabilitation, depending on the expertise of the therapist and the motivation of the patient.

Medical Issues/Complications

An expected outcome of surgical reduction of a wrist dislocation is some loss of motion (see Treatment, Acute Phase, Surgical Intervention). The goal of surgery and rehabilitation is to minimize the loss of motion in the athlete (see Maintenance Phase, Rehabilitation Program).

Surgical Intervention

Closed reduction of the wrist dislocation can be attempted after a complete neurovascular examination is performed and proper radiographs are obtained. The patient should be adequately anesthetized by means of an axillary block or general anesthesia. Closed reduction may be difficult if not impossible; the patient should be advised that open reduction may be needed.

For a perilunate dislocation, initial dorsiflexion is followed by gradual volar flexion. Pronation is then used to reduce the capitate back into the lunate.5

For a lunate dislocation, the steps of perilunate reduction are followed by the operators stabilizing the lunate with their thumb as the capitate is brought into palmar flexion. The initial stages of reduction reproduce the perilunate dislocation before the final reduction.

With a scapholunate dislocation, the wrist is dorsiflexed and radially deviated.

Once closed reduction is attempted, PA and lateral radiographic images are obtained to confirm adequate reduction. The carpal bones are then pinned with Kirschner (K) wires because closed reduction is rarely effective by itself. If closed reduction is not successful, the surgeon must attempt open reduction. The wrist is then placed in a cast for 4-6 weeks.

Early diagnosis and anatomic reduction was noted to be essential in a report by Martinage et al13 ; they can also provide satisfactory functional results. Thus, emergency surgical treatment is required. The investigators preferred a dorsal approach and did not perform primary closed reductions.13

Related eMedicine topics:
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Hand, Anesthesia: Blocks
Local Anesthesia and Regional Nerve Block Anesthesia

Related Medscape topics:
Resource Center Vascular Surgery
Specialty Site Neurology & Neurosurgery
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Specialty Site Radiology
Specialty Site Surgery

Consultations

Promptly consult an orthopedic specialist.

Other Treatment

Most complications can be avoided with prompt diagnosis and treatment.

Recovery Phase

Rehabilitation Program

Physical Therapy

The type of physical therapy depends on the needs of the individual patient. Therapeutic modalities may be continued throughout the recovery phase, in addition to ROM and strengthening activities as needed.

Maintenance Phase

Rehabilitation Program

Physical Therapy

The goal of the maintenance phase is for the patient to regain full strength and ROM of the wrist to enable him or her to complete functional daily activities and to gradually return to sports participation or work. The patient independently functions in a work-hardening or sport-specific training program during this phase to continue strengthening to the affected upper extremity.

Medication

Pain control is essential in providing quality patient care to those with wrist dislocations. Pain control ensures patient comfort and aids physical therapy regimens.

Related eMedicine topics:
Cyclooxygenase Deficiency
Toxicity, Acetaminophen
Toxicity, Narcotics
Toxicity, Nonsteroidal Anti-inflammatory Agents

Related Medscape topics:
Resource Center Adverse Drug Events Reporting
Resource Center Opioids: A Guide to State Opioid Prescribing Policies
Resource Center Pain Management: Advanced Approaches to Chronic Pain Management
Resource Center Pain Management: Pharmacologic Approaches

Nonsteroidal Anti-inflammatory Drugs (NSAIDs)

NSAIDs are most commonly used for the relief of mild to moderate pain. The effects of these agents in the treatment of pain tend to be patient specific, yet ibuprofen is usually the drug of choice (DOC) for initial therapy. Other NSAIDs may also be used.

Cyclooxygenase-2 (COX-2) inhibitors may be considered in patients with adverse effects to the NSAIDs discussed here.


Ibuprofen (Ibuprin, Advil, Motrin)

DOC for patients with mild to moderate pain. Inhibits inflammatory reactions and pain by decreasing prostaglandin synthesis.

Adult

400 mg PO q4-6h, 600 mg PO q6h, or 800 mg PO q8h while symptoms persist; not to exceed 3.2 g/d

Pediatric

20-70 mg/kg/d PO divided tid/qid; start at lower end of dosing range and titrate; not to exceed 2.4 g/d

Coadministration with aspirin increases the risk of serious NSAID-related adverse effects; probenecid may increase the concentrations and, possibly, the toxicity of NSAIDs; may decrease the effect of hydralazine, captopril, and beta-blockers; may decrease the diuretic effects of furosemide and thiazides; may increase PT duration with concurrent anticoagulants (instruct patients to watch for signs of bleeding); may increase the risk of methotrexate toxicity; phenytoin levels may be increased when administered concurrently

Documented hypersensitivity; peptic ulcer disease, recent GI bleeding or perforation, renal insufficiency, or high risk of bleeding

Pregnancy

B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals

D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus

Precautions

Caution in patients with congestive heart failure, hypertension, and decreased renal and hepatic function; caution in the presence of coagulation abnormalities or during anticoagulant therapy


Flurbiprofen (Ansaid)

May inhibit the cyclooxygenase enzyme, which, in turn, inhibits prostaglandin biosynthesis. These effects may result in analgesic, antipyretic, and anti-inflammatory activities.

Adult

200-300 mg/d PO divided bid/qid

Pediatric

Not established

Coadministration with aspirin increases the risk of serious NSAID-related adverse effects; probenecid may increase the concentrations and, possibly, the toxicity of NSAIDs; may decrease the effect of hydralazine, captopril, and beta-blockers; may decrease the diuretic effects of furosemide and thiazides; may increase PT duration with concurrent anticoagulants (instruct patients to watch for signs of bleeding); may increase the risk of methotrexate toxicity; phenytoin levels may be increased when administered concurrently

Pregnancy

C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus

D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus

Precautions

Acute renal insufficiency, interstitial nephritis, hyperkalemia, hyponatremia, and renal papillary necrosis may occur; patients with preexisting renal disease or compromised renal perfusion are at risk of acute renal failure; leukopenia occurs rarely, is transient, and usually resolves during therapy; persistent leukopenia, granulocytopenia, or thrombocytopenia warrants further evaluation and may require discontinuation of the drug.


Ketoprofen (Oruvail, Orudis, Actron)

For the relief of mild to moderate pain and inflammation. Small initial doses are indicated in small or elderly patients and in those with renal or liver disease.

Doses >75 mg do not increase the therapeutic effects. Administer high doses with caution, and closely observe patient for response.

Adult

25-50 mg PO q6-8h prn; not to exceed 300 mg/d

Pediatric

<3 months: Not established

3 months to 12 years: 0.1-1 mg/kg PO q6-8h

>12 years: Administer as in adults

Coadministration with aspirin increases the risk of serious NSAID-related adverse effects; probenecid may increase the concentrations and, possibly, the toxicity of NSAIDs; may decrease the effect of hydralazine, captopril, and beta-blockers; may decrease the diuretic effects of furosemide and thiazides; may increase PT duration with concurrent anticoagulants (instruct patients to watch for signs of bleeding); may increase the risk of methotrexate toxicity; phenytoin levels may be increased when administered concurrently

Pregnancy

B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals

D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus

Precautions

Caution in patients with congestive heart failure, hypertension, and decreased renal and hepatic function; caution in the presence of coagulation abnormalities or during anticoagulant therapy


Naproxen (Naprelan, Aleve, Anaprox, Naprosyn)

For the relief of mild to moderate pain. Inhibits inflammatory reactions and pain by decreasing the activity of cyclooxygenase, which is responsible for prostaglandin synthesis.

Adult

250-500 mg PO bid; may increase to 1.5 g/d for limited periods

Pediatric

<2 years: Not established

>2 years: 2.5 mg/kg/dose PO; not to exceed 10 mg/kg/d

Coadministration with aspirin increases the risk serious NSAID-related adverse effects; probenecid may increase the concentrations and, possibly, the toxicity of NSAIDs; may decrease the effect of hydralazine, captopril, and beta-blockers; may decrease the diuretic effects of furosemide and thiazides; may increase PT duration with concurrent anticoagulants (instruct patients to watch for signs of bleeding); may increase the risk of methotrexate toxicity; phenytoin levels may be increased when administered concurrently

Documented hypersensitivity; peptic ulcer disease; recent GI bleeding or perforation; renal insufficiency

Pregnancy

B - Fetal risk not confirmed in studies in humans but has been shown in some studies in animals

D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus

Precautions

Acute renal insufficiency, interstitial nephritis, hyperkalemia, hyponatremia, and renal papillary necrosis may occur; patients with preexisting renal disease or compromised renal perfusion risk acute renal failure; leukopenia occurs rarely, is transient, and usually resolves during therapy; persistent leukopenia, granulocytopenia, or thrombocytopenia warrants further evaluation and may require discontinuation of the drug.


Celecoxib (Celebrex)

Inhibits primarily COX-2. COX-2 is considered an inducible isoenzyme, induced during pain and inflammatory stimuli. Inhibition of COX-1 may contribute to NSAID GI toxicity. At therapeutic concentrations, COX-1 isoenzyme is not inhibited; thus, GI toxicity may be decreased. Seek the lowest dose of celecoxib for each patient.

Adult

200 mg/d PO qd; alternatively, 100 mg PO bid

Pediatric

Not established

Coadministration with fluconazole may cause an increase in celecoxib plasma concentrations because of inhibition of celecoxib metabolism; coadministration of celecoxib with rifampin may decrease plasma celecoxib concentrations.

Pregnancy

C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus

D - Fetal risk shown in humans; use only if benefits outweigh risk to fetus

Precautions

May cause fluid retention and peripheral edema; caution in patients with compromised cardiac function, hypertension, conditions predisposing patient to fluid retention; caution in the presence of severe heart failure and hyponatremia because circulatory hemodynamics may deteriorate; NSAIDs may mask the usual signs of infection; caution in the presence of existing controlled infections; evaluate therapy when symptoms or laboratory results suggest liver dysfunction

Analgesics

Pain control is essential to providing the quality of care for patients with wrist dislocations. Pain control ensures the patient's comfort and aids in physical therapy regimens. Many analgesics have sedating properties that benefit patients with fractures. Hydrocodone and oxycodone preparations are generally more effective and better tolerated than other narcotic-acetaminophen combinations, such as those containing codeine.


Acetaminophen with codeine (Tylenol-3)

Indicated for the treatment of mild to moderate pain.

Adult

30-60 mg/dose PO q4-6h based on the codeine content or 1-2 tab q4h; not to exceed 4 g/d of acetaminophen

Pediatric

0.5-1 mg/kg/dose PO q4-6h based on the codeine content; 10-15 mg/kg/dose based on the acetaminophen content; not to exceed 2.6 g/d of acetaminophen

The toxicity of codeine increases with CNS depressants, tricyclic antidepressants, MAOIs, neuromuscular blockers, CNS depressants, phenothiazines, and narcotic analgesics; rifampin can reduce the analgesic effects; coadministration with barbiturates, carbamazepine, hydantoins, and isoniazid may increase hepatotoxicity.

Pregnancy

C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus

Precautions

Caution in patients who are dependent on opiates because this substitution may result in acute opiate-withdrawal symptoms; caution in patients with severe renal or hepatic dysfunction; hepatotoxicity with acetaminophen is possible with various dose levels in those with chronic alcoholism; severe or recurrent pain or high or continued fever may indicate serious illness; many OTC products contain acetaminophen, and combined use may result in cumulative acetaminophen doses that exceed the recommended maximum dose.


Hydrocodone and acetaminophen (Vicodin, Norcet, Lortab, Lorcet-HD)

Drug combination indicated for moderate to severe pain.

Adult

1-2 tab or cap PO q4-6h prn pain

Pediatric

<12 years: 10-15 mg/kg acetaminophen/dose PO q4-6h prn; not to exceed 2.6 g/d acetaminophen

>12 years: 750 mg acetaminophen PO q4h; not to exceed 10 mg hydrocodone bitartrate per dose or 5 doses/24 h

Coadministration with phenothiazines may decrease the analgesic effects; toxicity increases with CNS depressants or tricyclic antidepressants

Documented hypersensitivity; high-altitude cerebral edema or elevated intracranial pressure

Pregnancy

C - Fetal risk revealed in studies in animals but not established or not studied in humans; may use if benefits outweigh risk to fetus

Precautions

The tablets contain metabisulfite, which may cause hypersensitivity; caution in patients who are dependent on opiates because this substitution may result in acute opiate-withdrawal symptoms; caution in the presence of severe renal or hepatic dysfunction

More on Wrist Dislocation

Overview: Wrist Dislocation
Differential Diagnoses & Workup: Wrist Dislocation
Treatment & Medication: Wrist Dislocation
Follow-up: Wrist Dislocation
Multimedia: Wrist Dislocation
References

References

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  2. Schwartz DT, Reisdorff EJ. Emergency Radiology. New York, NY: McGraw-Hill Book Co; 2000:47-75.

  3. Cheng CY, Hsu KY, Tseng IC, Shih HN. Concurrent scaphoid fracture with scapholunate ligament rupture. Acta Orthop Belg. Oct 2004;70(5):485-91. [Medline].

  4. Browner BD, Jupiter JB, Levine AM, Trafton PG, eds. Skeletal Trauma. Philadelphia, Pa: WB Saunders Co; 1998:1359-81.

  5. Lichtman DM, Alexander AH, eds. The Wrist and Its Disorders. 2nd ed. Philadelphia, Pa: WB Saunders Co; 1997.

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  8. Reid DC. Sports Injury Assessment and Rehabilitation. New York, NY: Churchill Livingstone; 1992.

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  10. Carlisle JC, Goldfarb CA, Mall N, Matava MJ. Upper extremity injuries in the National Football League. Part II: elbow, forearm, and wrist Injuries. Am J Sports Med. Jun 30 2008;epub ahead of print. [Medline].

  11. Alt V, Sicre G. Dorsal transscaphoid-transtriquetral perilunate dislocation in pseudarthrosis of the scaphoid. Clin Orthop Relat Res. Sep 2004;426:135-7. [Medline].

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  14. Infanger M, Grimm D. Meniscus and discus lesions of triangular fibrocartilage complex (TFCC): treatment by laser-assisted wrist arthroscopy. J Plast Reconstr Aesthet Surg. May 9 2008;epub ahead of print. [Medline].

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Further Reading

Keywords

wrist dislocation, wrist pain, dislocated wrist, dislocation of wrist, lunate dislocation, perilunate dislocation, scapholunate dislocation, hyperextension injury of the wrist, carpal bone injuries, carpal injury, ballottement test, volar tilt test, dorsal tilt test

Contributor Information and Disclosures

Author

Kadeer M Halimi, DO, Department of Emergency Medicine, Texas A&M University Health Sciences Center
Kadeer M Halimi, DO is a member of the following medical societies: American College of Emergency Physicians
Disclosure: Nothing to disclose.

Coauthor(s)

Derek K Lichota, MD, Assistant Professor, Department of Surgery, Texas A&M University College of Medicine; Senior Staff, Department of Orthopedics, Division of Sports Medicine, Scott and White Memorial Hospital
Disclosure: Nothing to disclose.

Medical Editor

Craig C Young, MD, Professor, Departments of Orthopedic Surgery and Community and Family Medicine, Medical Director of Sports Medicine, Sports Medicine Fellowship Director, Medical College of Wisconsin
Craig C Young, MD is a member of the following medical societies: American Academy of Family Physicians, American College of Sports Medicine, American Medical Society for Sports Medicine, Phi Beta Kappa, and Wilderness Medical Society
Disclosure: Nothing to disclose.

Pharmacy Editor

Francisco Talavera, PharmD, PhD, Senior Pharmacy Editor, eMedicine
Disclosure: Nothing to disclose.

Managing Editor

Russell D White, MD, Professor of Medicine, Department of Community and Family Medicine, University of Missouri-Kansas City School of Medicine, Truman Medical Center Lakewood
Disclosure: Nothing to disclose.

CME Editor

Jon B Whitehurst, MD, Clinical Instructor of Surgery, University of Illinois College of Medicine; Partner and Executive Board Member, Rockford Orthopedic Associates; Orthopedic Chairman, Rockford Memorial Hospital
Jon B Whitehurst, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, American Orthopaedic Society for Sports Medicine, and Arthroscopy Association of North America
Disclosure: Nothing to disclose.

Chief Editor

Sherwin SW Ho, MD, Associate Professor, Department of Surgery, Section of Orthopedic Surgery and Rehabilitation Medicine, University of Chicago
Sherwin SW Ho, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, American Orthopaedic Society for Sports Medicine, and Arthroscopy Association of North America
Disclosure: Nothing to disclose.

 
 
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